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Glutathionylation of the L-type Ca<sup>2+</sup> Channel in Oxidative Stress-Induced Pathology of the Heart.
- Published in:
- International Journal of Molecular Sciences, 2014, v. 15, n. 10, p. 19203, doi. 10.3390/ijms151019203
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- Article
Differential regulation of the slow and rapid components of guinea-pig cardiac delayed rectifier K<sup>+</sup> channels by hypoxia.
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- Journal of Physiology, 2004, v. 554, n. 3, p. 743, doi. 10.1113/jphysiol.2003.055442
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- Article
Auto-regulation in the powerhouse.
- Published in:
- eLife, 2017, p. 1, doi. 10.7554/eLife.28757
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- Article
Physiology: A perspective from the Australian experience.
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- Physiological Reports, 2024, v. 12, n. 17, p. 1, doi. 10.14814/phy2.70045
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- Article
Stress signaling and cellular proliferation reverse the effects of mitochondrial mistranslation.
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- EMBO Journal, 2019, v. 38, n. 24, p. N.PAG, doi. 10.15252/embj.2019102155
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- Article
The impact of non-severe burn injury on cardiac function and long-term cardiovascular pathology.
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- Scientific Reports, 2016, p. 34650, doi. 10.1038/srep34650
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- Article
Evidence for redox sensing by a human cardiac calcium channel.
- Published in:
- Scientific Reports, 2016, p. 19067, doi. 10.1038/srep19067
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- Article
The cardiac L-type calcium channel alpha subunit is a target for direct redox modification during oxidative stress—the role of cysteine residues in the alpha interacting domain.
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- Clinical & Experimental Pharmacology & Physiology, 2017, v. 44, p. 46, doi. 10.1111/1440-1681.12750
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- Article
Role of the cytoskeleton in communication between L-type Ca<sup>2+</sup> channels and mitochondria.
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- Clinical & Experimental Pharmacology & Physiology, 2013, v. 40, n. 4, p. 295, doi. 10.1111/1440-1681.12072
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- Article
Cross-talk between L-type Ca<sup>2+</sup> channels and mitochondria.
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- Clinical & Experimental Pharmacology & Physiology, 2010, v. 37, n. 2, p. 229, doi. 10.1111/j.1440-1681.2009.05277.x
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- Article
EVIDENCE FOR THE REGULATION OF L-TYPE Ca<sup>2+</sup> CHANNELS IN THE HEART BY REACTIVE OXYGEN SPECIES: MECHANISM FOR MEDIATING PATHOLOGY.
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- 2008
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- Publication type:
- Other
REACTIVE OXYGEN SPECIES IN CARDIAC SIGNALLING: FROM MITOCHONDRIA TO PLASMA MEMBRANE ION CHANNELS.
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- Clinical & Experimental Pharmacology & Physiology, 2006, v. 33, n. 1/2, p. 146, doi. 10.1111/j.1440-1681.2006.04341.x
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- Article
A new methodology for assessment of the performance of heartbeat classification systems.
- Published in:
- BMC Medical Informatics & Decision Making, 2008, v. 8, n. 1, p. 1, doi. 10.1186/1472-6947-8-7
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- Article
Concerted regulation of mitochondrial and nuclear non‐coding RNAs by a dual‐targeted RNase Z.
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- EMBO Reports, 2018, v. 19, n. 10, p. 1, doi. 10.15252/embr.201846198
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- Article
Myoglobinopathy is an adult-onset autosomal dominant myopathy with characteristic sarcoplasmic inclusions.
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- Nature Communications, 2019, v. 10, n. 1, p. N.PAG, doi. 10.1038/s41467-019-09111-2
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- Article
Role of NAD(P)H oxidase in the regulation of cardiac L-type Ca<sup>2+</sup> channel function during acute hypoxia
- Published in:
- Cardiovascular Research, 2005, v. 67, n. 4, p. 624, doi. 10.1016/j.cardiores.2005.04.025
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- Article
Volume adaptation of neonatal cardiomyocyte spheroids in 3D stiffness gradient GelMA.
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- Journal of Biomedical Materials Research, Part A, 2023, v. 111, n. 6, p. 801, doi. 10.1002/jbm.a.37456
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- Article
Neuronal nitric oxide synthase regulation of calcium cycling in ventricular cardiomyocytes is independent of Cav1.2 channel modulation under basal conditions.
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- Pflügers Archiv: European Journal of Physiology, 2020, v. 472, n. 1, p. 61, doi. 10.1007/s00424-019-02335-7
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- Article
What's the difference? Understanding sexual dimorphism in physiology.
