Found: 20
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LHX3 Interacts with Inhibitor of Histone Acetyltransferase Complex Subunits LANP and TAF-1β to Modulate Pituitary Gene Regulation.
- Published in:
- PLoS ONE, 2013, v. 8, n. 7, p. 1, doi. 10.1371/journal.pone.0068898
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- Publication type:
- Article
Three novel missense mutations within the LHX4 gene are associated with variable pituitary hormone deficiencies.
- Published in:
- 2008
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- Publication type:
- journal article
Four Novel Mutations of the LHX3 Gene Cause Combined Pituitary Hormone Deficiencies with or without Limited Neck Rotation.
- Published in:
- Journal of Clinical Endocrinology & Metabolism, 2007, v. 92, n. 5, p. 1909, doi. 10.1210/jc.2006-2177
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- Article
A Novel LHX3 Mutation Presenting as Combined Pituitary Hormonal Deficiency.
- Published in:
- Journal of Clinical Endocrinology & Metabolism, 2006, v. 91, n. 3, p. 747, doi. 10.1210/jc.2005-2360
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- Publication type:
- Article
Glucagon-Receptor Signaling Reverses Hepatic Steatosis Independent of Leptin Receptor Expression.
- Published in:
- Endocrinology, 2020, v. 161, n. 1, p. 1, doi. 10.1210/endocr/bqz013
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- Publication type:
- Article
Prolactin Receptor Signaling Regulates a Pregnancy-Specific Transcriptional Program in Mouse Islets.
- Published in:
- Endocrinology, 2019, v. 160, n. 5, p. 1150, doi. 10.1210/en.2018-00991
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- Publication type:
- Article
The Ldb1 transcriptional co‐regulator is required for establishment and maintenance of the pancreatic endocrine lineage.
- Published in:
- FASEB Journal, 2022, v. 36, n. 8, p. 1, doi. 10.1096/fj.202200410R
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- Publication type:
- Article
LIM domain-binding 1 maintains the terminally differentiated state of pancreatic β cells.
- Published in:
- 2017
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- Publication type:
- journal article
Partners in Crime: Beta-Cells and Autoimmune Responses Complicit in Type 1 Diabetes Pathogenesis.
- Published in:
- Frontiers in Immunology, 2021, v. 12, p. 1, doi. 10.3389/fimmu.2021.756548
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- Publication type:
- Article
Evidence for Loss in Identity, De-Differentiation, and Trans-Differentiation of Islet β-Cells in Type 2 Diabetes.
- Published in:
- Frontiers in Genetics, 2017, v. 8, p. 1, doi. 10.3389/fgene.2017.00035
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- Publication type:
- Article
Xenotransplantation of tannic acid‐encapsulated neonatal porcine islets decreases proinflammatory innate immune responses.
- Published in:
- Xenotransplantation, 2021, v. 28, n. 6, p. 1, doi. 10.1111/xen.12706
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- Publication type:
- Article
Analyzing Behavioral and Cultural Selection Contingencies.
- Published in:
- Revista Latinoamericana de Psicología, 2012, v. 44, n. 1, p. 43
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- Publication type:
- Article
164-OR: Brown Adipose Tissue as a Novel Site for Islet Transplantation.
- Published in:
- Diabetes, 2021, v. 70, p. N.PAG, doi. 10.2337/db21-164-OR
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- Publication type:
- Article
1686-P: Brown Adipose Tissue as a Novel Site for Islet Transplantation.
- Published in:
- Diabetes, 2020, v. 69, p. N.PAG, doi. 10.2337/db20-1686-P
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- Publication type:
- Article
46-OR: The Transcriptional Coregulator Ldb1 Directly Regulates Brown Adipose Thermogenic Function.
- Published in:
- Diabetes, 2020, v. 69, p. N.PAG, doi. 10.2337/db20-46-OR
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- Publication type:
- Article
143-OR: Illuminating Ldb1 as a Novel Transcriptional Regulator of Brown Adipose Function.
- Published in:
- Diabetes, 2019, v. 68, p. N.PAG, doi. 10.2337/db19-143-OR
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- Publication type:
- Article
Islet-1 Is Essential for Pancreatic β-Cell Function.
- Published in:
- Diabetes, 2014, v. 63, n. 12, p. 4206, doi. 10.2337/db14-0096
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- Publication type:
- Article
Islet α-, β-, and δ-cell development is controlled by the Ldb1 coregulator, acting primarily with the islet-1 transcription factor.
- Published in:
- 2013
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- Publication type:
- journal article
Islet α-, β-, and δ-Cell Development Is Controlled a b d by the Ldb1 Coregulator, Acting Primarily With the Islet-1 Transcription Factor.
- Published in:
- Diabetes, 2013, v. 62, n. 3, p. 875, doi. 10.2337/db12-0952
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- Publication type:
- Article
Islet-1 is Required for the Maturation, Proliferation, and Survival of the Endocrine Pancreas.
- Published in:
- Diabetes, 2009, v. 58, n. 9, p. 2059, doi. 10.2337/db08-0987
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- Publication type:
- Article