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Combined exposure to fine particulate matter and high glucose aggravates endothelial damage by increasing inflammation and mitophagy: the involvement of vitamin D.
- Published in:
- Particle & Fibre Toxicology, 2022, v. 19, n. 1, p. 1, doi. 10.1186/s12989-022-00462-1
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- Article
Particulate matters increase epithelial-mesenchymal transition and lung fibrosis through the ETS-1/NF-κB-dependent pathway in lung epithelial cells.
- Published in:
- Particle & Fibre Toxicology, 2020, v. 17, n. 1, p. N.PAG, doi. 10.1186/s12989-020-00373-z
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- Article
Vitamin D<sub>3</sub> decreases TNF-α-induced inflammation in lung epithelial cells through a reduction in mitochondrial fission and mitophagy.
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- Cell Biology & Toxicology, 2022, v. 38, n. 3, p. 427, doi. 10.1007/s10565-021-09629-6
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- Article
Vitamin D Attenuates Ischemia/Reperfusion-Induced Cardiac Injury by Reducing Mitochondrial Fission and Mitophagy.
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- Frontiers in Pharmacology, 2020, v. 11, p. N.PAG, doi. 10.3389/fphar.2020.604700
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- Article
Nanocurcumin Reduces High Glucose and Particulate Matter-Induced Endothelial Inflammation: Mitochondrial Function and Involvement of miR-221/222.
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- International Journal of Nanomedicine, 2023, v. 18, p. 7379, doi. 10.2147/IJN.S433658
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- Article
Interleukin-6 from Adipose-Derived Stem Cells Promotes Tissue Repair by the Increase of Cell Proliferation and Hair Follicles in Ischemia/Reperfusion-Treated Skin Flaps.
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- Mediators of Inflammation, 2019, p. 1, doi. 10.1155/2019/2343867
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- Article
PM<sub>2.5</sub>-induced oxidative stress increases intercellular adhesion molecule-1 expression in lung epithelial cells through the IL-6/AKT/STAT3/NF-κB-dependent pathway.
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- Particle & Fibre Toxicology, 2018, v. 15, p. 1, doi. 10.1186/s12989-018-0240-x
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- Article