Found: 6
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Xeroderma Pigmentosum C (XPC) Mutations in Primary Fibroblasts Impair Base Excision Repair Pathway and Increase Oxidative DNA Damage.
- Published in:
- Frontiers in Genetics, 2020, v. 11, p. N.PAG, doi. 10.3389/fgene.2020.561687
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- Article
Comment: the mystery of melanocyte demise in vitiligo.
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- Experimental Dermatology, 2015, v. 24, n. 4, p. 260, doi. 10.1111/exd.12659
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- Article
Oxidative and Energy Metabolism as Potential Clues for Clinical Heterogeneity in Nucleotide Excision Repair Disorders.
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- Journal of Investigative Dermatology, 2015, v. 135, n. 2, p. 341, doi. 10.1038/jid.2014.365
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- Article
HIF-1α in Epidermis: Oxygen Sensing, Cutaneous Angiogenesis, Cancer, and Non-Cancer Disorders.
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- Journal of Investigative Dermatology, 2011, v. 131, n. 9, p. 1793, doi. 10.1038/jid.2011.141
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- Article
UVB-induced DHODH upregulation, which is driven by STAT3, is a promising target for chemoprevention and combination therapy of photocarcinogenesis.
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- Oncogenesis, 2019, v. 8, n. 10, p. N.PAG, doi. 10.1038/s41389-019-0161-z
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- Article
Nucleotide excision repair diseases.
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- Journal of the Egyptian Women's Dermatologic Society, 2013, v. 10, n. 2, p. 49, doi. 10.1097/01.EWX.0000428203.18568.bf
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- Article