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The Bradycardic Agent Ivabradine Acts as an Atypical Inhibitor of Voltage-Gated Sodium Channels.
- Published in:
- Frontiers in Pharmacology, 2022, v. 13, p. 1, doi. 10.3389/fphar.2022.809802
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- Article
Evidence for a Physiological Role of T-Type Ca Channels in Ventricular Cardiomyocytes of Adult Mice.
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- Membranes, 2022, v. 12, n. 6, p. 566, doi. 10.3390/membranes12060566
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- Article
Voltage-Dependent Sarcolemmal Ion Channel Abnormalities in the Dystrophin-Deficient Heart.
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- International Journal of Molecular Sciences, 2018, v. 19, n. 11, p. 3296, doi. 10.3390/ijms19113296
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- Article
Ivabradine acutely improves cardiac Ca handling and function in a rat model of Duchenne muscular dystrophy.
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- Physiological Reports, 2023, v. 11, n. 7, p. 1, doi. 10.14814/phy2.15664
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- Article
Neuronal nitric oxide synthase regulation of calcium cycling in ventricular cardiomyocytes is independent of Cav1.2 channel modulation under basal conditions.
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- Pflügers Archiv: European Journal of Physiology, 2020, v. 472, n. 1, p. 61, doi. 10.1007/s00424-019-02335-7
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- Article
Oral batyl alcohol supplementation rescues decreased cardiac conduction in ether phospholipid‐deficient mice.
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- Journal of Inherited Metabolic Disease, 2020, v. 43, n. 5, p. 1046, doi. 10.1002/jimd.12264
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- Article
Pharmacological Profile of the Bradycardic Agent Ivabradine on Human Cardiac Ion Channels.
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- Cellular Physiology & Biochemistry (Cell Physiol Biochem Press GmbH & Co. KG), 2019, v. 53, n. 1, p. 36, doi. 10.33594/000000119
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- Article