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Antisense-Oligonucleotide Mediated Exon Skipping in Activin-Receptor-Like Kinase 2: Inhibiting the Receptor That Is Overactive in Fibrodysplasia Ossificans Progressiva.
- Published in:
- PLoS ONE, 2013, v. 8, n. 7, p. 1, doi. 10.1371/journal.pone.0069096
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- Article
Lack of Tgfbr1 and Acvr1b synergistically stimulates myofibre hypertrophy and accelerates muscle regeneration.
- Published in:
- eLife, 2022, p. 1, doi. 10.7554/eLife.77610
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- Article
Dual exon skipping in myostatin and dystrophin for Duchenne muscular dystrophy.
- Published in:
- BMC Medical Genomics, 2011, v. 4, n. 1, p. 36, doi. 10.1186/1755-8794-4-36
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- Article
New function of the myostatin/activin type I receptor (ALK4) as a mediator of muscle atrophy and muscle regeneration.
- Published in:
- FASEB Journal, 2017, v. 31, n. 1, p. 238, doi. 10.1096/fj.201600675r
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- Article
New function of the myostatin/activin type I receptor (ALK4) as a mediator of muscle atrophy and muscle regeneration.
- Published in:
- FASEB Journal, 2017, v. 31, n. 1, p. 238, doi. 10.1096/fj.201600675R
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- Publication type:
- Article
Cell-type specific regulation of myostatin signaling.
- Published in:
- FASEB Journal, 2012, v. 26, n. 4, p. 1462, doi. 10.1096/fj.11-191189
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- Article
TGF-β Regulates Collagen Type I Expression in Myoblasts and Myotubes via Transient Ctgf and Fgf-2 Expression.
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- Cells (2073-4409), 2020, v. 9, n. 2, p. 375, doi. 10.3390/cells9020375
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- Article
ORIGINAL ARTICLE Vasopressin-induced vasoconstriction is dependent on MAPK<sup>erk1/2</sup> phosphorylation.
- Published in:
- Fundamental & Clinical Pharmacology, 2004, v. 18, n. 1, p. 45, doi. 10.1046/j.1472-8206.2003.00221.x
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- Article
TBX3 and its splice variant TBX3 + exon 2a are functionally similar.
- Published in:
- Pigment Cell & Melanoma Research, 2008, v. 21, n. 3, p. 379, doi. 10.1111/j.1755-148X.2008.00461.x
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- Article
Novel in vivo zebrafish model and beta cell reporters to address whether exocrine malfunction leads to beta cell stress.
- Published in:
- Nederlands Tijdschrift voor Diabetologie (NTvD), 2023, v. 21, n. 4, p. 28
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- Article