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CCR6: A Biomarker for Alzheimer's-like Disease in a Triple Transgenic Mouse Model.
- Published in:
- Journal of Alzheimer's Disease, 2010, v. 22, n. 2, p. 619, doi. 10.3233/JAD-2010-100852
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- Article
Partial MHC class II constructs inhibit MIF/ CD74 binding and downstream effects.
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- European Journal of Immunology, 2013, v. 43, n. 5, p. 1309, doi. 10.1002/eji.201243162
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- Article
TCR-like antibodies distinguish conformational and functional differences in two- versus four-domain auto reactive MHC class II-peptide complexes.
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- European Journal of Immunology, 2011, v. 41, n. 5, p. 1465, doi. 10.1002/eji.201041241
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- Article
Estrogen-induced protection against experimental autoimmune encephalomyelitis is abrogated in the absence of B cells.
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- European Journal of Immunology, 2011, v. 41, n. 4, p. 1165, doi. 10.1002/eji.201040992
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- Article
Spleen participation in partial MHC class II construct neuroprotection in stroke.
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- CNS Neuroscience & Therapeutics, 2020, v. 26, n. 7, p. 663, doi. 10.1111/cns.13369
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- Article
Attenuated T-Lymphocyte Lines As Vaccinating Agents Against Experimental Autoimmune Encephalomyelitisa.
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- Annals of the New York Academy of Sciences, 1988, v. 540, n. 1, p. 540, doi. 10.1111/j.1749-6632.1988.tb27165.x
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- Article
Suppressor Cell Regulation of Chronic Relapsing Experimental Allergic Encephalomyelitis.
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- Annals of the New York Academy of Sciences, 1988, v. 540, n. 1, p. 537, doi. 10.1111/j.1749-6632.1988.tb27164.x
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- Article
Response of Rat Encephalitogenic T Cells to Synthetic Peptides of Guinea Pig Myelin Basic Proteina.
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- Annals of the New York Academy of Sciences, 1988, v. 540, n. 1, p. 337, doi. 10.1111/j.1749-6632.1988.tb27091.x
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- Article
Treg suppressive activity involves estrogen-dependent expression of programmed death-1 (PD-1).
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- International Immunology, 2007, v. 19, n. 3, p. 337
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- Article
Differential susceptibility of human Th1 versus T h 2 cells to induction of anergy and apoptosis by ECDI/antigen-coupled antigen-presenting cells.
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- International Immunology, 2000, v. 12, n. 1, p. 57, doi. 10.1093/intimm/12.1.57
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- Article
Estrogen-induced compensatory mechanisms protect IL-10-deficient mice from developing EAE.
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- 2019
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- Publication type:
- journal article
GPR30, but not estrogen receptor-α, is crucial in the treatment of experimental autoimmune encephalomyelitis by oral ethinyl estradiol.
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- BMC Immunology, 2010, v. 11, p. 20, doi. 10.1186/1471-2172-11-20
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- Article
A Potential Role for Estrogen in Experimental Autoimmune Encephalomyelitis and Multiple Sclerosis.
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- Annals of the New York Academy of Sciences, 2006, v. 1089, n. 1, p. 343, doi. 10.1196/annals.1386.021
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- Publication type:
- Article
Cross-Talk of the CNS With Immune Cells and Functions in Health and Disease.
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- Frontiers in Neurology, 2021, v. 12, p. 1, doi. 10.3389/fneur.2021.672455
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- Publication type:
- Article
RTL551 Treatment of EAE Reduces CD226 and T-bet+ CD4 T Cells in Periphery and Prevents Infiltration of T-bet+ IL-17, IFN-&ggr; Producing T Cells into CNS.
- Published in:
- PLoS ONE, 2011, v. 6, n. 7, p. 1, doi. 10.1371/journal.pone.0021868
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- Article
Sex differences in the immune response to experimental stroke: Implications for translational research.
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- Journal of Neuroscience Research, 2017, v. 95, n. 1/2, p. 437, doi. 10.1002/jnr.23784
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- Publication type:
- Article
Estrogen‐mediated immunomodulation involves reduced activation of effector T cells, potentiation of treg cells, and enhanced expression of the PD‐1 costimulatory pathway.
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- Journal of Neuroscience Research, 2006, v. 84, n. 2, p. 370, doi. 10.1002/jnr.20881
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- Article
Decreased FOXP3 levels in multiple sclerosis patientsA.A.V. has a significant financial interest in The Immune Response Corporation, a company that may have a commercial interest in the results of this research and technology. This potential conflict of interest has been reviewed and managed by the OHSU and VAMC Conflict of Interest Committees.
