Found: 14
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In Vivo Stimulation of α- and β-Adrenoceptors in Mice Differentially Alters Small RNA Content of Circulating Extracellular Vesicles.
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- Cells (2073-4409), 2021, v. 10, n. 5, p. 1211, doi. 10.3390/cells10051211
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- Article
Mitochondrial GRK2 is a Novel Regulator of Cardiac Energetics.
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- FASEB Journal, 2022, v. 36, p. N.PAG, doi. 10.1096/fasebj.2022.36.S1.R2991
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- Article
Cardiac Progenitor Cells Engineered With βARKct Have Enhanced β-Adrenergic Tolerance.
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- Molecular Therapy, 2014, v. 22, n. 1, p. 178, doi. 10.1038/mt.2013.200
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- Article
A peptide of the N terminus of GRK5 attenuates pressure-overload hypertrophy and heart failure.
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- Science Signaling, 2021, v. 14, n. 676, p. 1, doi. 10.1126/scisignal.abb5968
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- Article
Restricting mitochondrial GRK2 post-ischemia confers cardioprotection by reducing myocyte death and maintaining glucose oxidation.
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- Science Signaling, 2018, v. 11, n. 560, p. 1, doi. 10.1126/scisignal.aau0144
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- Article
S100A1 Gene Therapy Preserves in Vivo Cardiac Function after Myocardial Infarction
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- Molecular Therapy, 2005, v. 12, n. 6, p. 1120, doi. 10.1016/j.ymthe.2005.08.002
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- Article
FOXD1-dependent MICU1 expression regulates mitochondrial activity and cell differentiation.
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- Nature Communications, 2018, v. 9, n. 1, p. 1, doi. 10.1038/s41467-018-05856-4
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- Article
Inhibition of Fas-Associated Death Domain-Containing Protein (FADD) Protects against Myocardial Ischemia/Reperfusion Injury in a Heart Failure Mouse Model.
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- PLoS ONE, 2013, v. 8, n. 9, p. 1, doi. 10.1371/journal.pone.0073537
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- Article
Myocardial Ablation of G Protein–Coupled Receptor Kinase 2 (GRK2) Decreases Ischemia/Reperfusion Injury through an Anti-Intrinsic Apoptotic Pathway.
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- PLoS ONE, 2013, v. 8, n. 6, p. 1, doi. 10.1371/journal.pone.0066234
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- Article
Nuclear Translocation of Cardiac G Protein-Coupled Receptor Kinase 5 Downstream of Select Gq-Activating Hypertrophic Ligands Is a Calmodulin-Dependent Process.
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- PLoS ONE, 2013, v. 8, n. 3, p. 1, doi. 10.1371/journal.pone.0057324
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- Article
Paroxetine-mediated GRK2 inhibition reverses cardiac dysfunction and remodeling after myocardial infarction.
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- Science Translational Medicine, 2015, v. 7, n. 277, p. 1, doi. 10.1126/scitranslmed.aaa0154
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- Article
A peptide of the RGS domain of GRK2 binds and inhibits Gα<sub>q</sub> to suppress pathological cardiac hypertrophy and dysfunction.
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- Science Signaling, 2016, v. 9, n. 420, p. 1, doi. 10.1126/scisignal.aae0549
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- Article
Convergence of G Protein-Coupled Receptor and S-Nitrosylation Signaling Determines the Outcome to Cardiac Ischemic Injury.
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- Science Signaling, 2013, v. 6, n. 299, p. 1, doi. 10.1126/scisignal.2004225
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- Article
Dynamic Changes in Lymphocyte GRK2 Levels in Cardiac Transplant Patients: A Biomarker for Left Ventricular Function.
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- CTS: Clinical & Translational Science, 2010, v. 3, n. 1, p. 14, doi. 10.1111/j.1752-8062.2010.00176.x
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- Article