Found: 26
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Defects in mitophagy promote redox-driven metabolic syndrome in the absence of TP53 INP1.
- Published in:
- EMBO Molecular Medicine, 2015, v. 7, n. 6, p. 802, doi. 10.15252/emmm.201404318
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- Article
Mechanisms of Beta-Cell Apoptosis in Type 2 Diabetes-Prone Situations and Potential Protection by GLP-1-Based Therapies.
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- International Journal of Molecular Sciences, 2021, v. 22, n. 10, p. 5303, doi. 10.3390/ijms22105303
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- Article
ERK1 is dispensable for mouse pancreatic beta cell function but is necessary for glucose-induced full activation of MSK1 and CREB.
- Published in:
- Diabetologia, 2017, v. 60, n. 10, p. 1999, doi. 10.1007/s00125-017-4356-6
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- Article
β-Arrestin2 plays a key role in the modulation of the pancreatic beta cell mass in mice.
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- Diabetologia, 2014, v. 57, n. 3, p. 532, doi. 10.1007/s00125-013-3130-7
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- Article
The Mitochondrial Ca<sup>2+</sup> Uniporter MCU Is Essential for Glucose-Induced ATP Increases in Pancreatic <sup>β</sup>-Cells.
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- PLoS ONE, 2012, v. 7, n. 7, p. 1, doi. 10.1371/journal.pone.0039722
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- Article
Frequency-dependent mitochondrial Ca accumulation regulates ATP synthesis in pancreatic β cells.
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- Pflügers Archiv: European Journal of Physiology, 2013, v. 465, n. 4, p. 543, doi. 10.1007/s00424-012-1177-9
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- Publication type:
- Article
The nuclear receptor REV-ERBα is implicated in the alteration of β-cell autophagy and survival under diabetogenic conditions.
- Published in:
- Cell Death & Disease, 2022, v. 13, n. 4, p. 1, doi. 10.1038/s41419-022-04767-z
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- Article
Proteasomal degradation of the histone acetyl transferase p300 contributes to beta-cell injury in a diabetes environment.
- Published in:
- Cell Death & Disease, 2018, v. 9, n. 6, p. 1, doi. 10.1038/s41419-018-0603-0
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- Article
Glucose-dependent regulation of gamma-aminobutyric acid (GABA A) receptor expression in mouse pancreatic islet alpha-cells.
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- 2007
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- Publication type:
- journal article
Glucose-Dependent Regulation of γ-Aminobutyric Acid (GABA<sub>A</sub>) Receptor Expression in Mouse Pancreatic Islet α-Cells.
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- Diabetes, 2007, v. 56, n. 2, p. 320, doi. 10.2337/db06-0712
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- Article
Sustained exposure to high glucose concentrations modifies glucose signaling and the mechanics of secretory vesicle fusion in primary rat pancreatic beta-cells.
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- 2006
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- Publication type:
- journal article
Sustained Exposure to High Glucose Concentrations Modifies Glucose Signaling and the Mechanics of Secretory Vesicle Fusion in Primary Rat Pancreatic β-Cells.
- Published in:
- Diabetes, 2006, v. 55, n. 4, p. 1057, doi. 10.2337/diabetes.55.04.06.db05-1577
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- Publication type:
- Article
Loss of connexin36 channels alters beta-cell coupling, islet synchronization of glucose-induced Ca2+ and insulin oscillations, and basal insulin release.
- Published in:
- 2005
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- Publication type:
- journal article
Loss of Connexin36 Channels Alters β-Cell Coupling, Islet Synchronization of Glucose-Induced Ca<sup>2+</sup> and Insulin Oscillations, and Basal Insulin Release.
- Published in:
- Diabetes, 2005, v. 54, n. 6, p. 1798, doi. 10.2337/diabetes.54.6.1798
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- Publication type:
- Article
Glucose or insulin, but not zinc ions, inhibit glucagon secretion from mouse pancreatic alpha-cells.
- Published in:
- 2005
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- Publication type:
- journal article
Do oscillations of insulin secretion occur in the absence of cytoplasmic Ca2+ oscillations in beta-cells?
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- 2002
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- journal article
Control mechanisms of the oscillations of insulin secretion in vitro and in vivo.
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- 2002
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- Publication type:
- journal article
Signals and pools underlying biphasic insulin secretion.
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- 2002
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- journal article
Oscillations of insulin secretion can be triggered by imposed oscillations of cytoplasmic Ca2+ or metabolism in normal mouse islets.
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- 1999
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- Publication type:
- journal article
Metabolic Stress Impairs Pericyte Response to Optogenetic Stimulation in Pancreatic Islets.
- Published in:
- Frontiers in Endocrinology, 2022, v. 13, p. 1, doi. 10.3389/fendo.2022.918733
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- Article
Time and amplitude regulation of pulsatile insulin secretion by triggering and amplifying pathways in mouse islets
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- FEBS Letters, 2002, v. 530, n. 1-3, p. 215, doi. 10.1016/S0014-5793(02)03491-9
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- Publication type:
- Article
325-LB: Circadian Clock Nuclear Receptor REV-ERBa Is a Novel Regulator of Beta-Cell Function, Survival, and Autophagy under Diabetogenic Conditions.
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- Diabetes, 2019, v. 68, p. N.PAG, doi. 10.2337/db19-325-LB
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- Publication type:
- Article
Tolbutamide controls glucagon release from mouse islets differently than glucose: involvement of K(ATP) channels from both α-cells and δ-cells.
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- 2013
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- Publication type:
- journal article
Tolbutamide Controls Glucagon Release From Mouse Islets Differently Than Glucose.
- Published in:
- Diabetes, 2013, v. 62, n. 5, p. 1612, doi. 10.2337/db12-0347
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- Publication type:
- Article
Mechanisms of control of the free Ca2+ concentration in the endoplasmic reticulum of mouse pancreatic β-cells: interplay with cell metabolism and [Ca2+]c and role of SERCA2b and SERCA3.
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- 2011
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- journal article
Mechanisms of Control of the Free Ca<sup>2+</sup> Concentration in the Endoplasmic Reticulum of Mouse Pancreatic β-Cells.
- Published in:
- Diabetes, 2011, v. 60, n. 10, p. 2533, doi. 10.2337/db10-1543
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- Publication type:
- Article