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Intermittent hypoxia reverses the diurnal glucose rhythm and causes pancreatic β-cell replication in mice.
- Published in:
- Journal of Physiology, 2008, v. 586, n. 3, p. 899, doi. 10.1113/jphysiol.2007.143586
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- Publication type:
- Article
Overnutrition in the early postnatal period influences lifetime metabolic risk: Evidence for impact on pancreatic β-cell mass and function.
- Published in:
- Journal of Diabetes Investigation, 2024, v. 15, n. 3, p. 263, doi. 10.1111/jdi.14136
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- Publication type:
- Article
The Islet Estrogen Receptor-α Is Induced by Hyperglycemia and Protects Against Oxidative Stress-Induced Insulin-Deficient Diabetes.
- Published in:
- PLoS ONE, 2014, v. 9, n. 2, p. 1, doi. 10.1371/journal.pone.0087941
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- Publication type:
- Article
Exogenous Glucose Administration Impairs Glucose Tolerance and Pancreatic Insulin Secretion during Acute Sepsis in Non-Diabetic Mice.
- Published in:
- PLoS ONE, 2013, v. 8, n. 6, p. 1, doi. 10.1371/journal.pone.0067716
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- Publication type:
- Article
Ergocalciferol in New-onset Type 1 Diabetes: A Randomized Controlled Trial.
- Published in:
- Journal of the Endocrine Society, 2022, v. 6, n. 1, p. 1, doi. 10.1210/jendso/bvab179
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- Publication type:
- Article
Continuous glucose monitoring reduces pubertal hyperglycemia of type 1 diabetes.
- Published in:
- Journal of Pediatric Endocrinology & Metabolism, 2020, v. 33, n. 7, p. 865, doi. 10.1515/jpem-2020-0057
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- Publication type:
- Article
Partial clinical remission in type 1 diabetes: a comparison of the accuracy of total daily dose of insulin of <0.3 units/kg/day to the gold standard insulin-dose adjusted hemoglobin A<sub>1c</sub> of ≤9 for the detection of partial clinical remission
- Published in:
- Journal of Pediatric Endocrinology & Metabolism, 2017, v. 30, n. 8, p. 823, doi. 10.1515/jpem-2017-0019
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- Publication type:
- Article
Endoplasmic Reticulum Stress Induced Proliferation Remains Intact in Aging Mouse β-Cells.
- Published in:
- Frontiers in Endocrinology, 2021, v. 12, p. 1, doi. 10.3389/fendo.2021.734079
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- Publication type:
- Article
Insulin demand regulates β cell number via the unfolded protein response.
- Published in:
- 2015
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- Publication type:
- journal article
Early initiation of low-level parenteral dextrose induces an accelerated diabetic phenotype in septic C57BL/6J mice.
- Published in:
- Applied Physiology, Nutrition & Metabolism, 2016, v. 41, n. 1, p. 12, doi. 10.1139/apnm-2015-0213
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- Publication type:
- Article
Intermittent Hypoxia Causes Insulin Resistance in Lean Mice Independent of Autonomic Activity.
- Published in:
- American Journal of Respiratory & Critical Care Medicine, 2007, v. 175, n. 8, p. 851, doi. 10.1164/rccm.200610-1527oc
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- Publication type:
- Article
ANRIL: A lncRNA at the CDKN2A/B Locus With Roles in Cancer and Metabolic Disease.
- Published in:
- Frontiers in Endocrinology, 2018, p. N.PAG, doi. 10.3389/fendo.2018.00405
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- Publication type:
- Article
Hypusine biosynthesis in β cells links polyamine metabolism to facultative cellular proliferation to maintain glucose homeostasis.
- Published in:
- Science Signaling, 2019, v. 12, n. 610, p. 1, doi. 10.1126/scisignal.aax0715
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- Publication type:
- Article
Glucose infusion in mice: a new model to induce beta-cell replication.
- Published in:
- 2007
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- Publication type:
- journal article
Glucose Infusion in Mice.
- Published in:
- Diabetes, 2007, v. 56, n. 7, p. 1792, doi. 10.2337/db06-1513
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- Publication type:
- Article
Cover Picture: Elucidation of the Physicochemical Properties Ruling the Colloidal Stability of Iron Oxide Nanoparticles under Physiological Conditions (ChemNanoMat 3/2017).
- Published in:
- 2017
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- Publication type:
- Cover Art
Elucidation of the Physicochemical Properties Ruling the Colloidal Stability of Iron Oxide Nanoparticles under Physiological Conditions.
- Published in:
- ChemNanoMat, 2017, v. 3, n. 3, p. 183, doi. 10.1002/cnma.201600333
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- Publication type:
- Article
Weight Loss Outcomes With Telemedicine During COVID-19.
- Published in:
- Frontiers in Endocrinology, 2022, v. 13, p. 1, doi. 10.3389/fendo.2022.793290
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- Publication type:
- Article
Children with type 1 diabetes who experienced a honeymoon phase had significantly lower LDL cholesterol 5 years after diagnosis.
