Found: 30
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A Simple Logic of the Hide and Seek Game.
- Published in:
- Studia Logica, 2023, v. 111, n. 5, p. 821, doi. 10.1007/s11225-023-10039-4
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- Article
Transgenic Overexpression of IL-37 Protects Against Atherosclerosis and Strengthens Plaque Stability.
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- Cellular Physiology & Biochemistry (Karger AG), 2018, v. 45, n. 3, p. 1034, doi. 10.1159/000487344
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- Article
Transgenic Overexpression of IL-37 Protects Against Atherosclerosis and Strengthens Plaque Stability.
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- Cellular Physiology & Biochemistry (Karger AG), 2018, v. 45, n. 3, p. 1034, doi. 10.1159/000487344
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- Article
Cutting Force Fluctuation Suppression and Error Homogenization of Noncircular Gear Hobbing Based on the Tool Shifting Method.
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- Chinese Journal of Mechanical Engineering, 2023, v. 36, n. 1, p. 1, doi. 10.1186/s10033-023-00930-5
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- Article
Inflammatory Cytokine TSLP Stimulates Platelet Secretion and Potentiates Platelet Aggregation via a TSLPR-Dependent PI3K/Akt Signaling Pathway.
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- Cellular Physiology & Biochemistry (Karger AG), 2015, v. 35, n. 1, p. 160, doi. 10.1159/000369684
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- Article
Human Platelets Express Functional Thymic Stromal Lymphopoietin Receptors: a Potential Role in Platelet Activation in Acute Coronary Syndrome.
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- Cellular Physiology & Biochemistry (Karger AG), 2013, v. 32, n. 6, p. 1741, doi. 10.1159/000356608
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- Article
Kruppel-Like Factor 2 Regulates Dendritic Cell Activation in Patients with Acute Coronary Syndrome.
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- Cellular Physiology & Biochemistry (Karger AG), 2013, v. 32, n. 4, p. 931, doi. 10.1159/000354496
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- Article
Thymic Stromal Lymphopoietin Over-Expressed in Human Atherosclerosis: Potential Role in Th17 Differentiation.
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- Cellular Physiology & Biochemistry (Karger AG), 2013, v. 31, n. 2/3, p. 305, doi. 10.1159/000343369
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- Article
Angiotensin II Induces TSLP via an AT1 Receptor/NF-KappaB Pathway, Promoting Th17 Differentiation.
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- Cellular Physiology & Biochemistry (Karger AG), 2012, v. 30, n. 6, p. 1383, doi. 10.1159/000343327
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- Article
CD4CD25<sup>+</sup>Foxp3<sup>+</sup>Regulatory T Cells Protect Endothelial Function Impaired by Oxidized Low Density Lipoprotein via the KLF-2 Transcription Factor.
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- Cellular Physiology & Biochemistry (Karger AG), 2011, v. 28, n. 4, p. 639, doi. 10.1159/000335759
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- Article
Oxidized Low-density Lipoprotein-induced Proinflammatory Cytokine Response in Macrophages are Suppressed by CD4CD25<sup>+</sup>Foxp3<sup>+</sup> Regulatory T Cells Through Downregulating Toll Like Receptor 2-mediated Activation of NF-κB.
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- Cellular Physiology & Biochemistry (Karger AG), 2010, v. 25, n. 6, p. 649, doi. 10.1159/000315084
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- Article
Innate Lymphoid Cells and Myocardial Infarction.
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- Frontiers in Immunology, 2021, v. 12, p. 1, doi. 10.3389/fimmu.2021.758272
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- Article
A Potential Mechanism of High-Dose Ticagrelor in Modulating Platelet Activity and Atherosclerosis Mediated by Thymic Stromal Lymphopoietin Receptor.
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- PLoS ONE, 2015, v. 10, n. 10, p. 1, doi. 10.1371/journal.pone.0141464
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- Article
Kruppel-Like Factor 2-Mediated Suppression of MicroRNA-155 Reduces the Proinflammatory Activation of Macrophages.