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- Journal of Neuroscience Research, 2005, v. 81, n. 1, p. 45, doi. 10.1002/jnr.20522
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- Publication type:
- Article
Neuroimmunoprotective effects of estrogen and derivatives in experimental autoimmune encephalomyelitis: Therapeutic implications for multiple sclerosis.
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- Journal of Neuroscience Research, 2004, v. 78, n. 5, p. 603, doi. 10.1002/jnr.20330
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- Article
HLA‐DRB1*1501 risk association in multiple sclerosis may not be related to presentation of myelin epitopes.
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- Journal of Neuroscience Research, 2004, v. 78, n. 1, p. 100, doi. 10.1002/jnr.20227
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- Publication type:
- Article
T‐cell hybridoma specific for myelin oligodendrocyte glycoprotein‐35–55 peptide produced from HLA‐DRB1*1501‐transgenic mice.
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- Journal of Neuroscience Research, 2004, v. 77, n. 5, p. 670, doi. 10.1002/jnr.20201
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- Publication type:
- Article
Estrogen treatment induces a novel population of regulatory cells, which suppresses experimental autoimmune encephalomyelitis.
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- Journal of Neuroscience Research, 2004, v. 77, n. 1, p. 119, doi. 10.1002/jnr.20145
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- Publication type:
- Article
Effects of cytokine deficiency on chemokine expression in CNS of mice with EAE.
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- Journal of Neuroscience Research, 2002, v. 67, n. 5, p. 680, doi. 10.1002/jnr.10156
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- Publication type:
- Article
Gender differences in protection from EAE induced by oral tolerance with a peptide analogue of MBP-Ac1-11.
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- Journal of Neuroscience Research, 1999, v. 55, n. 4, p. 432, doi. 10.1002/(SICI)1097-4547(19990215)55:4<432::AID-JNR4>3.0.CO;2-2
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- Publication type:
- Article
Neonatal exposure of TCR BV8S2 transgenic mice to recombinant TCR BV8S2 results in reduced T cell proliferation and elevated antibody response to BV8S2, and increased severity of EAE.
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- Journal of Neuroscience Research, 1998, v. 52, n. 6, p. 750, doi. 10.1002/(SICI)1097-4547(19980615)52:6<750::AID-JNR14>3.0.CO;2-2
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- Publication type:
- Article
Gender differences in experimental autoimmune encephalomyelitis develop during the induction of the immune response to encephalitogenic peptides.
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- Journal of Neuroscience Research, 1998, v. 52, n. 4, p. 420, doi. 10.1002/(SICI)1097-4547(19980515)52:4<420::AID-JNR5>3.0.CO;2-B
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- Publication type:
- Article
Sex differences in the therapeutic effects of anti-PDL2 neutralizing antibody on stroke.
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- Metabolic Brain Disease, 2019, v. 34, n. 6, p. 1705, doi. 10.1007/s11011-019-00476-3
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- Article
Increased CD74 binding and EAE treatment efficacy of a modified DRα1 molecular construct.
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- Metabolic Brain Disease, 2019, v. 34, n. 1, p. 153, doi. 10.1007/s11011-018-0331-2
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- Article
Antibiotics protect against EAE by increasing regulatory and anti-inflammatory cells.
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- Metabolic Brain Disease, 2018, v. 33, n. 5, p. 1599, doi. 10.1007/s11011-018-0266-7
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- Article
Estrogen protects both sexes against EAE by promoting common regulatory cell subtypes independent of endogenous estrogen.
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- Metabolic Brain Disease, 2017, v. 32, n. 5, p. 1747, doi. 10.1007/s11011-017-0063-8
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- Article
DRα1-MOG-35-55 treatment reduces lesion volumes and improves neurological deficits after traumatic brain injury.
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- Metabolic Brain Disease, 2017, v. 32, n. 5, p. 1395, doi. 10.1007/s11011-017-9991-6
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- Article
Sex differences and the role of PPAR alpha in experimental stroke.
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- Metabolic Brain Disease, 2016, v. 31, n. 3, p. 539, doi. 10.1007/s11011-015-9766-x
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- Article
Loss of PPARα perpetuates sex differences in stroke reflected by peripheral immune mechanisms.
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- Metabolic Brain Disease, 2016, v. 31, n. 3, p. 683, doi. 10.1007/s11011-016-9805-2
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- Article
Treatment with IL-10 producing B cells in combination with E2 ameliorates EAE severity and decreases CNS inflammation in B cell-deficient mice.
