Found: 24
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Contactin Associates with Sodium Channel Na<sub>v</sub>1.3 in Native Tissues and Increases Channel Density at the Cell Surface.
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- Journal of Neuroscience, 2004, v. 24, n. 33, p. 9387, doi. 10.1523/JNEUROSCI.0322-04.2004
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- Article
Pigmentation and Proliferation of Human Melanocytes and the Effects of Melanocyte-Stimulating Hormone and Ultraviolet B Light<sup>a</sup>.
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- Annals of the New York Academy of Sciences, 1993, v. 680, n. 1, p. 290, doi. 10.1111/j.1749-6632.1993.tb19691.x
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- Article
Type 73 human papillomavirus in esophageal squamous cell carcinoma: A novel association.
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- Cancer (0008543X), 1996, v. 77, n. 12, p. 2440, doi. 10.1002/(SICI)1097-0142(19960615)77:12<2440::AID-CNCR4>3.0.CO;2-Q
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- Article
ERK1/2 Mitogen-Activated Protein Kinase Phosphorylates Sodium Channel Na<sub>v</sub>1.7 and Alters Its Gating Properties.
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- Journal of Neuroscience, 2010, v. 30, n. 5, p. 1637, doi. 10.1523/JNEUROSCI.4872-09.2010
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- Publication type:
- Article
Phosphorylation of Sodium Channel Na<sub>v</sub>1.8 by p38 Mitogen-Activated Protein Kinase Increases Current Density in Dorsal Root Ganglion Neurons.
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- Journal of Neuroscience, 2008, v. 28, n. 12, p. 3190, doi. 10.1523/JNEUROSCI.4403-07.2008
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- Article
Na<sub>v</sub>1.7 Mutant A863P in Erythromelalgia: Effects of Altered Activation and Steady-State Inactivation on Excitability of Nociceptive Dorsal Root Ganglion Neurons.
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- Journal of Neuroscience, 2006, v. 26, n. 48, p. 12566, doi. 10.1523/JNEUROSCI.3424-06.2006
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- Article
Gain-of-function mutations in sodium channel Na(v)1.9 in painful neuropathy.
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- 2014
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- Publication type:
- journal article
Gain-of-function mutations in sodium channel NaV1.9 in painful neuropathy.
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- Brain: A Journal of Neurology, 2014, v. 137, n. 6, p. 1627, doi. 10.1093/brain/awu079
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- Article
Deletion mutation of sodium channel NaV1.7 in inherited erythromelalgia: enhanced slow inactivation modulates dorsal root ganglion neuron hyperexcitability.
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- Brain: A Journal of Neurology, 2011, v. 134, n. 7, p. 1972, doi. 10.1093/brain/awr143
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- Publication type:
- Article
Alternative splicing may contribute to time-dependent manifestation of inherited erythromelalgia.
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- Brain: A Journal of Neurology, 2010, v. 133, n. 6, p. 1823, doi. 10.1093/brain/awq114
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- Publication type:
- Article
Early- and late-onset inherited erythromelalgia: genotype-phenotype correlation.
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- Brain: A Journal of Neurology, 2009, v. 132, n. 7, p. 1711, doi. 10.1093/brain/awp078
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- Publication type:
- Article
Structural modelling and mutant cycle analysis predict pharmacoresponsiveness of a Na<sub>v</sub>1.7 mutant channel.
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- Nature Communications, 2012, v. 3, n. 11, p. 1186, doi. 10.1038/ncomms2184
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- Article
Chronically KIT-Stimulated Clonally-Derived Human Mast Cells Show Heterogeneity in Different Tissue Microenvironments.
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- Journal of Investigative Dermatology, 1997, v. 108, n. 5, p. 792, doi. 10.1111/1523-1747.ep12292240
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- Article
A channelopathy contributes to cerebellar dysfunction in a model of multiple sclerosis.
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- Annals of Neurology, 2012, v. 71, n. 2, p. 186, doi. 10.1002/ana.22665
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- Publication type:
- Article
A sodium channel gene SCN9A polymorphism that increases nociceptor excitability.
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- Annals of Neurology, 2009, v. 66, n. 6, p. 862, doi. 10.1002/ana.21895
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- Publication type:
- Article
Sporadic onset of erythermalgia: A gain‐of‐function mutation in Nav1.7.
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- Annals of Neurology, 2006, v. 59, n. 3, p. 553
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- Publication type:
- Article
Na<sub>v</sub>1.7 is the predominant sodium channel in rodent olfactory sensory neurons.
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- Molecular Pain, 2011, v. 7, n. 1, p. 32, doi. 10.1186/1744-8069-7-32
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- Publication type:
- Article
Mutations at opposite ends of the DIII/S4-S5 linker of sodium channel Na<sub>V</sub>1.7 produce distinct pain disorders.
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- Molecular Pain, 2010, v. 6, n. 1, p. 24, doi. 10.1186/1744-8069-6-24
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- Publication type:
- Article
Paroxysmal extreme pain disorder M1627K mutation in human Na<sub>v</sub>1.7 renders DRG neurons hyperexcitable.
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- Molecular Pain, 2008, v. 4, n. 1, p. 1, doi. 10.1186/1744-8069-4-37
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- Article
Mutation I136V alters electrophysiological properties of the Na<sub>v</sub>1.7 channel in a family with onset of erythromelalgia in the second decade.
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- Molecular Pain, 2008, v. 4, n. 1, p. 1, doi. 10.1186/1744-8069-4-1
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- Publication type:
- Article
Malignant and Normal T Cells Show Random Use of T-Cell Receptor ∝ Chain Variable Regions in Patients with Cutaneous T-Cell Lymphoma.
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- Journal of Investigative Dermatology, 1995, v. 105, n. 1, p. 62, doi. 10.1111/1523-1747.ep12312571
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- Publication type:
- Article
Differential modulation of sodium channel Na<sub>v</sub>1.6 by two members of the fibroblast growth factor homologous factor 2 subfamily.
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- European Journal of Neuroscience, 2006, v. 23, n. 10, p. 2551, doi. 10.1111/j.1460-9568.2006.04789.x
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- Publication type:
- Article
Functional role of the C-terminus of voltage-gated sodium channel Na<sub>v</sub>1.8
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- FEBS Letters, 2004, v. 572, n. 1-3, p. 256, doi. 10.1016/j.febslet.2004.07.047
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- Publication type:
- Article
Structure of the sodium channel gene SCN11A.
- Published in:
- Molecular Neurobiology, 2002, v. 26, n. 2/3, p. 235, doi. 10.1385/MN:26:2-3:235
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- Publication type:
- Article