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- PLoS ONE, 2015, v. 10, n. 9, p. 1, doi. 10.1371/journal.pone.0139060
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- Article
Oxidized Low Density Lipoprotein Induced Caspase-1 Mediated Pyroptotic Cell Death in Macrophages: Implication in Lesion Instability?
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- PLoS ONE, 2013, v. 8, n. 4, p. 1, doi. 10.1371/journal.pone.0062148
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- Article
Group 2 Innate Lymphoid Cells Protect Mice from Abdominal Aortic Aneurysm Formation via IL5 and Eosinophils.
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- Advanced Science, 2023, v. 10, n. 7, p. 1, doi. 10.1002/advs.202206958
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- Article
Two-sample Mendelian randomization to study the causal association between gut microbiota and atherosclerosis.
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- Frontiers in Immunology, 2024, p. 1, doi. 10.3389/fimmu.2023.1282072
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- Article
Single-cell landscape dissecting the transcription and heterogeneity of innate lymphoid cells in ischemic heart.
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- Frontiers in Immunology, 2023, p. 1, doi. 10.3389/fimmu.2023.1129007
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- Article
Group 2 innate lymphoid cells protect mouse heart from myocardial infarction injury via interleukin 5, eosinophils, and dendritic cells.
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- Cardiovascular Research, 2023, v. 119, n. 4, p. 1046, doi. 10.1093/cvr/cvac144
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- Article
MicroRNA‐181 in cardiovascular disease: Emerging biomarkers and therapeutic targets.
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- FASEB Journal, 2024, v. 38, n. 9, p. 1, doi. 10.1096/fj.202400306R
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- Article
MicroRNA-181b inhibits thrombin-mediated endothelial activation and arterial thrombosis by targeting caspase recruitment domain family member 10.
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- FASEB Journal, 2016, v. 30, n. 9, p. 3216, doi. 10.1096/fj.201500163R
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- Article
CircRNA/lncRNA–miRNA–mRNA network and gene landscape in calcific aortic valve disease.
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- BMC Genomics, 2023, v. 24, n. 1, p. 1, doi. 10.1186/s12864-023-09441-y
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- Article
Losing connection: the modal logic of definable link deletion.
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- Journal of Logic & Computation, 2020, v. 30, n. 3, p. 715, doi. 10.1093/logcom/exz036
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- Article
MEREOLOGICAL BIMODAL LOGICS.
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- Review of Symbolic Logic, 2022, v. 15, n. 4, p. 823, doi. 10.1017/S175502032200003X
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- Article
Sestrin2 Sipperesses Classiccally Activated Macrophages-Mediated Inflammatory Response in Myocerdia Infarction Inhibition pf mTORC1 Signaling.
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- Frontiers in Immunology, 2017, p. 1, doi. 10.3389/fimmu.2017.00728
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- Article
Construction and Bioinformatics Analysis of circRNA-miRNA-mRNA Network in Acute Myocardial Infarction.
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- Frontiers in Genetics, 2022, v. 13, p. 1, doi. 10.3389/fgene.2022.854993
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- Article
Meshing principle and transmission analysis of a beveloid non-circular gear.
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- Advances in Mechanical Engineering (Sage Publications Inc.), 2020, v. 12, n. 11, p. 1, doi. 10.1177/1687814020971909
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- Article
Toll-like receptor-4 and mitogen-activated protein kinase signal system are involved in activation of dendritic cells in patients with acute coronary syndrome.
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- Immunology, 2008, v. 125, n. 1, p. 122, doi. 10.1111/j.1365-2567.2008.02827.x
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- Article
mTORC1 signaling is crucial for regulatory T cells to suppress macrophage-mediated inflammatory response after acute myocardial infarction.
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- Immunology & Cell Biology, 2016, v. 94, n. 3, p. 274, doi. 10.1038/icb.2015.88
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- Article
Eosinophils improve cardiac function after myocardial infarction.
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- Nature Communications, 2020, v. 11, n. 1, p. 1, doi. 10.1038/s41467-020-19297-5
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- Article