Found: 7
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UBR5 metabolically reprograms nasopharyngeal carcinoma cells to promote glycolysis and M2 polarization via SPLUNC1 signaling.
- Published in:
- NPJ Precision Oncology, 2024, v. 8, n. 1, p. 1, doi. 10.1038/s41698-024-00747-y
- By:
- Publication type:
- Article
Metabolic reprogramming and immune evasion: the interplay in the tumor microenvironment.
- Published in:
- Biomarker Research, 2024, v. 12, n. 1, p. 1, doi. 10.1186/s40364-024-00646-1
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- Publication type:
- Article
Nasopharyngeal carcinoma: current views on the tumor microenvironment's impact on drug resistance and clinical outcomes.
- Published in:
- Molecular Cancer, 2024, v. 23, n. 1, p. 1, doi. 10.1186/s12943-023-01928-2
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- Publication type:
- Article
Cancer-associated fibroblasts secrete FGF5 to inhibit ferroptosis to decrease cisplatin sensitivity in nasopharyngeal carcinoma through binding to FGFR2.
- Published in:
- Cell Death & Disease, 2024, v. 15, n. 4, p. 1, doi. 10.1038/s41419-024-06671-0
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- Publication type:
- Article
USP7 inhibits the progression of nasopharyngeal carcinoma via promoting SPLUNC1-mediated M1 macrophage polarization through TRIM24.
- Published in:
- Cell Death & Disease, 2023, v. 14, n. 12, p. 1, doi. 10.1038/s41419-023-06368-w
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- Publication type:
- Article
Pyroptosis in health and disease: mechanisms, regulation and clinical perspective.
- Published in:
- Signal Transduction & Targeted Therapy, 2024, v. 9, n. 1, p. 1, doi. 10.1038/s41392-024-01958-2
- By:
- Publication type:
- Article
SPLUNC1 regulates LPS-induced progression of nasopharyngeal carcinoma and proliferation of myeloid-derived suppressor cells.
- Published in:
- 2022
- By:
- Publication type:
- journal article