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Progression to type 1 diabetes in the DPT-1 and TN07 clinical trials is critically associated with specific residues in HLA-DQA1-B1 heterodimers.
- Published in:
- Diabetologia, 2024, v. 67, n. 11, p. 2481, doi. 10.1007/s00125-024-06274-6
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- Publication type:
- Article
The increased ability to present citrullinated peptides is not unique to HLA-SE molecules: arginine-to-citrulline conversion also enhances peptide affinity for HLA-DQ molecules.
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- Arthritis Research & Therapy, 2016, v. 18, p. 1, doi. 10.1186/s13075-016-1153-4
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- Publication type:
- Article
Structure of celiac disease-associated HLA-DQ8 and non-associated HLA-DQ9 alleles in complex with two disease-specific epitopes.
- Published in:
- International Immunology, 2000, v. 12, n. 8, p. 1157, doi. 10.1093/intimm/12.8.1157
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- Article
A modified flow cytometry method for objective estimation of human CD4<sup>+</sup> regulatory T cells (CD4<sup>+</sup> Tregs) in peripheral blood, via CD4/CD25/CD45RO/FoxP3 labeling.
- Published in:
- Cytometry. Part B, 2020, v. 98, n. 3, p. 259, doi. 10.1002/cyto.b.21841
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- Article
Crossreactivity to vinculin and microbes provides a molecular basis for HLA-based protection against rheumatoid arthritis.
- Published in:
- Nature Communications, 2015, v. 6, n. 5, p. 6681, doi. 10.1038/ncomms7681
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- Publication type:
- Article
Nine residues in HLA-DQ molecules determine with susceptibility and resistance to type 1 diabetes among young children in Sweden.
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- Scientific Reports, 2021, v. 11, n. 1, p. 1, doi. 10.1038/s41598-021-86229-8
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- Publication type:
- Article
The spectrum of HLA-DQ and HLA-DR alleles, 2006: a listing correlating sequence and structure with function.
- Published in:
- Immunogenetics, 2007, v. 59, n. 7, p. 539, doi. 10.1007/s00251-007-0224-8
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- Publication type:
- Article
Open reading frame sequencing and structure-based alignment of polypeptides encoded byRT1-Bb,RT1-Ba,RT1-Db, andRT1-Daalleles.
- Published in:
- Immunogenetics, 2004, v. 56, n. 8, p. 585, doi. 10.1007/s00251-004-0725-7
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- Publication type:
- Article
Oral Insulin Delay of Stage 3 Type 1 Diabetes Revisited in HLA DR4-DQ8 Participants in the TrialNet Oral Insulin Prevention Trial (TN07).
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- Diabetes Care, 2024, v. 47, n. 9, p. 1608, doi. 10.2337/dc24-0573
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- Publication type:
- Article
HLA Class II (DR , DQ, DP) Genes Were Separately Associated With the Progression From Seroconversion to Onset of Type 1 Diabetes Among Participants in Two Diabetes Prevention Trials (DPT-1 and TN07).
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- Diabetes Care, 2024, v. 47, n. 5, p. 826, doi. 10.2337/dc23-1947
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- Publication type:
- Article
Association of HLA-DQ Heterodimer Residues -18β and β57 With Progression From Islet Autoimmunity to Diabetes in the Diabetes Prevention Trial-Type 1.
- Published in:
- 2022
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- Publication type:
- journal article
Emulsifiers from Partially Composted Olive Waste.
- Published in:
- Foods, 2019, v. 8, n. 7, p. 271, doi. 10.3390/foods8070271
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- Publication type:
- Article
Functional inhibition related to structure of a highly potent insulin-specific CD8 T cell clone using altered peptide ligands.
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- European Journal of Immunology, 2008, v. 38, n. 1, p. 240, doi. 10.1002/eji.200737762
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- Article
Peptide analysis, stability studies, and structural modeling explain contradictory peptide motifs and unique properties of the NOD mouse MHC class II molecule H2-A.
- Published in:
- European Journal of Immunology, 2002, v. 32, n. 8, p. 2105, doi. 10.1002/1521-4141(200208)32:8<2105::AID-IMMU2105>3.0.CO;2-Q
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- Article
Etiopathogenesis of Insulin Autoimmunity.
- Published in:
- Anatomy Research International, 2012, p. 1, doi. 10.1155/2012/457546
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- Publication type:
- Article
Next-Generation HLA Sequence Analysis Uncovers Seven HLA-DQ Amino Acid Residues and Six Motifs Resistant to Childhood Type 1 Diabetes.
- Published in:
- 2020
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- Publication type:
- journal article
Motifs of Three HLA-DQ Amino Acid Residues (α44, β57, β135) Capture Full Association With the Risk of Type 1 Diabetes in DQ2 and DQ8 Children.
- Published in:
- 2020
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- Publication type:
- journal article
Eleven Amino Acids of HLA-DRB1 and Fifteen Amino Acids of HLA-DRB3, 4, and 5 Include Potentially Causal Residues Responsible for the Risk of Childhood Type 1 Diabetes.
- Published in:
- 2019
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- Publication type:
- journal article