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Zn(II)- and Cu(II)-induced non-fibrillar aggregates of amyloid-β (1–42) peptide are transformed to amyloid fibrils, both spontaneously and under the influence of metal chelators.
- Published in:
- Journal of Neurochemistry, 2009, v. 110, n. 6, p. 1784, doi. 10.1111/j.1471-4159.2009.06269.x
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- Publication type:
- Article
Prolonged Aβ treatment leads to impairment in the ability of primary cortical neurons to maintain K<sup>+</sup> and Ca<sup>2+</sup> homeostasis.
- Published in:
- Molecular Neurodegeneration, 2010, v. 5, p. 30, doi. 10.1186/1750-1326-5-30
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- Article
Metallothionein-IIA blocks copper mediated Aβ<sup>1-40</sup> neurotoxicity
- Published in:
- 2009
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- Publication type:
- Abstract
Tg2576 Cortical Neurons That Express Human Ab Are Susceptible to Extracellular Aβ-Induced, K<sup>+</sup> Efflux Dependent Neurodegeneration.
- Published in:
- PLoS ONE, 2011, v. 6, n. 4, p. 1, doi. 10.1371/journal.pone.0019026
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- Publication type:
- Article
The Native Copper- and Zinc- Binding Protein Metallothionein Blocks Copper-Mediated Aβ Aggregation and Toxicity in Rat Cortical Neurons.
- Published in:
- PLoS ONE, 2010, v. 5, n. 8, p. 1, doi. 10.1371/journal.pone.0012030
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- Publication type:
- Article
Neuronal growth-inhibitory factor (metallothionein-3): evaluation of the biological function of growth-inhibitory factor in the injured and neurodegenerative brain.
- Published in:
- FEBS Journal, 2010, v. 277, n. 14, p. 2931, doi. 10.1111/j.1742-4658.2010.07718.x
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- Publication type:
- Article