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Yersinia enterocolitica exploits different pathways to accomplish adhesion and toxin injection into host cells.
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- Cellular Microbiology, 2015, v. 17, n. 8, p. 1179, doi. 10.1111/cmi.12429
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- Article
Haemorrhagic toxin and lethal toxin from C lostridium sordellii strain vpi9048: molecular characterization and comparative analysis of substrate specificity of the large clostridial glucosylating toxins.
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- Cellular Microbiology, 2014, v. 16, n. 11, p. 1706, doi. 10.1111/cmi.12321
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- Article
Pyknotic cell death induced by C lostridium difficile TcdB: chromatin condensation and nuclear blister are induced independently of the glucosyltransferase activity.
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- Cellular Microbiology, 2014, v. 16, n. 11, p. 1678, doi. 10.1111/cmi.12317
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- Article
Role of the small Rho GTPases Rac1 and Cdc42 in host cell invasion of Campylobacter jejuni.
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- Cellular Microbiology, 2007, v. 9, n. 10, p. 2431, doi. 10.1111/j.1462-5822.2007.00971.x
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- Article
Clostridium n ovyi's Alpha-Toxin Changes Proteome and Phosphoproteome of HEp-2 Cells.
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- International Journal of Molecular Sciences, 2022, v. 23, n. 17, p. 9939, doi. 10.3390/ijms23179939
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- Article
Activation of Focal Adhesion Kinase Restores Simulated Microgravity-Induced Inhibition of Osteoblast Differentiation via Wnt/Β-Catenin Pathway.
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- International Journal of Molecular Sciences, 2022, v. 23, n. 10, p. 5593, doi. 10.3390/ijms23105593
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- Article
Simulated Microgravity Reduces Focal Adhesions and Alters Cytoskeleton and Nuclear Positioning Leading to Enhanced Apoptosis via Suppressing FAK/RhoA-Mediated mTORC1/NF-κB and ERK1/2 Pathways.
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- International Journal of Molecular Sciences, 2018, v. 19, n. 7, p. 1994, doi. 10.3390/ijms19071994
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- Article
Activation of RhoB in simvastatin-induced apoptosis of differentiated human skeletal muscle cells.
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- BMC Pharmacology, 2007, v. 7, p. 1, doi. 10.1186/1471-2210-7-S2-A16
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- Article
Impaired glyoxalase activity is associated with reduced expression of neurotrophic factors and pro-inflammatory processes in diabetic skin cells.
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- Experimental Dermatology, 2017, v. 26, n. 1, p. 44, doi. 10.1111/exd.13118
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- Article
UV radiation induces CXCL5 expression in human skin.
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- Experimental Dermatology, 2015, v. 24, n. 4, p. 309, doi. 10.1111/exd.12652
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- Article
The Essential Role of Rac1 Glucosylation in Clostridioides difficile Toxin B-Induced Arrest of G1-S Transition.
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- Frontiers in Microbiology, 2022, v. 13, p. 1, doi. 10.3389/fmicb.2022.846215
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- Article
Low Density Lipoprotein Receptor-Related Protein-1 (LRP1) Is Involved in the Uptake of Clostridioides difficile Toxin A and Serves as an Internalizing Receptor.
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- Frontiers in Cellular & Infection Microbiology, 2020, v. 10, p. N.PAG, doi. 10.3389/fcimb.2020.565465
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- Article
Increased Cell-Matrix Adhesion upon Constitutive Activation of Rho Proteins by Cytotoxic Necrotizing Factors from E. coli and Y. pseudotuberculosis.
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- Journal of Signal Transduction, 2012, p. 1, doi. 10.1155/2012/570183
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- Article
Actin Re-Organization Induced by Chlamydia trachomatis Serovar D - Evidence for a Critical Role of the Effector Protein CT166 Targeting Rac.
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- PLoS ONE, 2010, v. 5, n. 3, p. 1, doi. 10.1371/journal.pone.0009887
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- Article
Quantification of small GTPase glucosylation by clostridial glucosylating toxins using multiplexed MRM analysis.
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- Proteomics, 2017, v. 17, n. 9, p. n/a, doi. 10.1002/pmic.201700016
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- Article
Inhibition of macrophage migration by C. botulinum exoenzyme C3.
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- Naunyn-Schmiedeberg's Archives of Pharmacology, 2012, v. 385, n. 9, p. 883, doi. 10.1007/s00210-012-0764-9
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- Article
Difference in the biological effects of Clostridium difficile toxin B in proliferating and non-proliferating cells.
