Found: 15
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Insulin Receptor Isoforms and Insulin-Like Growth Factor Receptor in Human Follicular Cell Precursors from Papillary Thyroid Cancer and Normal Thyroid.
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- Journal of Clinical Endocrinology & Metabolism, 2011, v. 96, n. 3, p. 766, doi. 10.1210/jc.2010-1255
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- Article
Sex Steroids Upregulate the IGF-1R in Prostate Cancer Cells through a Nongenotropic Pathway.
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- Annals of the New York Academy of Sciences, 2009, v. 1155, p. 263, doi. 10.1111/j.1749-6632.2009.04361.x
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- Article
Interleukin-4 Stimulates Papillary Thyroid Cancer Cell Survival: Implications in Patients with Thyroid Cancer and Concomitant Graves’ Disease.
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- Journal of Clinical Endocrinology & Metabolism, 2004, v. 89, n. 6, p. 2880, doi. 10.1210/jc.2003-031639
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- Article
A Novel Autocrine Loop Involving IGF-II and the Insulin Receptor Isoform-A Stimulates Growth of Thyroid Cancer.
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- Journal of Clinical Endocrinology & Metabolism, 2002, v. 87, n. 1, p. 245, doi. 10.1210/jcem.87.1.8142
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- Article
Insulin/Insulin-Like Growth Factor I Hybrid Receptors Overexpression Is Not an Early Defect in Insulin-Resistant Subjects.
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- Journal of Clinical Endocrinology & Metabolism, 2000, v. 85, n. 11, p. 4219, doi. 10.1210/jcem.85.11.6977
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- Article
IGF-II Binding to Insulin Receptor Isoform A Induces a Partially Different Gene Expression Profile from Insulin Binding.
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- Annals of the New York Academy of Sciences, 2004, v. 1028, n. 1, p. 450, doi. 10.1196/annals.1322.053
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- Article
In IGF-I receptor-deficient leiomyosarcoma cells autocrine IGF-II induces cell invasion and protection from apoptosis via the insulin receptor isoform A.
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- Oncogene, 2002, v. 21, n. 54, p. 8240, doi. 10.1038/sj.onc.1206058
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- Article
Insulin receptor activation by IGF-II in breast cancers: evidence for a new autocrine/paracrine mechanism.
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- Oncogene, 1999, v. 18, n. 15, p. 2471, doi. 10.1038/sj.onc.1202600
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- Article
Thyroid Stem Cells But Not Differentiated Thyrocytes Are Sensitive to Slightly Increased Concentrations of Heavy Metals.
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- Frontiers in Endocrinology, 2021, v. 11, p. N.PAG, doi. 10.3389/fendo.2021.652675
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- Article
Monomeric β-amyloid interacts with type-1 insulin-like growth factor receptors to provide energy supply to neurons.
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- Frontiers in Cellular Neuroscience, 2015, v. 9, p. 1, doi. 10.3389/fncel.2015.00297
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- Article
The Inorganic Side of NGF: Copper(II) and Zinc(II) Affect the NGF Mimicking Signaling of the N-Terminus Peptides Encompassing the Recognition Domain of TrkA Receptor.
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- Frontiers in Neuroscience, 2016, v. 10, p. 1, doi. 10.3389/fnins.2016.00569
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- Article
Reactivation of p53 mutants by p53 reactivation and induction of massive apoptosis in thyroid cancer cells.
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- International Journal of Cancer, 2012, v. 130, n. 10, p. 2259, doi. 10.1002/ijc.26228
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- Article
Autocrine IGF-II-Associated Cancers: From a Rare Paraneoplastic Event to a Hallmark in Malignancy.
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- Biomedicines, 2024, v. 12, n. 1, p. 40, doi. 10.3390/biomedicines12010040
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- Article
The Copper(II)-Assisted Connection between NGF and BDNF by Means of Nerve Growth Factor-Mimicking Short Peptides.
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- Cells (2073-4409), 2019, v. 8, n. 4, p. 301, doi. 10.3390/cells8040301
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- Article
Amyloid Beta monomers regulate cyclic adenosine monophosphate response element binding protein functions by activating type‐1 insulin‐like growth factor receptors in neuronal cells.
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- Aging Cell, 2018, v. 17, n. 1, p. 1, doi. 10.1111/acel.12684
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- Article