Found: 13
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Increased proteinase 3 and neutrophil elastase plasma concentrations are associated with non-alcoholic fatty liver disease (NAFLD) and type 2 diabetes.
- Published in:
- Molecular Medicine, 2019, v. 25, n. 1, p. N.PAG, doi. 10.1186/s10020-019-0084-3
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- Article
Increased proteinase 3 and neutrophil elastase plasma concentrations are associated with non-alcoholic fatty liver disease (NAFLD) and type 2 diabetes.
- Published in:
- Molecular Medicine, 2019, v. 25, n. 1, p. 1, doi. 10.1186/s10020-019-0084-3
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- Article
TET2 rs1548483 SNP Associating with Susceptibility to Molecularly Annotated Polycythemia Vera and Primary Myelofibrosis.
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- Journal of Personalized Medicine, 2020, v. 10, n. 4, p. 259, doi. 10.3390/jpm10040259
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- Article
Mice Deficient in the IL-1β Activation Genes Prtn3, Elane, and Casp1 Are Protected Against the Development of Obesity-Induced NAFLD.
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- Inflammation, 2020, v. 43, n. 3, p. 1054, doi. 10.1007/s10753-020-01190-4
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- Article
Skeletal Abnormalities and VDR1 Gene Polymorphisms in Mucopolysaccharidosis Patients.
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- Pharmacogenomics & Personalized Medicine, 2021, v. 14, p. 349, doi. 10.2147/PGPM.S295241
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- Article
Evaluation of Circulating Chitotriosidase Activity in Children with Obesity.
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- Journal of Clinical Medicine, 2022, v. 11, n. 13, p. 3634, doi. 10.3390/jcm11133634
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- Article
Exploratory Longitudinal Analysis of the Circulating CHIT1 Activity in Pediatric Patients with Obesity.
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- Children, 2023, v. 10, n. 1, p. 124, doi. 10.3390/children10010124
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- Article
Activation of Proteinase 3 Contributes to Nonalcoholic Fatty Liver Disease and Insulin Resistance.
- Published in:
- Molecular Medicine, 2016, v. 22, n. 1, p. 202, doi. 10.2119/molmed.2016.00033
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- Article
Activation of Proteinase 3 Contributes to Nonalcoholic Fatty Liver Disease and Insulin Resistance.
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- Molecular Medicine, 2016, v. 22, p. 202, doi. 10.2119/molmed.2016.00033
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- Publication type:
- Article
46,XX DSD: Developmental, Clinical and Genetic Aspects.
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- Diagnostics (2075-4418), 2021, v. 11, n. 8, p. 1379, doi. 10.3390/diagnostics11081379
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- Article
TGF-β is elevated in hyperuricemic individuals and mediates urate-induced hyperinflammatory phenotype in human mononuclear cells.
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- Arthritis Research & Therapy, 2023, v. 25, n. 1, p. 1, doi. 10.1186/s13075-023-03001-1
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- Article
GWAS-identified hyperuricemia-associated IGF1R variant rs6598541 has a limited role in urate mediated inflammation in human mononuclear cells.
- Published in:
- Scientific Reports, 2024, v. 14, n. 1, p. 1, doi. 10.1038/s41598-024-53209-7
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- Publication type:
- Article
GWAS-identified hyperuricemia-associated IGF1R variant rs6598541 has a limited role in urate mediated inflammation in human mononuclear cells.
- Published in:
- Scientific Reports, 2024, v. 14, n. 1, p. 1, doi. 10.1038/s41598-024-53209-7
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- Publication type:
- Article