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Identifying transgene insertions in Caenorhabditis elegans genomes with Oxford Nanopore sequencing.
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- PeerJ, 2024, p. 1, doi. 10.7717/peerj.18100
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- Article
Lipase regulation of cellular fatty acid homeostasis as a Parkinson's disease therapeutic strategy.
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- NPJ Parkinson's Disease, 2022, v. 8, n. 1, p. 1, doi. 10.1038/s41531-022-00335-6
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- Article
Genetic Defects in Mitochondrial Dynamics in Caenorhabditis elegans Impact Ultraviolet C Radiation- and 6-hydroxydopamine-Induced Neurodegeneration.
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- International Journal of Molecular Sciences, 2019, v. 20, n. 13, p. 3202, doi. 10.3390/ijms20133202
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- Article
Pharmacogenetic Analysis Reveals a Post-Developmental Role tor Rac GTPases in Caenorhabditis elegans GABAergic Neurotransmission.
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- Genetics, 2009, v. 183, n. 4, p. 1357, doi. 10.1534/genetics.109.106880
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- Article
Therapeutic genetic variation revealed in diverse Hsp104 homologs.
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- eLife, 2021, p. 1, doi. 10.7554/eLife.57457
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- Article
No Country for Old Worms: A Systematic Review of the Application of C. elegans to Investigate a Bacterial Source of Environmental Neurotoxicity in Parkinson's Disease.
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- Metabolites (2218-1989), 2018, v. 8, n. 4, p. 70, doi. 10.3390/metabo8040070
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- Article
P3-294: The impact of chip on neurodegenerative diseases
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- 2006
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- Abstract
P3-294: The impact of chip on neurodegenerative diseases
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- 2006
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- Publication type:
- Abstract
α-Synuclein is part of a diverse and highly conserved interaction network that includes PARK9 and manganese toxicity.
- Published in:
- Nature Genetics, 2009, v. 41, n. 3, p. 308, doi. 10.1038/ng.300
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- Article
Investigating Bacterial Sources of Toxicity as an Environmental Contributor to Dopaminergic Neurodegeneration.
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- PLoS ONE, 2009, v. 4, n. 10, p. 1, doi. 10.1371/journal.pone.0007227
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- Article
Found in Translation: The Utility of C. elegans Alpha-Synuclein Models of Parkinson's Disease.
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- Brain Sciences (2076-3425), 2019, v. 9, n. 4, p. 73, doi. 10.3390/brainsci9040073
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- Article
Deletion of the Ubiquitin Ligase CHIP Leads to the Accumulation, But Not the Aggregation, of Both Endogenous Phospho- and Caspase-3-Cleaved Tau Species.
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- Journal of Neuroscience, 2006, v. 26, n. 26, p. 6985, doi. 10.1523/JNEUROSCI.0746-06.2006
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- Article
Torsin-Mediated Protection from Cellular Stress in the Dopaminergic Neurons of Caenorhabditis elegans.
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- Journal of Neuroscience, 2005, v. 25, n. 15, p. 3801, doi. 10.1523/JNEUROSCI.5157-04.2005
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- Publication type:
- Article
Attenuation of Dopaminergic Neurodegeneration in a C. elegans Parkinson's Model through Regulation of Xanthine Dehydrogenase (XDH-1) Expression by the RNA Editase, ADR-2.
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- Journal of Developmental Biology, 2023, v. 11, n. 2, p. 20, doi. 10.3390/jdb11020020
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- Article
The effects of pdr1, djr1.1 and pink1 loss in manganese-induced toxicity and the role of α-synuclein in C. elegans.
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- Metallomics, 2014, v. 6, n. 3, p. 476, doi. 10.1039/c3mt00325f
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- Publication type:
- Article
Low-dose bafilomycin attenuates neuronal cell death associated with autophagy-lysosome pathway dysfunction.
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- Journal of Neurochemistry, 2010, v. 114, n. 4, p. 1193, doi. 10.1111/j.1471-4159.2010.06838.x
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- Article
C. elegans as a model organism to investigate molecular pathways involved with Parkinson's disease.
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- Developmental Dynamics, 2010, v. 239, n. 5, p. 1282, doi. 10.1002/dvdy.22231
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- Publication type:
- Article
Evolutionarily conserved nuclear migration genes required for early embryonic development in Caenorhabditis elegans.
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- Development Genes & Evolution, 2001, v. 211, n. 8/9, p. 434, doi. 10.1007/s004270100176
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- Article
From bugs to bedside: functional annotation of human genetic variation for neurological disorders using invertebrate models.
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- Human Molecular Genetics, 2022, v. 31, p. R37, doi. 10.1093/hmg/ddac203
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- Article
Distinct functional roles of Vps41-mediated neuroprotection in Alzheimer's and Parkinson's disease models of neurodegeneration.
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- Human Molecular Genetics, 2018, v. 27, n. 24, p. 4176, doi. 10.1093/hmg/ddy308
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- Publication type:
- Article
Alpha-synuclein inhibits Snx3-retromer-mediated retrograde recycling of iron transporters in S. cerevisiae and C. elegans models of Parkinson's disease.
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- Human Molecular Genetics, 2018, v. 27, n. 9, p. 1514, doi. 10.1093/hmg/ddy059
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- Publication type:
- Article
NCEH-1 modulates cholesterol metabolism and protects against α-synuclein toxicity in a C. elegans model of Parkinson's disease.
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- Human Molecular Genetics, 2017, v. 26, n. 19, p. 3823, doi. 10.1093/hmg/ddx269
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- Article
Glutaredoxin deficiency exacerbates neurodegeneration in C. elegans models of Parkinson's disease.