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- Naunyn-Schmiedeberg's Archives of Pharmacology, 2011, v. 383, n. 3, p. 275, doi. 10.1007/s00210-010-0595-5
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- Article
Distinct biological activities of C3 and ADP-ribosyltransferase-deficient C3-E174Q.
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- FEBS Journal, 2012, v. 279, n. 15, p. 2657, doi. 10.1111/j.1742-4658.2012.08645.x
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- Article
The Cytotoxic Necrotizing Factor of <i>Yersinia pseudotuberculosis</i> (CNF<sub>Y</sub>) Enhances Inflammation and Yop Delivery during Infection by Activation of Rho GTPases.
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- PLoS Pathogens, 2013, v. 9, n. 11, p. 1, doi. 10.1371/journal.ppat.1003746
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- Article
Expression and (Lacking) Internalization of the Cell Surface Receptors of <italic>Clostridioides difficile</italic> Toxin B.
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- Frontiers in Microbiology, 2018, p. N.PAG, doi. 10.3389/fmicb.2018.01483
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- Article
Role of p38<sub>alpha/beta</sub> MAP Kinase in Cell Susceptibility to Clostridium sordellii Lethal Toxin and Clostridium difficile Toxin B.
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- Toxins, 2017, v. 9, n. 1, p. 2, doi. 10.3390/toxins9010002
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Metal Ion Activation of Clostridium sordellii Lethal Toxin and Clostridium difficile Toxin B.
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- Toxins, 2016, v. 8, n. 4, p. 1, doi. 10.3390/toxins8040109
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DXD Motif-Dependent and -Independent Effects of the Chlamydia trachomatis Cytotoxin CT166.
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- Toxins, 2015, v. 7, n. 2, p. 621, doi. 10.3390/toxins7020621
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- Article
Difference in F-Actin Depolymerization Induced by Toxin B from the Clostridium difficile Strain VPI 10463 and Toxin B from the Variant Clostridium difficile Serotype F Strain 1470.
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- Toxins, 2013, v. 5, n. 1, p. 106, doi. 10.3390/toxins5010106
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- Article
Evaluation of Immunomodulatory Responses and Changed Wound Healing in Type 2 Diabetes—A Study Exploiting Dermal Fibroblasts from Diabetic and Non-Diabetic Human Donors.
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- Cells (2073-4409), 2021, v. 10, n. 11, p. 2931, doi. 10.3390/cells10112931
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- Article
Cytoprotective effect of the small GTPase RhoB expressed upon treatment of fibroblasts with the Ras-glucosylating Clostridium sordellii lethal toxin
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- FEBS Letters, 2012, v. 586, n. 20, p. 3665, doi. 10.1016/j.febslet.2012.08.024
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- Article
Distinct kinetics of (H/K/N)Ras glucosylation and Rac1 glucosylation catalysed by Clostridium sordellii lethal toxin
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- FEBS Letters, 2009, v. 583, n. 19, p. 3133, doi. 10.1016/j.febslet.2009.09.006
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- Article
Prevention of the cytopathic effect induced by Clostridium difficile Toxin B by active Rac1
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- FEBS Letters, 2008, v. 582, n. 27, p. 3751, doi. 10.1016/j.febslet.2008.10.003
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- Article
Clostridium difficile toxin A-induced apoptosis is p53-independent but depends on glucosylation of Rho GTPases.
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- Apoptosis, 2007, v. 12, n. 8, p. 1443, doi. 10.1007/s10495-007-0074-8
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- Article
Rac1 and Rho contribute to the migratory and invasive phenotype associated with somatic E-cadherin mutation.
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- Human Molecular Genetics, 2009, v. 18, n. 19, p. 3632, doi. 10.1093/hmg/ddp312
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- Article
Cellular stability of Rho-GTPases glucosylated by Clostridium difficile toxin B
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- FEBS Letters, 2006, v. 580, n. 14, p. 3565, doi. 10.1016/j.febslet.2006.04.100
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- Article
Protection from Clostridium difficile toxin B-catalysed Rac1/Cdc42 glucosylation by tauroursodeoxycholic acid-induced Rac1/Cdc42 phosphorylation.
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- Biological Chemistry, 2012, v. 393, n. 1/2, p. 77, doi. 10.1515/BC-2011-198
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- Article