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TGF-β1 promotes microglial amyloid-β clearance and reduces plaque burden in transgenic mice.
- Published in:
- Nature Medicine, 2001, v. 7, n. 5, p. 612, doi. 10.1038/87945
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- Article
Fyn Kinase Modulates Synaptotoxicity, But Not Aberrant Sprouting, in Human Amyloid Precursor Protein Transgenic Mice.
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- Journal of Neuroscience, 2004, v. 24, n. 19, p. 4692, doi. 10.1523/JNEUROSCI.0277-04.2004
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- Article
Neuron-Specific Apolipoprotein E4 Proteolysis Is Associated with Increased Tau Phosphorylation in Brains of Transgenic Mice.
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- Journal of Neuroscience, 2004, v. 24, n. 10, p. 2527, doi. 10.1523/JNEUROSCI.4315-03.2004
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- Article
F1‐07‐01: ASTROGLIAL AND MICROGLIAL MODULATION OF NEURAL FUNCTIONS IN MODELS OF ALZHEIMER'S DISEASE.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2019, v. 15, p. P188, doi. 10.1016/j.jalz.2019.06.4393
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- Article
Phenotypic Differences between the Alzheimer's Disease-Related hAPP-J20 Model and Heterozygous Zbtb20 Knock-OutMice.
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- eNeuro, 2021, v. 8, n. 3, p. 1, doi. 10.1523/ENEURO.0089-21.2021
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- Article
Reducing epileptiform activity by levetiracetam rescues synaptic and cognitive functions in a mouse model of Alzheimer's disease
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- 2011
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- Abstract
Excitotoxicity in Alzheimer's disease or network dys function in Alzheimer's disease
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- 2011
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Modulation of EphB2, but not of cellular prion protein, rescues cognitive functions in a mouse model of Alzheimer's disease
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- 2011
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Preventing C31 generation does not prevent many Aβ-dependenent neuronal and behavioral impairments in the happ-J20 mouse model of Alzheimer's disease
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- 2009
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- Abstract
P1-465: Role of collagen VI in Alzheimer's disease: Potential mechanisms of protection
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- 2008
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- Abstract
P1-078: Alterations in voltage-gated sodium channels in transgenic mouse models of alzheimer's disease: Implications for Aβ-induced network dysfunction
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- 2008
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O4-02-03: Phospholipase A2 reduction ameliorates cognitive deficits in mouse model of Alzheimer's disease
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- 2008
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- Abstract
P4-314: Overexpression of neprilysin decreases neuronal and behavioral impairments in human amyloid precursor protein transgenic mice
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- 2006
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- Abstract
P3-359: Reduction of mitochondrial superoxide dismutase modulates Alzheimer’s disease-like pathology in human amyloid precursor protein transgenic mice
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- 2006
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- Abstract
P1-110: Reelin expression in pyramidal cells of the entorhinal cortex is decreased in human amyloid precursor protein transgenic mice
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- 2006
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- Abstract
P1-069: Hippocampal subregion-specific upregulation of alpha 7 nicotinic acetylcholine receptors and striatal-enriched phosphatase in APP transgenic mice
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- 2006
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- Abstract
O2-04-05: Reducing endogenous tau prevents APP/Aβ-induced deficits in a mouse model of Alzheimer’s disease
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- 2006
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- Abstract
P4-314: Overexpression of neprilysin decreases neuronal and behavioral impairments in human amyloid precursor protein transgenic mice
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- 2006
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- Abstract
P3-359: Reduction of mitochondrial superoxide dismutase modulates Alzheimer’s disease-like pathology in human amyloid precursor protein transgenic mice
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- 2006
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- Abstract
P1-110: Reelin expression in pyramidal cells of the entorhinal cortex is decreased in human amyloid precursor protein transgenic mice
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- 2006
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- Abstract
P1-069: Hippocampal subregion-specific upregulation of alpha 7 nicotinic acetylcholine receptors and striatal-enriched phosphatase in APP transgenic mice
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- 2006
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- Abstract
O2-04-05: Reducing endogenous tau prevents APP/Aβ-induced deficits in a mouse model of Alzheimer’s disease
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- 2006
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- Abstract
Tau Reduction Diminishes Spatial Learning and Memory Deficits after Mild Repetitive Traumatic Brain Injury in Mice.
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- PLoS ONE, 2014, v. 9, n. 12, p. 1, doi. 10.1371/journal.pone.0115765
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- Article
Human P301L-Mutant Tau Expression in Mouse Entorhinal-Hippocampal Network Causes Tau Aggregation and Presynaptic Pathology but No Cognitive Deficits.
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- PLoS ONE, 2012, v. 7, n. 9, p. 1, doi. 10.1371/journal.pone.0045881
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- Article
Expression of A152T human tau causes age-dependent neuronal dysfunction and loss in transgenic mice.
