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mGluR5 Negative Modulators for Fragile X: Treatment Resistance and Persistence.
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- Frontiers in Psychiatry, 2021, v. 12, p. 1, doi. 10.3389/fpsyt.2021.718953
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- Article
Selective inhibition of glycogen synthase kinase 3α corrects pathophysiology in a mouse model of fragile X syndrome.
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- Science Translational Medicine, 2020, v. 12, n. 544, p. 1, doi. 10.1126/scitranslmed.aam8572
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- Article
A decrease in eukaryotic elongation factor 2 phosphorylation is required for local translation of sensorin and long-term facilitation in Aplysia.
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- Journal of Neurochemistry, 2017, v. 142, n. 2, p. 246, doi. 10.1111/jnc.14030
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- Article
Compartment-specific, differential regulation of eukaryotic elongation factor 2 and its kinase within Aplysia sensory neurons.
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- Journal of Neurochemistry, 2011, v. 117, n. 5, p. 841, doi. 10.1111/j.1471-4159.2011.07251.x
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- Article
Insights into the Role of Postsynaptic cJun and CREB2 in Persistent Long-Term Synaptic Facilitation.
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- Journal of Neuroscience, 2015, v. 35, n. 21, p. 8039, doi. 10.1523/JNEUROSCI.0897-15.2015
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- Article
Bidirectional Regulation of eEF2 Phosphorylation Controls Synaptic Plasticity by Decoding Neuronal Activity Patterns.
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- Journal of Neuroscience, 2015, v. 35, n. 10, p. 4403, doi. 10.1523/JNEUROSCI.2376-14.2015
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- Article
Serotonin-Induced Cleavage of the Atypical Protein Kinase C Apl III in Aplysia.
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- Journal of Neuroscience, 2012, v. 32, n. 42, p. 14630, doi. 10.1523/JNEUROSCI.3026-11.2012
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- Article