- Published in:
- PLoS ONE, 2018, v. 13, n. 5, p. 1, doi. 10.1371/journal.pone.0196912
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- Publication type:
- Article
Career Advancement for Women in Diabetes-Related Research: Developing and Retaining Female Talent.
- Published in:
- 2021
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- Publication type:
- journal article
The Independent Risk of Obesity and Diabetes and Their Interaction in COVID-19: A Retrospective Cohort Study.
- Published in:
- 2021
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- Publication type:
- journal article
Simultaneous Measurement of Insulin Sensitivity, Insulin Secretion, and the Disposition Index in Conscious Unhandled Mice.
- Published in:
- Obesity (19307381), 2012, v. 20, n. 7, p. 1403, doi. 10.1038/oby.2012.36
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- Publication type:
- Article
A predictive model for lack of partial clinical remission in new-onset pediatric type 1 diabetes.
- Published in:
- PLoS ONE, 2017, v. 12, n. 5, p. 1, doi. 10.1371/journal.pone.0176860
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- Publication type:
- Article
Living Dangerously: Protective and Harmful ER Stress Responses in Pancreatic β-Cells.
- Published in:
- 2021
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- Publication type:
- journal article
Career Advancement for Women in Diabetes-Related Research: Developing and Retaining Female Talent.
- Published in:
- 2021
- By:
- Publication type:
- journal article
51-OR: Loss of Critical ER Chaperone GRP78 Leads to Beta-Cell Death by a JNK Dependent Mechanism.
- Published in:
- Diabetes, 2020, v. 69, p. N.PAG, doi. 10.2337/db20-51-OR
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- Publication type:
- Article
261-OR: CDK4-R24C Compensates for IRS2 Deficiency by Enhancing Beta-Cell Proliferation and Restoring Signaling through the AKT-Foxo1-PDX1 Pathway.
- Published in:
- Diabetes, 2020, v. 69, p. N.PAG, doi. 10.2337/db20-261-OR
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- Publication type:
- Article
322-LB: Balancing Act: Atf6alpha Plays Positive and Negative Roles in the Pancreatic Beta-Cell Stress Response.
- Published in:
- Diabetes, 2019, v. 68, p. N.PAG, doi. 10.2337/db19-322-LB
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- Publication type:
- Article
DNA Damage Does Not Cause BrdU Labeling of Mouse or Human β-Cells.
- Published in:
- 2019
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- Publication type:
- journal article
T2D Genome-Wide Association Study Risk SNPs Impact Locus Gene Expression and Proliferation in Human Islets.
- Published in:
- 2018
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- Publication type:
- journal article
PKCζ Is Essential for Pancreatic β-Cell Replication During Insulin Resistance by Regulating mTOR and Cyclin-D2.
- Published in:
- 2016
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- Publication type:
- journal article
Glucose Induces Mouse β-Cell Proliferation via IRS2, MTOR, and Cyclin D2 but Not the Insulin Receptor.
- Published in:
- 2016
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- Publication type:
- journal article
ChREBP mediates glucose-stimulated pancreatic β-cell proliferation.
- Published in:
- 2012
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- Publication type:
- journal article
ChREBP Mediates Glucose-Stimulated Pancreatic β-Cell Proliferation.
- Published in:
- Diabetes, 2012, v. 61, n. 8, p. 2004, doi. 10.2337/db11-0802
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- Publication type:
- Article
Loss of HGF/c-Met signaling in pancreatic β-cells leads to incomplete maternal β-cell adaptation and gestational diabetes mellitus.
- Published in:
- 2012
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- Publication type:
- journal article
Loss of HGF/c-Met Signaling in Pancreatic β-Cells Leads to Incomplete Maternal β-Cell Adaptation and Gestational Diabetes Mellitus.
- Published in:
- Diabetes, 2012, v. 61, n. 5, p. 1143, doi. 10.2337/db11-1154
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- Publication type:
- Article
Free fatty acids block glucose-induced β-cell proliferation in mice by inducing cell cycle inhibitors p16 and p18.
- Published in:
- 2012
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- Publication type:
- journal article
Free Fatty Acids Block Glucose-Induced β-Cell Proliferation in Mice by Inducing Cell Cycle Inhibitors p16 and p18.
- Published in:
- Diabetes, 2012, v. 61, n. 3, p. 632, doi. 10.2337/db11-0991
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- Publication type:
- Article
Activation of Protein Kinase C-ζ in Pancreatic β-Cells In Vivo Improves Glucose Tolerance and Induces β-Cell Expansion via mTOR Activation.
- Published in:
- Diabetes, 2011, v. 60, n. 10, p. 2546, doi. 10.2337/db10-1783
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- Publication type:
- Article
Disruption of Hepatocyte Growth Factor/c-Met Signaling Enhances Pancreatic β-Cell Death and Accelerates the Onset of Diabetes.
- Published in:
- Diabetes, 2011, v. 60, n. 2, p. 525, doi. 10.2337/db09-1305
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- Publication type:
- Article