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- EMBO Reports, 2016, v. 17, n. 4, p. 530, doi. 10.15252/embr.201541438
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- Article
Many Neuronal and Behavioral Impairments in Transgenic Mouse Models of Alzheimer's Disease Are Independent of Caspase Cleavage of the Amyloid Precursor Protein.
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- Journal of Neuroscience, 2010, v. 30, n. 1, p. 372, doi. 10.1523/JNEUROSCI.5341-09.2010
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- Article
Neprilysin Overexpression Inhibits Plaque Formation But Fails to Reduce Pathogenic Aβ Oligomers and Associated Cognitive Deficits in Human Amyloid Precursor Protein Transgenic Mice.
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- Journal of Neuroscience, 2009, v. 29, n. 7, p. 1977, doi. 10.1523/JNEUROSCI.2984-08.2009
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- Article
Enkephalin Elevations Contribute to Neuronal and Behavioral Impairments in a Transgenic Mouse Model of Alzheimer's Disease.
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- Journal of Neuroscience, 2008, v. 28, n. 19, p. 5007, doi. 10.1523/JNEUROSCI.0590-08.2008
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- Article
Reelin Depletion in the Entorhinal Cortex of Human Amyloid Precursor Protein Transgenic Mice and Humans with Alzheimer's Disease.
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- Journal of Neuroscience, 2007, v. 27, n. 11, p. 2727, doi. 10.1523/JNEUROSCI.3758-06.2007
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- Article
Vulnerability of Dentate Granule Cells to Disruption of Arc Expression in Human Amyloid Precursor Protein Transgenic Mice.
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- Journal of Neuroscience, 2005, v. 25, n. 42, p. 9686, doi. 10.1523/JNEUROSCI.2829-05.2005
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- Article
Reversing EphB2 depletion rescues cognitive functions in Alzheimer model.
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- Nature, 2011, v. 469, n. 7328, p. 47, doi. 10.1038/nature09635
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- Article
Astrocytic adenosine receptor A<sub>2A</sub> and G<sub>s</sub>-coupled signaling regulate memory.
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- Nature Neuroscience, 2015, v. 18, n. 3, p. 423, doi. 10.1038/nn.3930
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- Article
Collagen VI protects neurons against Aβ toxicity.
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- Nature Neuroscience, 2009, v. 12, n. 2, p. 119, doi. 10.1038/nn.2240
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- Article
Phospholipase A<sub>2</sub> reduction ameliorates cognitive deficits in a mouse model of Alzheimer's disease.
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- Nature Neuroscience, 2008, v. 11, n. 11, p. 1311, doi. 10.1038/nn.2213
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- Article
TAU ablation in excitatory neurons and postnatal TAU knockdown reduce epilepsy, SUDEP, and autism behaviors in a Dravet syndrome model.
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- Science Translational Medicine, 2022, v. 14, n. 642, p. 1, doi. 10.1126/scitranslmed.abm5527
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- Article
A second X chromosome contributes to resilience in a mouse model of Alzheimer's disease.
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- Science Translational Medicine, 2020, v. 12, n. 558, p. 1, doi. 10.1126/scitranslmed.aaz5677
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- Article
Intracellularly generated amyloid-β peptide counteracts the antiapoptotic function of its precursor protein and primes proapoptotic pathways for activation by other insults in neuroblastoma cells.
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- Journal of Neurochemistry, 2004, v. 91, n. 6, p. 1260, doi. 10.1111/j.1471-4159.2004.02816.x
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- Article
Network dysfunction in α-synuclein transgenic mice and human Lewy body dementia.
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- Annals of Clinical & Translational Neurology, 2015, v. 2, n. 11, p. 1012, doi. 10.1002/acn3.257
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- Article
Behavioral and neural network abnormalities in human APP transgenic mice resemble those of App knock-in mice and are modulated by familial Alzheimer's disease mutations but not by inhibition of BACE1.
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- Molecular Neurodegeneration, 2020, v. 15, n. 1, p. N.PAG, doi. 10.1186/s13024-020-00393-5
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- Article
Life Extension Factor Klotho Prevents Mortality and Enhances Cognition in hAPP Transgenic Mice.
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- Journal of Neuroscience, 2015, v. 35, n. 6, p. 2358, doi. 10.1523/JNEUROSCI.5791-12.2015
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- Article
Ablation of Cellular Prion Protein Does Not Ameliorate Abnormal Neural Network Activity or Cognitive Dysfunction in the J20 Line of Human Amyloid Precursor Protein Transgenic Mice.
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- Journal of Neuroscience, 2011, v. 31, n. 29, p. 10427, doi. 10.1523/JNEUROSCI.1459-11.2011
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- Article
Amyloid-β/Fyn-Induced Synaptic, Network, and Cognitive Impairments Depend on Tau Levels in Multiple Mouse Models of Alzheimer's Disease.
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- Journal of Neuroscience, 2011, v. 31, n. 2, p. 700, doi. 10.1523/JNEUROSCI.4152-10.2011
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- Article