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- Metabolic Brain Disease, 2015, v. 30, n. 5, p. 1117, doi. 10.1007/s11011-015-9661-5
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- Article
Role for microglia in sex differences after ischemic stroke: importance of M2.
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- Metabolic Brain Disease, 2015, v. 30, n. 6, p. 1515, doi. 10.1007/s11011-015-9714-9
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- Publication type:
- Article
Regulatory CD8CD122 T-cells predominate in CNS after treatment of experimental stroke in male mice with IL-10-secreting B-cells.
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- Metabolic Brain Disease, 2015, v. 30, n. 4, p. 911, doi. 10.1007/s11011-014-9639-8
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- Article
Thrombin mutant W215A/E217A treatment improves neurological outcome and attenuates central nervous system damage in experimental autoimmune encephalomyelitis.
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- Metabolic Brain Disease, 2015, v. 30, n. 1, p. 57, doi. 10.1007/s11011-014-9558-8
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- Article
Erratum to: A novel HLA-DRα1-MOG-35-55 construct treats experimental stroke.
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- 2014
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- Publication type:
- Erratum
Treatment of experimental stroke with IL-10-producing B-cells reduces infarct size and peripheral and CNS inflammation in wild-type B-cell-sufficient mice.
- Published in:
- Metabolic Brain Disease, 2014, v. 29, n. 1, p. 59, doi. 10.1007/s11011-013-9474-3
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- Publication type:
- Article
A novel HLA-DRα1-MOG-35-55 construct treats experimental stroke.
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- Metabolic Brain Disease, 2014, v. 29, n. 1, p. 37, doi. 10.1007/s11011-013-9440-0
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- Publication type:
- Article
IL-10-producing B-cells limit CNS inflammation and infarct volume in experimental stroke.
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- Metabolic Brain Disease, 2013, v. 28, n. 3, p. 375, doi. 10.1007/s11011-013-9413-3
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- Publication type:
- Article
Intrastriatal B-cell administration limits infarct size after stroke in B-cell deficient mice.
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- Metabolic Brain Disease, 2012, v. 27, n. 4, p. 487, doi. 10.1007/s11011-012-9317-7
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- Publication type:
- Article
Gilt required for RTL550-CYS-MOG to treat experimental autoimmune encephalomyelitis.
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- Metabolic Brain Disease, 2012, v. 27, n. 2, p. 143, doi. 10.1007/s11011-012-9289-7
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- Publication type:
- Article
Myelin specific cells infiltrate MCAO lesions and exacerbate stroke severity.
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- Metabolic Brain Disease, 2012, v. 27, n. 1, p. 7, doi. 10.1007/s11011-011-9267-5
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- Publication type:
- Article
Contribution of GPR30 for 1,25 dihydroxyvitamin D protection in EAE.
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- Metabolic Brain Disease, 2012, v. 27, n. 1, p. 29, doi. 10.1007/s11011-011-9266-6
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- Publication type:
- Article
Sustained expression of circulating human alpha-1 antitrypsin reduces inflammation, increases CD4+FoxP3+ Treg cell population and prevents signs of experimental autoimmune encephalomyelitis in mice.
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- Metabolic Brain Disease, 2011, v. 26, n. 2, p. 107, doi. 10.1007/s11011-011-9239-9
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- Publication type:
- Article
Therapy with recombinant T-cell receptor ligand reduces infarct size and infiltrating inflammatory cells in brain after middle cerebral artery occlusion in mice.
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- Metabolic Brain Disease, 2011, v. 26, n. 2, p. 123, doi. 10.1007/s11011-011-9241-2
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- Publication type:
- Article
CD4FoxP3 regulatory T-cells in cerebral ischemic stroke.
- Published in:
- Metabolic Brain Disease, 2011, v. 26, n. 1, p. 87, doi. 10.1007/s11011-010-9226-6
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- Publication type:
- Article
Cytokine Switch and Bystander Suppression of Autoimmune Responses to Multiple Antigens in Experimental Autoimmune Encephalomyelitis by a Single Recombinant T-Cell Receptor Ligand.
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- Journal of Neuroscience, 2009, v. 29, n. 12, p. 3816, doi. 10.1523/JNEUROSCI.5812-08.2009
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- Publication type:
- Article
A Promising Therapeutic Approach for Multiple Sclerosis: Recombinant T-Cell Receptor Ligands Modulate Experimental Autoimmune Encephalomyelitis by Reducing Interleukin-17 Production and Inhibiting Migration of Encephalitogenic Cells into the CNS.
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- Journal of Neuroscience, 2007, v. 27, n. 46, p. 12531, doi. 10.1523/JNEUROSCI.3599-07.2007
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- Article