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Connexin 43: a New Therapeutic Target Against Chronic Kidney Disease?
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- Cellular Physiology & Biochemistry (Karger AG), 2018, v. 49, n. 3, p. 998, doi. 10.1159/000493230
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- Article
The Angiogenesis Inhibitor Isthmin-1 (ISM1) Is Overexpressed in Experimental Models of Glomerulopathy and Impairs the Viability of Podocytes.
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- International Journal of Molecular Sciences, 2023, v. 24, n. 3, p. 2723, doi. 10.3390/ijms24032723
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- Article
Deletion of Notch3 Impairs Contractility of Renal Resistance Vessels Due to Deficient Ca 2+ Entry.
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- International Journal of Molecular Sciences, 2022, v. 23, n. 24, p. 16068, doi. 10.3390/ijms232416068
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- Article
The Role of Connexin 43 in Renal Disease: Insights from In Vivo Models of Experimental Nephropathy.
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- International Journal of Molecular Sciences, 2022, v. 23, n. 21, p. 13090, doi. 10.3390/ijms232113090
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- Article
Galectin-3 in Kidney Diseases: From an Old Protein to a New Therapeutic Target.
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- International Journal of Molecular Sciences, 2022, v. 23, n. 6, p. 3124, doi. 10.3390/ijms23063124
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- Article
Activation of Notch3 in Renal Tubular Cells Leads to Progressive Cystic Kidney Disease.
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- International Journal of Molecular Sciences, 2022, v. 23, n. 2, p. 884, doi. 10.3390/ijms23020884
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- Article
Connexin 43: A Target for the Treatment of Inflammation in Secondary Complications of the Kidney and Eye in Diabetes.
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- International Journal of Molecular Sciences, 2022, v. 23, n. 2, p. 600, doi. 10.3390/ijms23020600
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- Article
Targeting connexin 43 protects against the progression of experimental chronic kidney disease in mice.
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- Kidney International, 2014, v. 86, n. 4, p. 768, doi. 10.1038/ki.2014.108
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- Article
Connexin37 protects against atherosclerosis by regulating monocyte adhesion.
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- Nature Medicine, 2006, v. 12, n. 8, p. 950, doi. 10.1038/nm1441
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- Article
Reduced NOV/CCN3 Expression Limits Inflammation and Interstitial Renal Fibrosis after Obstructive Nephropathy in Mice.
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- PLoS ONE, 2015, v. 10, n. 9, p. 1, doi. 10.1371/journal.pone.0137876
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- Article
The RenTg Mice: A Powerful Tool to Study Renin-Dependent Chronic Kidney Disease.
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- PLoS ONE, 2012, v. 7, n. 12, p. 1, doi. 10.1371/journal.pone.0052362
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- Article
Connexin-43 hemichannels orchestrate NOD-like receptor protein-3 (NLRP3) inflammasome activation and sterile inflammation in tubular injury.
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- Cell Communication & Signaling, 2023, v. 21, n. 1, p. 1, doi. 10.1186/s12964-023-01245-7
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- Article
Progression of renal fibrosis: the underestimated role of endothelial alterations.
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- Fibrogenesis & Tissue Repair, 2012, v. 5, n. Suppl 1, p. 1, doi. 10.1186/1755-1536-5-S1-S15
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- Article
Subclinical and clinical acute kidney injury share similar urinary peptide signatures and prognosis.
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- Intensive Care Medicine, 2023, v. 49, n. 10, p. 1191, doi. 10.1007/s00134-023-07198-2
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- Article
MiR‐21 is up‐regulated in urinary exosomes of chronic kidney disease patients and after glomerular injury.
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- Journal of Cellular & Molecular Medicine, 2019, v. 23, n. 7, p. 4839, doi. 10.1111/jcmm.14317
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- Article
Super‐resolved highly multiplexed immunofluorescence imaging for precise protein localization and podocyte ultrastructure.
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- Journal of Cellular & Molecular Medicine, 2024, v. 28, n. 18, p. 1, doi. 10.1111/jcmm.70066
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- Article
The role of CD146 in renal disease: from experimental nephropathy to clinics.
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- Journal of Molecular Medicine, 2024, v. 102, n. 1, p. 11, doi. 10.1007/s00109-023-02392-7
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- Article
Discoidin domain receptor-1 and periostin: new players in chronic kidney disease.
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- Nephrology Dialysis Transplantation, 2015, v. 30, n. 12, p. 1965, doi. 10.1093/ndt/gfv074
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- Article
Blocking Connexin-43 mediated hemichannel activity protects against early tubular injury in experimental chronic kidney disease.
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- Cell Communication & Signaling, 2020, v. 18, n. 1, p. 1, doi. 10.1186/s12964-020-00558-1
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- Article