Found: 14
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Transcriptomic point of departure determination: a comparison of distribution-based and gene set-based approaches.
- Published in:
- Frontiers in Genetics, 2024, p. 1, doi. 10.3389/fgene.2024.1374791
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- Article
A Transcriptome-Wide Screen for mRNAs Enriched in Fetal Leydig Cells: CRHR1 Agonism Stimulates Rat and Mouse Fetal Testis Steroidogenesis.
- Published in:
- PLoS ONE, 2012, v. 7, n. 10, p. 1, doi. 10.1371/journal.pone.0047359
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- Article
A microRNA or messenger RNA point of departure estimates an apical endpoint point of departure in a rat developmental toxicity model.
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- Birth Defects Research, 2022, v. 114, n. 11, p. 559, doi. 10.1002/bdr2.2046
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- Article
Development of a human liver microphysiological coculture system for higher throughput chemical safety assessment.
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- Toxicological Sciences, 2024, v. 199, n. 2, p. 227, doi. 10.1093/toxsci/kfae018
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- Article
Transformative Vision for an Omics-Based Regulatory Chemical Testing Paradigm.
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- Toxicological Sciences, 2022, v. 190, n. 2, p. 127, doi. 10.1093/toxsci/kfac097
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- Article
A Rat Liver Transcriptomic Point of Departure Predicts a Prospective Liver or Non-liver Apical Point of Departure.
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- Toxicological Sciences, 2020, v. 176, n. 1, p. 86, doi. 10.1093/toxsci/kfaa062
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- Article
Of Mice and Men (and Rats): Phthalate-Induced Fetal Testis Endocrine Disruption Is Species-Dependent.
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- Toxicological Sciences, 2012, v. 129, n. 2, p. 235, doi. 10.1093/toxsci/kfs206
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- Article
Species-Specific Dibutyl Phthalate Fetal Testis Endocrine Disruption Correlates with Inhibition of SREBP2-Dependent Gene Expression Pathways.
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- Toxicological Sciences, 2011, v. 120, n. 2, p. 460, doi. 10.1093/toxsci/kfr020
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- Article
The orl Rat with Inherited Cryptorchidism Has Increased Susceptibility to the Testicular Effects of In Utero Dibutyl Phthalate Exposure.
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- Toxicological Sciences, 2008, v. 105, n. 2, p. 360, doi. 10.1093/toxsci/kfn140
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- Article
Fetal Mouse Phthalate Exposure Shows that Gonocyte Multinucleation is Not Associated with Decreased Testicular Testosterone.
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- Toxicological Sciences, 2007, v. 97, n. 2, p. 491, doi. 10.1093/toxsci/kfm049
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- Article
Testicular Gene Expression Profiling following Prepubertal Rat Mono-(2-ethylhexyl) Phthalate Exposure Suggests a Common Initial Genetic Response at Fetal and Prepubertal Ages.
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- Toxicological Sciences, 2006, v. 93, n. 2, p. 369, doi. 10.1093/toxsci/kfl049
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- Article
Mono-(2-ethylhexyl) Phthalate Rapidly Increases Celsr2 Protein Phosphorylation in HeLa Cells via Protein Kinase C and Casein Kinase 1.
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- Toxicological Sciences, 2006, v. 91, n. 1, p. 255, doi. 10.1093/toxsci/kfj135
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- Article
Plasma fibronectin supports neuronal survival and reduces brain injury following transient focal cerebral ischemia but is not essential for skin-wound healing and hemostasis.
- Published in:
- Nature Medicine, 2001, v. 7, n. 3, p. 324, doi. 10.1038/85471
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- Article
Human Fetal Testis Xenografts Are Resistant to Phthalate-Induced Endocrine Disruption.
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- Environmental Health Perspectives, 2012, v. 120, n. 8, p. 1137, doi. 10.1289/ehp.1104711
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- Article