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- Human Molecular Genetics, 2015, v. 24, n. 5, p. 1322, doi. 10.1093/hmg/ddu542
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- Article
Identification of novel ATP13A2 interactors and their role in α-synuclein misfolding and toxicity.
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- Human Molecular Genetics, 2012, v. 21, n. 17, p. 3785, doi. 10.1093/hmg/dds206
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- Publication type:
- Article
The early-onset torsion dystonia-associated protein, torsinA, is a homeostatic regulator of endoplasmic reticulum stress response.
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- Human Molecular Genetics, 2012, v. 21, n. 5, p. 1201, doi. 10.1093/hmg/ddr571
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- Publication type:
- Article
Inhibitors of LRRK2 kinase attenuate neurodegeneration and Parkinson-like phenotypes in Caenorhabditis elegans and Drosophila Parkinson's disease models.
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- Human Molecular Genetics, 2011, v. 20, n. 20, p. 3933, doi. 10.1093/hmg/ddr312
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- Publication type:
- Article
Gene-by-environment interactions that disrupt mitochondrial homeostasis cause neurodegeneration in C. elegans Parkinson’s models.
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- Cell Death & Disease, 2018, v. 9, n. 5, p. 1, doi. 10.1038/s41419-018-0619-5
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- Article
Chemical Compensation of Mitochondrial Phospholipid Depletion in Yeast and Animal Models of Parkinson’s Disease.
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- PLoS ONE, 2016, v. 11, n. 10, p. 1, doi. 10.1371/journal.pone.0164465
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- Article
Xanthine Dehydrogenase Is a Modulator of Dopaminergic Neurodegeneration in Response to Bacterial Metabolite Exposure in C. elegans.
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- Cells (2073-4409), 2023, v. 12, n. 8, p. 1170, doi. 10.3390/cells12081170
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- Article
Dysregulation of the Mitochondrial Unfolded Protein Response Induces Non-Apoptotic Dopaminergic Neurodegeneration in C. elegans Models of Parkinson's Disease.
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- Journal of Neuroscience, 2017, v. 37, n. 46, p. 11085, doi. 10.1523/JNEUROSCI.1294-17.2017
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- Article
Ubiquitin conjugating enzymes participate in polyglutamine protein aggregation.
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- BMC Cell Biology, 2007, v. 8, p. 32, doi. 10.1186/1471-2121-8-32
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- Article
Rescue of ApoE4-related lysosomal autophagic failure in Alzheimer's disease by targeted small molecules.
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- Communications Biology, 2024, v. 7, n. 1, p. 1, doi. 10.1038/s42003-024-05767-9
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- Publication type:
- Article
The early-onset torsion dystonia-associated protein, torsinA, is a homeostatic regulator of endoplasmic reticulum stress response.
- Published in:
- Human Molecular Genetics, 2010, v. 19, n. 18, p. 3502, doi. 10.1093/hmg/ddq266
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- Publication type:
- Article
Epileptic-like convulsions associated with LIS-1 in the cytoskeletal control of neurotransmitter signaling in Caenorhabditis elegans.
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- Human Molecular Genetics, 2004, v. 13, n. 18, p. 2043, doi. 10.1093/hmg/ddh209
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- Publication type:
- Article
Suppression of polyglutamine-induced protein aggregation in Caenorhabditis elegans by torsin proteins.
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- Human Molecular Genetics, 2003, v. 12, n. 3, p. 307, doi. 10.1093/hmg/ddg027
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- Publication type:
- Article
Rapid selection of cyclic peptides that reduce α-synuclein toxicity in yeast and animal models.
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- Nature Chemical Biology, 2009, v. 5, n. 9, p. 655, doi. 10.1038/nchembio.193
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- Publication type:
- Article
Systemic RNA Interference Defective (SID) genes modulate dopaminergic neurodegeneration in C. elegans.
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- PLoS Genetics, 2022, v. 18, n. 8, p. 1, doi. 10.1371/journal.pgen.1010115
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- Publication type:
- Article
Correction to: The Small GTPase RAC1/CED-10 Is Essential in Maintaining Dopaminergic Neuron Function and Survival Against α-Synuclein-Induced Toxicity.
- Published in:
- Molecular Neurobiology, 2018, v. 55, n. 9, p. 7553, doi. 10.1007/s12035-018-1010-3
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- Article
The Small GTPase RAC1/CED-10 Is Essential in Maintaining Dopaminergic Neuron Function and Survival Against α-Synuclein-Induced Toxicity.
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- Molecular Neurobiology, 2018, v. 55, n. 9, p. 7533, doi. 10.1007/s12035-018-0881-7
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- Publication type:
- Article
Lysosomal impairment in Parkinson's disease.
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- Movement Disorders, 2013, v. 28, n. 6, p. 725, doi. 10.1002/mds.25462
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- Publication type:
- Article
RTCB-1 Mediates Neuroprotection via XBP-1 mRNA Splicing in the Unfolded Protein Response Pathway.
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- Journal of Neuroscience, 2014, v. 34, n. 48, p. 16076, doi. 10.1523/JNEUROSCI.1945-14.2014
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- Publication type:
- Article
Functional Analysis of VPS41-Mediated Neuroprotection in Caenorhabditis elegans and Mammalian Models of Parkinson's Disease.
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- Journal of Neuroscience, 2012, v. 32, n. 6, p. 2142, doi. 10.1523/JNEUROSCI.2606-11.2012
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- Publication type:
- Article