Found: 12
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Endosomes: guardians against [Ru(Phen)<sub>3</sub>]<sup>2+</sup> photo-action in endothelial cells during in vivo pO<sub>2</sub> detection?
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- Metallomics, 2014, v. 6, n. 12, p. 2279, doi. 10.1039/c4mt00190g
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- Article
The Effect of Uncoated SPIONs on hiPSC-Differentiated Endothelial Cells.
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- International Journal of Molecular Sciences, 2019, v. 20, n. 14, p. 3536, doi. 10.3390/ijms20143536
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- Article
Metabolic switch from fatty acid oxidation to glycolysis in knock‐in mouse model of Barth syndrome.
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- EMBO Molecular Medicine, 2023, v. 15, n. 9, p. 1, doi. 10.15252/emmm.202317399
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- Article
Murine MPDZ‐linked hydrocephalus is caused by hyperpermeability of the choroid plexus.
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- EMBO Molecular Medicine, 2019, v. 11, n. 1, p. N.PAG, doi. 10.15252/emmm.201809540
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- Article
A review of standardized high-throughput cardiovascular phenotyping with a link to metabolism in mice.
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- Mammalian Genome, 2023, v. 34, n. 2, p. 107, doi. 10.1007/s00335-023-09997-w
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- Article
Structural variability of dyads relates to calcium release in rat ventricular myocytes.
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- Scientific Reports, 2020, v. 10, n. 1, p. 1, doi. 10.1038/s41598-020-64840-5
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- Article
Sexual dimorphism in bidirectional SR-mitochondria crosstalk in ventricular cardiomyocytes.
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- Basic Research in Cardiology, 2023, v. 118, n. 1, p. 1, doi. 10.1007/s00395-023-00988-1
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- Article
Capture at the ER-mitochondrial contacts licenses IP<sub>3</sub> receptors to stimulate local Ca<sup>2+</sup> transfer and oxidative metabolism.
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- Nature Communications, 2022, v. 13, n. 1, p. 1, doi. 10.1038/s41467-022-34365-8
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- Article
Defective mitochondrial COX1 translation due to loss of COX14 function triggers ROS-induced inflammation in mouse liver.
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- Nature Communications, 2024, v. 15, n. 1, p. 1, doi. 10.1038/s41467-024-51109-y
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- Article
IP<sub>3</sub> receptor isoforms differently regulate ER-mitochondrial contacts and local calcium transfer.
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- Nature Communications, 2019, v. 10, n. 1, p. N.PAG, doi. 10.1038/s41467-019-11646-3
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- Article
Defective dimerization of FoF1‐ATP synthase secondary to glycation favors mitochondrial energy deficiency in cardiomyocytes during aging.
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- Aging Cell, 2022, v. 21, n. 3, p. 1, doi. 10.1111/acel.13564
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- Article
Endoplasmic reticulum stress induces cardiac dysfunction through architectural modifications and alteration of mitochondrial function in cardiomyocytes.
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- Cardiovascular Research, 2019, v. 115, n. 2, p. 328, doi. 10.1093/cvr/cvy197
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- Article