Found: 19
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Interneurons, GABA currents, and subunit composition of the GABA<sub>A</sub> receptor in type I and type II cortical dysplasia.
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- Epilepsia (Series 4), 2010, v. 51, p. 166, doi. 10.1111/j.1528-1167.2010.02634.x
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- Article
Comparative study of cellular and synaptic abnormalities in brain tissue samples from pediatric tuberous sclerosis complex and cortical dysplasia type II.
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- Epilepsia (Series 4), 2010, v. 51, p. 160, doi. 10.1111/j.1528-1167.2010.02633.x
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- Article
Immature Neurons and GABA Networks May Contribute to Epileptogenesis in Pediatric Cortical Dysplasia.
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- Epilepsia (Series 4), 2007, v. 48, p. 79, doi. 10.1111/j.1528-1167.2007.01293.x
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Are Cytomegalic Neurons and Balloon Cells Generators of Epileptic Activity in Pediatric Cortical Dysplasia?
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- Epilepsia (Series 4), 2005, v. 46, p. 82, doi. 10.1111/j.1528-1167.2005.01013.x
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Dopamine and Glutamate in Huntington's Disease: A Balancing Act.
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- CNS Neuroscience & Therapeutics, 2010, v. 16, n. 3, p. 163, doi. 10.1111/j.1755-5949.2010.00134.x
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- Article
Alterations in N‐methyl‐D‐aspartate receptor sensitivity and magnesium blockade occur early in development in the R6/2 mouse model of Huntington's disease.
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- Journal of Neuroscience Research, 2005, v. 82, n. 3, p. 377, doi. 10.1002/jnr.20651
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Increased GABAergic function in mouse models of Huntington's disease: Reversal by BDNF.
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- Journal of Neuroscience Research, 2004, v. 78, n. 6, p. 855, doi. 10.1002/jnr.20344
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Morphological and electrophysiological characterization of abnormal cell types in pediatric cortical dysplasia.
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- Journal of Neuroscience Research, 2003, v. 72, n. 4, p. 472, doi. 10.1002/jnr.10604
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Alterations in Cortical Excitation and Inhibition in Genetic Mouse Models of Huntington's Disease.
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- Journal of Neuroscience, 2009, v. 29, n. 33, p. 10371, doi. 10.1523/JNEUROSCI.1592-09.2009
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- Article
Age-Dependent Alterations of Corticostriatal Activity in the YAC128 Mouse Model of Huntington Disease.
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- Journal of Neuroscience, 2009, v. 29, n. 8, p. 2414, doi. 10.1523/JNEUROSCI.5687-08.2009
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- Article
Differential Susceptibility to Excitotoxic Stress in YAC128 Mouse Models of Huntington Disease between Initiation and Progression of Disease.
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- Journal of Neuroscience, 2009, v. 29, n. 7, p. 2193, doi. 10.1523/JNEUROSCI.5473-08.2009
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- Article
Full-Length Human Mutant Huntingtin with a Stable Polyglutamine Repeat Can Elicit Progressive and Selective Neuropathogenesis in BACHD Mice.
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- Journal of Neuroscience, 2008, v. 28, n. 24, p. 6182, doi. 10.1523/JNEUROSCI.0857-08.2008
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- Article
Age-Dependent Alterations of Corticostriatal Activity in the YAC128 Mouse Model of Huntington Disease.
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- Journal of Neuroscience, 2008, p. 2414, doi. 10.1523/JNEUROSCI.5687-08.2009
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- Article
Altered expression of α3-containing GABAA receptors in the neocortex of patients with focal epilepsy.
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- Brain: A Journal of Neurology, 2006, v. 129, n. 12, p. 3277, doi. 10.1093/brain/awl287
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- Article
Dopamine modulation of excitatory currents in the striatum is dictated by the expression of D1 or D2 receptors and modified by endocannabinoids.
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- European Journal of Neuroscience, 2010, v. 31, n. 1, p. 14, doi. 10.1111/j.1460-9568.2009.07047.x
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Differential electrophysiological properties of dopamine D1 and D2 receptor-containing striatal medium-sized spiny neurons.
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- European Journal of Neuroscience, 2008, v. 27, n. 3, p. 671, doi. 10.1111/j.1460-9568.2008.06038.x
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Pathological cell-cell interactions are necessary for striatal pathogenesis in a conditional mouse model of Huntington's disease.
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- Molecular Neurodegeneration, 2007, v. 2, p. 8, doi. 10.1186/1750-1326-2-8
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Multiple Sources of Striatal Inhibition Are Differentially Affected in Huntington's Disease Mouse Models.
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- Journal of Neuroscience, 2013, v. 33, n. 17, p. 7393, doi. 10.1523/JNEUROSCI.2137-12.2013
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Differential Electrophysiological Changes in Striatal Output Neurons in Huntington's Disease.
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- Journal of Neuroscience, 2011, v. 31, n. 4, p. 1170, doi. 10.1523/JNEUROSCI.3539-10.2011
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