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Interferon regulatory factor 1 is essential for pathogenic CD8+ T cell migration and retention in the brain during experimental cerebral malaria.
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- Cellular Microbiology, 2018, v. 20, n. 5, p. 1, doi. 10.1111/cmi.12819
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- Article
Adaptive immunity is essential in preventing recrudescence of Plasmodium yoelii malaria parasites after artesunate treatment.
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- Cellular Microbiology, 2017, v. 19, n. 11, p. 1, doi. 10.1111/cmi.12763
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- Article
Spatiotemporal requirements for IRF7 in mediating type I IFN-dependent susceptibility to blood-stage Plasmodium infection.
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- European Journal of Immunology, 2015, v. 45, n. 1, p. 130, doi. 10.1002/eji.201444824
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- Article
CD41 is a reliable identification and activation marker for murine basophils in the steady state and during helminth and malarial infections.
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- European Journal of Immunology, 2014, v. 44, n. 6, p. 1823, doi. 10.1002/eji.201344254
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- Article
Co‐infection with Chikungunya virus alters trafficking of pathogenic CD8<sup>+</sup> T cells into the brain and prevents <italic>Plasmodium</italic>‐induced neuropathology.
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- EMBO Molecular Medicine, 2018, v. 10, n. 1, p. 121, doi. 10.15252/emmm.201707885
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- Article
Brain microvessel cross-presentation is a hallmark of experimental cerebral malaria.
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- EMBO Molecular Medicine, 2013, v. 5, n. 7, p. 916, doi. 10.1002/emmm.201202273
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- Article
Experimental Models to Study the Pathogenesis of Malaria-Associated Acute Respiratory Distress Syndrome.
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- Frontiers in Cellular & Infection Microbiology, 2022, v. 12, p. 1, doi. 10.3389/fcimb.2022.899581
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- Article
Inflammatory Flt3l is essential to mobilize dendritic cells and for T cell responses during Plasmodium infection.
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- Nature Medicine, 2013, v. 19, n. 6, p. 730, doi. 10.1038/nm.3197
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- Article
CD8<sup>+</sup> T Cells and IFN-γ Mediate the Time-Dependent Accumulation of Infected Red Blood Cells in Deep Organs during Experimental Cerebral Malaria.
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- PLoS ONE, 2011, v. 6, n. 4, p. 1, doi. 10.1371/journal.pone.0018720
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- Article
Pathogenic CD8 T cells in experimental cerebral malaria.
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- Seminars in Immunopathology, 2015, v. 37, n. 3, p. 221, doi. 10.1007/s00281-015-0476-6
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- Article
Unraveling the complex interplay: immunopathology and immune evasion strategies of alphaviruses with emphasis on neurological implications.
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- Frontiers in Cellular & Infection Microbiology, 2024, p. 1, doi. 10.3389/fcimb.2024.1421571
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- Article
Author Correction: Lung endothelial cell antigen cross-presentation to CD8<sup>+</sup>T cells drives malaria-associated lung injury.
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- 2019
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- Correction Notice
Lung endothelial cell antigen cross-presentation to CD8<sup>+</sup>T cells drives malaria-associated lung injury.
- Published in:
- Nature Communications, 2019, v. 10, n. 1, p. N.PAG, doi. 10.1038/s41467-019-12017-8
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- Article
The CTLA-4 and PD-1/PD-L1 Inhibitory Pathways Independently Regulate Host Resistance to Plasmodium-induced Acute Immune Pathology.
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- PLoS Pathogens, 2012, v. 8, n. 2, p. 1, doi. 10.1371/journal.ppat.1002504
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- Article
Interferons and Interferon Regulatory Factors in Malaria.
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- Mediators of Inflammation, 2014, v. 2014, p. 1, doi. 10.1155/2014/243713
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- Article
Plasmodium co-infection protects against chikungunya virus-induced pathologies.
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- Nature Communications, 2018, v. 9, n. 1, p. 1, doi. 10.1038/s41467-018-06227-9
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- Article
Adaptive Immunity against Leishmania Nucleoside Hydrolase Maps Its C-Terminal Domain as the Target of the CD4+ T Cell–Driven Protective Response.
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- PLoS Neglected Tropical Diseases, 2010, v. 4, n. 11, p. 1, doi. 10.1371/journal.pntd.0000866
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- Article
Adaptive Immunity against Leishmania Nucleoside Hydrolase Maps Its C-Terminal Domain as the Target of the CD4+ T Cell-Driven Protective Response.
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- PLoS Neglected Tropical Diseases, 2010, v. 4, n. 11, p. 1, doi. 10.1371/journal.pntd.0000866
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- Article
Measuring antigen presentation in mouse brain endothelial cells ex vivo and in vitro.
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- Nature Protocols, 2015, v. 10, n. 12, p. 2016, doi. 10.1038/nprot.2015.129
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- Article