Found: 13
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In Vivo Inhibition of miR-34a Modestly Limits Cardiac Enlargement and Fibrosis in a Mouse Model with Established Type 1 Diabetes-Induced Cardiomyopathy, but Does Not Improve Diastolic Function.
- Published in:
- Cells (2073-4409), 2022, v. 11, n. 19, p. 3117, doi. 10.3390/cells11193117
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- Publication type:
- Article
Therapeutic silencing of miR-652 restores heart function and attenuates adverse remodeling in a setting of established pathological hypertrophy.
- Published in:
- FASEB Journal, 2014, v. 28, n. 12, p. 5097, doi. 10.1096/fj.14-253856
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- Publication type:
- Article
Micro RNAs differentially regulated in cardiac and skeletal muscle in health and disease: Potential drug targets?
- Published in:
- Clinical & Experimental Pharmacology & Physiology, 2014, v. 41, n. 9, p. 727, doi. 10.1111/1440-1681.12281
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- Publication type:
- Article
The small-molecule BGP-15 protects against heart failure and atrial fibrillation in mice.
- Published in:
- Nature Communications, 2014, v. 5, n. 12, p. 5705, doi. 10.1038/ncomms6705
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- Publication type:
- Article
Smad7 gene delivery prevents muscle wasting associated with cancer cachexia in mice.
- Published in:
- Science Translational Medicine, 2016, v. 8, n. 348, p. 1, doi. 10.1126/scitranslmed.aac4976
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- Publication type:
- Article
Silencing of miR-34a Attenuates Cardiac Dysfunction in a Setting of Moderate, but Not Severe, Hypertrophic Cardiomyopathy.
- Published in:
- PLoS ONE, 2014, v. 9, n. 2, p. 1, doi. 10.1371/journal.pone.0090337
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- Publication type:
- Article
Inhibition of miR-154 Protects Against Cardiac Dysfunction and Fibrosis in a Mouse Model of Pressure Overload.
- Published in:
- Scientific Reports, 2016, p. 22442, doi. 10.1038/srep22442
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- Publication type:
- Article
Sex differences in response to miRNA-34a therapy in mouse models of cardiac disease: identification of sex-, disease- and treatment-regulated miRNAs.
- Published in:
- Journal of Physiology, 2016, v. 594, n. 20, p. 5959, doi. 10.1113/JP272512
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- Publication type:
- Article
A gene therapy targeting medium-chain acyl-CoA dehydrogenase (MCAD) did not protect against diabetes-induced cardiac pathology.
- Published in:
- Journal of Molecular Medicine, 2024, v. 102, n. 1, p. 95, doi. 10.1007/s00109-023-02397-2
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- Publication type:
- Article
β-Adrenergic Stimulation Induces Histone Deacetylase 5 (HDAC5) Nuclear Accumulation in Cardiomyocytes by B55α-PP2A-Mediated Dephosphorylation.
- Published in:
- 2017
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- Publication type:
- journal article
Gene delivery of medium chain acyl-coenzyme A dehydrogenase induces physiological cardiac hypertrophy and protects against pathological remodelling.
- Published in:
- Clinical Science, 2018, v. 132, n. 3, p. 381, doi. 10.1042/CS20171269
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- Publication type:
- Article
Old Drug, New Trick: Tilorone, a Broad-Spectrum Antiviral Drug as a Potential Anti-Fibrotic Therapeutic for the Diseased Heart.
- Published in:
- Pharmaceuticals (14248247), 2021, v. 14, n. 3, p. 263, doi. 10.3390/ph14030263
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- Publication type:
- Article
Long-Term Overexpression of Hsp70 Does Not Protect against Cardiac Dysfunction and Adverse Remodeling in a MURC Transgenic Mouse Model with Chronic Heart Failure and Atrial Fibrillation.
- Published in:
- PLoS ONE, 2015, v. 10, n. 12, p. 1, doi. 10.1371/journal.pone.0145173
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- Publication type:
- Article