Found: 10
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Caspase-14 overexpression in hairless mice is not involved in utricle formation.
- Published in:
- Experimental Dermatology, 2013, v. 22, n. 7, p. 484, doi. 10.1111/exd.12165
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- Article
Compound A, a Selective Glucocorticoid Receptor Modulator, Enhances Heat Shock Protein Hsp70 Gene Promoter Activation.
- Published in:
- PLoS ONE, 2013, v. 8, n. 7, p. 1, doi. 10.1371/journal.pone.0069115
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- Article
Optimisation of Weed Control in Organic Processing Spinach (Spinacia oleracea L.): Impacts of Cultivar, Seeding Rate, Plant Spacing and Integrated Weed Management Strategy.
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- Agronomy, 2021, v. 11, n. 1, p. 53, doi. 10.3390/agronomy11010053
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- Article
Functions of the RIP kinase family members in the skin.
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- Cellular & Molecular Life Sciences, 2023, v. 80, n. 10, p. 1, doi. 10.1007/s00018-023-04917-2
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- Article
IDO Expression in Cancer: Different Compartment, Different Functionality?
- Published in:
- Frontiers in Immunology, 2020, v. 11, p. N.PAG, doi. 10.3389/fimmu.2020.531491
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- Article
Filaggrin Degradation by Caspase-14 Is Required for UVB Photoprotection but Does Not Influence Allergic Sensitization in a Mouse Model of Atopic Dermatitis.
- Published in:
- Journal of Investigative Dermatology, 2012, v. 132, n. 12, p. 2857, doi. 10.1038/jid.2012.236
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- Article
Caspase-14 Is Required for Filaggrin Degradation to Natural Moisturizing Factors in the Skin.
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- Journal of Investigative Dermatology, 2011, v. 131, n. 11, p. 2233, doi. 10.1038/jid.2011.153
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- Article
Correction: A20 protects cells from TNF-induced apoptosis through linear ubiquitin-dependent and -independent mechanisms.
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- 2020
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- Correction Notice
A20 protects cells from TNF-induced apoptosis through linear ubiquitin-dependent and -independent mechanisms.
- Published in:
- Cell Death & Disease, 2019, v. 10, n. 10, p. N.PAG, doi. 10.1038/s41419-019-1937-y
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- Article
Keratinocyte-specific ablation of Mcpip1 impairs skin integrity and promotes local and systemic inflammation.
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- Journal of Molecular Medicine, 2019, v. 97, n. 12, p. 1669, doi. 10.1007/s00109-019-01853-2
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- Publication type:
- Article