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- Title
Oxidative Stress and Mitochondrial Dysfunction in Human Diseases: Pathophysiology, Predictive Biomarkers, Therapeutic.
- Authors
Li, Chia-Jung
- Abstract
Mitochondria are important sites for a variety of cellular processes, including amino acid and fatty acid metabolism, the citric acid cycle, nitrogen metabolism, and oxidative phosphorylation to produce ATP. This toxic waste can cause specific changes (mutations) in the genetic material of the mitochondria that damage the mitochondrion itself and can cause cell dysfunction and disease. RSV treatment also resulted in a decrease in the degree of liver fibrosis, as indicated by the decreased protein expression of the fibrosis markers collagen I, collagen III, and SMA.
- Subjects
OXIDATIVE stress; PATHOLOGICAL physiology; DISEASES; KREBS cycle; BIOMARKERS
- Publication
Biomolecules (2218-273X), 2020, Vol 10, Issue 11, p1558
- ISSN
2218-273X
- Publication type
Editorial
- DOI
10.3390/biom10111558