Found: 20
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Silencing at Drosophila telomeres: nuclear organization and chromatin structure play critical roles.
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- EMBO Journal, 1999, v. 18, n. 13, p. 3724, doi. 10.1093/emboj/18.13.3724
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- Article
In Silico and In Vivo Analysis of Amino Acid Substitutions That Cause Laminopathies.
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- International Journal of Molecular Sciences, 2021, v. 22, n. 20, p. 11226, doi. 10.3390/ijms222011226
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- Article
Domains of Heterochromatin Protein 1 Required for Drosophila melanogaster Heterochromatin Spreading.
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- Genetics, 2009, v. 182, n. 4, p. 967, doi. 10.1534/genetics.109.105338
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- Article
Tissue-Specific Defects Are Caused by Loss of the Drosophila MAN1 LEM Domain Protein.
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- Genetics, 2008, v. 180, n. 1, p. 133, doi. 10.1534/genetics.108.091371
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- Article
Molecular Genetic Analysis of the Nested Drosophila melanogaster Lamin C Gene.
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- Genetics, 2005, v. 171, n. 1, p. 185, doi. 10.1534/genetics.105.043208
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- Article
Enforcing silencing: dynamic HP1 complexes in Neurospora.
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- Nature Structural & Molecular Biology, 2012, v. 19, n. 5, p. 465, doi. 10.1038/nsmb.2291
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- Article
A Comparative Study of Drosophila and Human A-Type Lamins.
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- PLoS ONE, 2009, v. 4, n. 10, p. 1, doi. 10.1371/journal.pone.0007564
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- Article
Nucleosome positioning and gene regulation.
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- Journal of Cellular Biochemistry, 1994, v. 55, n. 1, p. 83, doi. 10.1002/jcb.240550110
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- Article
Heterochromatic silencing ofDrosophilaheat shock genes acts at the level of promoter potentiation.
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- Nucleic Acids Research, 1999, v. 27, n. 16, p. 3364, doi. 10.1093/nar/27.16.3364
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- Article
Architectural variations of inducible eukaryotic promoters: Preset and remodeling chromatin structures.
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- BioEssays, 1994, v. 16, n. 3, p. 165, doi. 10.1002/bies.950160306
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- Article
A Drosophila Model of Epidermolysis Bullosa Simplex.
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- Journal of Investigative Dermatology, 2015, v. 135, n. 8, p. 2031, doi. 10.1038/jid.2015.129
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- Article
Role of Drosophila HP1 in euchromatic gene expression.
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- Developmental Dynamics, 2005, v. 232, n. 3, p. 767
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- Article
Suppression of myopathic lamin mutations by muscle-specific activation of AMPK and modulation of downstream signaling.
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- Human Molecular Genetics, 2019, v. 28, n. 3, p. 351, doi. 10.1093/hmg/ddy332
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- Article
Myopathic lamin mutations impair nuclear stability in cells and tissue and disrupt nucleo-cytoskeletal coupling.
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- Human Molecular Genetics, 2013, v. 22, n. 12, p. 2335, doi. 10.1093/hmg/ddt079
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- Article
LMNA variants cause cytoplasmic distribution of nuclear pore proteins in Drosophila and human muscle.
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- Human Molecular Genetics, 2012, v. 21, n. 7, p. 1544, doi. 10.1093/hmg/ddr592
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- Article
Drosophila Models Reveal Properties of Mutant Lamins That Give Rise to Distinct Diseases.
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- Cells (2073-4409), 2023, v. 12, n. 8, p. 1142, doi. 10.3390/cells12081142
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- Article
Shining Light on the Dark Side of the Genome.
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- Cells (2073-4409), 2022, v. 11, n. 3, p. 330, doi. 10.3390/cells11030330
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- Article
Myopathic Lamin Mutations Cause Reductive Stress and Activate the Nrf2/Keap-1 Pathway.
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- PLoS Genetics, 2015, v. 11, n. 5, p. 1, doi. 10.1371/journal.pgen.1005231
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- Article
The Influence of a Genetic Variant in CCDC78 on LMNA -Associated Skeletal Muscle Disease.
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- International Journal of Molecular Sciences, 2024, v. 25, n. 9, p. 4930, doi. 10.3390/ijms25094930
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- Article
Increasing autophagy and blocking Nrf2 suppress laminopathy‐induced age‐dependent cardiac dysfunction and shortened lifespan.
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- Aging Cell, 2018, v. 17, n. 3, p. 1, doi. 10.1111/acel.12747
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- Article