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Defective cAMP Generation Underlies the Sensitivity of CNS Neurons to Neurofibromatosis-1 Heterozygosity.
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- Journal of Neuroscience, 2010, v. 30, n. 16, p. 5579, doi. 10.1523/JNEUROSCI.3994-09.2010
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- Article
Conditional KIAA1549:BRAF mice reveal brain region- and cell type-specific effects.
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- Genesis: The Journal of Genetics & Development, 2013, v. 51, n. 10, p. 708, doi. 10.1002/dvg.22415
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- Article
Reply.
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- Annals of Neurology, 2014, v. 75, n. 5, p. 800, doi. 10.1002/ana.24156
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- Article
Sex Is a major determinant of neuronal dysfunction in neurofibromatosis type 1.
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- Annals of Neurology, 2014, v. 75, n. 2, p. 309, doi. 10.1002/ana.24093
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- Article
Reduced microglial CX3CR1 expression delays neurofibromatosis-1 glioma formation.
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- Annals of Neurology, 2013, v. 73, n. 2, p. 303, doi. 10.1002/ana.23813
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- Article
Microarray analyses reveal regional astrocyte heterogeneity with implications for neurofibromatosis type 1 (NF1)-regulated glial proliferation.
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- Glia, 2009, v. 57, n. 11, p. 1239, doi. 10.1002/glia.20845
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- Article
NF1 germline mutation differentially dictates optic glioma formation and growth in neurofibromatosis-1.
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- Human Molecular Genetics, 2016, v. 25, n. 9, p. 1703, doi. 10.1093/hmg/ddw039
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- Article
Akt- or MEK-mediated mTOR inhibition suppresses Nf1 optic glioma growth.
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- Neuro-Oncology, 2015, v. 17, n. 6, p. 843, doi. 10.1093/neuonc/nou329
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- Article
The impact of coexisting genetic mutations on murine optic glioma biology.
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- Neuro-Oncology, 2015, v. 17, n. 5, p. 670, doi. 10.1093/neuonc/nou287
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- Article