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Airway Epithelial Inflammation In Vitro Augments the Rescue of Mutant CFTR by Current CFTR Modulator Therapies.
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- Frontiers in Pharmacology, 2021, v. 11, p. N.PAG, doi. 10.3389/fphar.2021.628722
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- Article
TheLegionellaIcmS–IcmW protein complex is important for Dot/Icm-mediated protein translocation.
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- Molecular Microbiology, 2005, v. 55, n. 3, p. 912, doi. 10.1111/j.1365-2958.2004.04435.x
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- Article
Bioactive Thymosin Alpha-1 Does Not Influence F508del-CFTR Maturation and Activity.
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- Scientific Reports, 2019, v. 9, n. 1, p. N.PAG, doi. 10.1038/s41598-019-46639-1
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- Article
Cigarette smoke exposure induces CFTR internalization and insolubility, leading to airway surface liquid dehydration.
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- FASEB Journal, 2012, v. 26, n. 2, p. 533, doi. 10.1096/fj.11-192377
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- Article
Potentiator ivacaftor abrogates pharmacological correction of ΔF508 CFTR in cystic fibrosis.
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- Science Translational Medicine, 2014, v. 6, n. 246, p. 1, doi. 10.1126/scitranslmed.3008680
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- Article
A Novel Co-Culture Model Reveals Enhanced CFTR Rescue in Primary Cystic Fibrosis Airway Epithelial Cultures with Persistent Pseudomonas aeruginosa Infection.
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- Cells (2073-4409), 2023, v. 12, n. 22, p. 2618, doi. 10.3390/cells12222618
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- Article
Cystic fibrosis airway inflammation enables elexacaftor/ tezacaftor/ivacaftor-mediated rescue of N1303K CFTR mutation.
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- ERJ Open Research, 2024, v. 10, n. 1, p. 1, doi. 10.1183/23120541.00746-2023
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- Article