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- Journal of Physiology, 2023, v. 601, n. 3, p. 417, doi. 10.1113/JP284342
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- Article
Loss of the RNA-binding protein TACO1 causes late-onset mitochondrial dysfunction in mice.
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- Nature Communications, 2016, v. 7, n. 6, p. 11884, doi. 10.1038/ncomms11884
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- Article
Verapamil is Less Effective than Triamcinolone for Prevention of Keloid Scar Recurrence After Excision in a Randomized Controlled Trial.
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- Acta Dermato-Venereologica, 2016, v. 96, n. 6, p. 774, doi. 10.2340/00015555-2384
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- Article
Mitochondria at the Crossroads of Survival and Demise.
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- Oxidative Medicine & Cellular Longevity, 2019, p. 1, doi. 10.1155/2019/2608187
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- Publication type:
- Article
Decreased Myocardial Injury and Improved Contractility After Administration of a Peptide Derived Against the Alpha-Interacting Domain of the L-Type Calcium Channel.
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- Journal of the American Heart Association, 2014, v. 3, n. 3, p. 1, doi. 10.1161/JAHA.114.000961
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- Article
L-type Ca<sup>2+</sup> channel–mediated Ca<sup>2+</sup> influx adjusts neuronal mitochondrial function to physiological and pathophysiological conditions.
- Published in:
- Science Signaling, 2020, v. 13, n. 618, p. 1, doi. 10.1126/scisignal.aaw6923
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- Article
Preventative therapeutic approaches for hypertrophic cardiomyopathy.
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- Journal of Physiology, 2021, v. 599, n. 14, p. 3495, doi. 10.1113/JP279410
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- Article
Unravelling the mysteries of mitochondria in health and disease.
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- Journal of Physiology, 2021, v. 599, n. 14, p. 3447, doi. 10.1113/JP281833
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- Article
Fidelity and coordination of mitochondrial protein synthesis in health and disease.
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- Journal of Physiology, 2021, v. 599, n. 14, p. 3449, doi. 10.1113/JP280359
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- Article
The L-type Ca<sup>2+</sup> channel facilitates abnormal metabolic activity in the cTnI-G203S mouse model of hypertrophic cardiomyopathy.
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- Journal of Physiology, 2016, v. 594, n. 14, p. 4051, doi. 10.1113/JP271681
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- Article
Deranged sodium to sudden death.
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- Journal of Physiology, 2015, v. 593, n. 6, p. 1331, doi. 10.1113/jphysiol.2014.281204
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- Article
TGF‐β‐induced fibrotic stress increases G‐quadruplex formation in human fibroblasts.
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- FEBS Letters, 2019, v. 593, n. 22, p. 3149, doi. 10.1002/1873-3468.13658
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- Article
Mutation in MRPS34 Compromises Protein Synthesis and Causes Mitochondrial Dysfunction.
- Published in:
- PLoS Genetics, 2015, v. 11, n. 3, p. 1, doi. 10.1371/journal.pgen.1005089
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- Article
Acute hypoxia differentially regulates K<sup>+</sup> channels. Implications with respect to cardiac arrhythmia.
- Published in:
- European Biophysics Journal, 2005, v. 34, n. 5, p. 369, doi. 10.1007/s00249-005-0462-3
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- Publication type:
- Article
Dystrophic Cardiomyopathy--Potential Role of Calcium in Pathogenesis, Treatment and Novel Therapies.
- Published in:
- Genes, 2017, v. 8, n. 4, p. 108, doi. 10.3390/genes8040108
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- Article
A maladaptive feedback mechanism between the extracellular matrix and cytoskeleton contributes to hypertrophic cardiomyopathy pathophysiology.
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- Communications Biology, 2023, v. 6, n. 1, p. 1, doi. 10.1038/s42003-022-04278-9
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- Article
Mitochondrial mistranslation modulated by metabolic stress causes cardiovascular disease and reduced lifespan.
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- Aging Cell, 2021, v. 20, n. 7, p. 1, doi. 10.1111/acel.13408
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- Article
Cytoskeletal disarray increases arrhythmogenic vulnerability during sympathetic stimulation in a model of hypertrophic cardiomyopathy.
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- Scientific Reports, 2023, v. 13, n. 1, p. 1, doi. 10.1038/s41598-023-38296-2
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- Article
Gene editing and cardiac disease modelling for the interpretation of genetic variants of uncertain significance in congenital heart disease.
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- Stem Cell Research & Therapy, 2023, v. 14, n. 1, p. 1, doi. 10.1186/s13287-023-03592-1
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- Article