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Synaptic Plasticity and Oscillations in Alzheimer's Disease: A Complex Picture of a Multifaceted Disease.
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- Frontiers in Molecular Neuroscience, 2021, v. 14, p. 1, doi. 10.3389/fnmol.2021.696476
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- Article
Pre-synaptic kainate receptor-mediated facilitation of glutamate release involves PKA and Ca<sup>2+</sup>-calmodulin at thalamocortical synapses.
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- Journal of Neurochemistry, 2013, v. 126, n. 5, p. 565, doi. 10.1111/jnc.12310
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- Article
Presynaptic kainate receptor-mediated facilitation of glutamate release involves Ca<sup>2+</sup>-calmodulin at mossy fiber-CA3 synapses.
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- Journal of Neurochemistry, 2012, v. 122, n. 5, p. 891, doi. 10.1111/j.1471-4159.2012.07844.x
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- Article
Kainate receptor-mediated depression of glutamatergic transmission involving protein kinase A in the lateral amygdala.
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- Journal of Neurochemistry, 2012, v. 121, n. 1, p. 36, doi. 10.1111/j.1471-4159.2012.07665.x
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- Article
Receptor–Receptor Interactions in Multiple 5-HT1A Heteroreceptor Complexes in Raphe-Hippocampal 5-HT Transmission and Their Relevance for Depression and Its Treatment.
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- Molecules, 2018, v. 23, n. 6, p. 1341, doi. 10.3390/molecules23061341
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- Article
Targeting galectin-3 to counteract spike-phase uncoupling of fast-spiking interneurons to gamma oscillations in Alzheimer's disease.
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- Translational Neurodegeneration, 2023, v. 12, n. 1, p. 1, doi. 10.1186/s40035-023-00338-0
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- Article
Bri2 BRICHOS client specificity and chaperone activity are governed by assembly state.
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- Nature Communications, 2017, v. 8, n. 1, p. 1, doi. 10.1038/s41467-017-02056-4
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- Article
Role of Group I Metabotropic Glutamate Receptors in Spike Timing-Dependent Plasticity.
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- International Journal of Molecular Sciences, 2022, v. 23, n. 14, p. 7807, doi. 10.3390/ijms23147807
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- Article
Capsaicin-Induced Impairment of Functional Network Dynamics in Mouse Hippocampus via a TrpV1 Receptor-Independent Pathway: Putative Involvement of Na+/K+-ATPase.
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- Molecular Neurobiology, 2020, v. 57, n. 2, p. 1170, doi. 10.1007/s12035-019-01779-3
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- Article
Adenosine Receptor-Mediated Developmental Loss of Spike Timing-Dependent Depression in the Hippocampus.
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- Cerebral Cortex, 2019, v. 29, n. 8, p. 3266, doi. 10.1093/cercor/bhy194
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- Article
Presynaptic Spike Timing-Dependent Long-Term Depression in the Mouse Hippocampus.
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- Cerebral Cortex, 2016, v. 26, n. 8, p. 3637, doi. 10.1093/cercor/bhw172
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- Article
Astrocyte-mediated switch in spike timing-dependent plasticity during hippocampal development.
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- Nature Communications, 2020, v. 11, n. 1, p. N.PAG, doi. 10.1038/s41467-020-18024-4
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- Article
Kainate receptors in the CA2 region of the hippocampus.
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- Neural Regeneration Research, 2023, v. 18, n. 2, p. 320, doi. 10.4103/1673-5374.343912
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- Article
Targeting galectin-3 to counteract spike-phase uncoupling of fast-spiking interneurons to gamma oscillations in Alzheimer's disease.
- Published in:
- Translational Neurodegeneration, 2023, v. 12, n. 1, p. 1, doi. 10.1186/s40035-023-00338-0
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- Article
Augmentation of Bri2 molecular chaperone activity against amyloid-β reduces neurotoxicity in mouse hippocampus in vitro.
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- Communications Biology, 2020, v. 3, n. 1, p. 1, doi. 10.1038/s42003-020-0757-z
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- Article
Modulation of Kv3.1/Kv3.2 promotes gamma oscillations by rescuing Aβ‐induced desynchronization of fast‐spiking interneuron firing in an AD mouse model in vitro.
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- Journal of Physiology, 2020, v. 598, n. 17, p. 3711, doi. 10.1113/JP279718
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- Article
Disturbances in the FGFR1-5-HT1A Heteroreceptor Complexes in the Raphe-Hippocampal 5-HT System Develop in a Genetic Rat Model of Depression.
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- Frontiers in Cellular Neuroscience, 2017, p. 1, doi. 10.3389/fncel.2017.00309
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Ablation of p75NTR signaling strengthens gamma–theta rhythm interaction and counteracts Aβ-induced degradation of neuronal dynamics in mouse hippocampus in vitro.
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- Translational Psychiatry, 2021, v. 11, n. 1, p. 1, doi. 10.1038/s41398-021-01332-8
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A novel therapeutic approach to treat Alzheimer's disease: The brain‐specific signal peptide peptidase‐like 2b (SPPL2b).
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2021, v. 17, n. 3, p. 1, doi. 10.1002/alz.052933
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- Article