Found: 13
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Disease-, region- and cell type specific diversity of α-synuclein carboxy terminal truncations in synucleinopathies.
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- Acta Neuropathologica Communications, 2021, v. 9, n. 1, p. 1, doi. 10.1186/s40478-021-01242-2
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- Article
Robust α-synuclein pathology in select brainstem neuronal populations is a potential instigator of multiple system atrophy.
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- Acta Neuropathologica Communications, 2021, v. 9, n. 1, p. 1, doi. 10.1186/s40478-021-01173-y
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- Article
Precision therapeutic targets for COVID-19.
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- Virology Journal, 2021, v. 18, n. 1, p. 1, doi. 10.1186/s12985-021-01526-y
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- Article
Prominent amyloid plaque pathology and cerebral amyloid angiopathy in APP V717I (London) carrier – phenotypic variability in autosomal dominant Alzheimer's disease.
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- Acta Neuropathologica Communications, 2020, v. 8, n. 1, p. 1, doi. 10.1186/s40478-020-0891-3
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- Article
Neuronal tissue collection from intra-cranial instruments used in deep brain stimulation surgery for Parkinson's disease with implications for study of alpha-synuclein.
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- Scientific Reports, 2024, v. 14, n. 1, p. 1, doi. 10.1038/s41598-024-72542-5
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- Article
A multiverse of α-synuclein: investigation of prion strain properties with carboxyl-terminal truncation specific antibodies in animal models.
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- Acta Neuropathologica Communications, 2024, v. 12, n. 1, p. 1, doi. 10.1186/s40478-024-01805-z
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- Article
Carboxyl truncation of α-synuclein occurs early and is influenced by human APOE genotype in transgenic mouse models of α-synuclein pathogenesis.
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- Acta Neuropathologica Communications, 2023, v. 11, n. 1, p. 1, doi. 10.1186/s40478-023-01623-9
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- Article
Targeted proteolytic products of t and α-synuclein in neurodegeneration.
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- Essays in Biochemistry, 2021, v. 65, n. 7, p. 905, doi. 10.1042/EBC20210028
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- Article
Unique seeding profiles and prion-like propagation of synucleinopathies are highly dependent on the host in human α-synuclein transgenic mice.
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- Acta Neuropathologica, 2022, v. 143, n. 6, p. 663, doi. 10.1007/s00401-022-02425-4
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- Article
Transgenic Mice Expressing Human α-Synuclein in Noradrenergic Neurons Develop Locus Ceruleus Pathology and Nonmotor Features of Parkinson's Disease.
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- Journal of Neuroscience, 2020, v. 40, n. 39, p. 7559, doi. 10.1523/JNEUROSCI.1468-19.2020
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- Article
Correction: Collusion of α-Synuclein and Aβ aggravating co-morbidities in a novel prion-type mouse model.
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- 2024
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- Correction Notice
Correction: Collusion of α-Synuclein and Aβ aggravating co-morbidities in a novel prion-type mouse model.
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- 2024
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- Publication type:
- Correction Notice
Collusion of α-Synuclein and Aβ aggravating co-morbidities in a novel prion-type mouse model.
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- Molecular Neurodegeneration, 2021, v. 16, n. 1, p. 1, doi. 10.1186/s13024-021-00486-9
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- Article