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Control of experimental spasticity by targeting the degradation of endocannabinoids using selective fatty acid amide hydrolase inhibitors.
Published in:
Multiple Sclerosis Journal, 2013, v. 19, n. 14, p. 1896, doi. 10.1177/1352458513485982
By:
- Pryce, G;
- Cabranes, A;
- Fernández-Ruiz, J;
- Bisogno, T;
- Di Marzo, V;
- Long, JZ;
- Cravatt, BF;
- Giovannoni, G;
- Baker, D
Publication type:
Article
β-Amyloid exacerbates inflammation in astrocytes lacking fatty acid amide hydrolase through a mechanism involving PPAR-α, PPAR-γ and TRPV1, but not CB₁ or CB₂ receptors.
Published in:
2012
By:
- Benito C;
- Tolón RM;
- Castillo AI;
- Ruiz-Valdepeñas L;
- Martínez-Orgado JA;
- Fernández-Sánchez FJ;
- Vázquez C;
- Cravatt BF;
- Romero J;
- Benito, Cristina;
- Tolón, Rosa María;
- Castillo, Ana Isabel;
- Ruiz-Valdepeñas, Lourdes;
- Martínez-Orgado, José Antonio;
- Fernández-Sánchez, Francisco Javier;
- Vázquez, Carmen;
- Cravatt, Benjamin F;
- Romero, Julián
Publication type:
journal article
Inhibiting fatty acid amide hydrolase normalizes endotoxin-induced enhanced gastrointestinal motility in mice.
Published in:
British Journal of Pharmacology, 2012, v. 165, n. 5, p. 1556, doi. 10.1111/j.1476-5381.2011.01644.x
By:
- Bashashati, M;
- Storr, MA;
- Nikas, SP;
- Wood, JT;
- Godlewski, G;
- Liu, J;
- Ho, W;
- Keenan, CM;
- Zhang, H;
- Alapafuja, SO;
- Cravatt, BF;
- Lutz, B;
- Mackie, K;
- Kunos, G;
- Patel, KD;
- Makriyannis, A;
- Davison, JS;
- Sharkey, KA
Publication type:
Article
COX-2 and fatty acid amide hydrolase can regulate the time course of depolarization-induced suppression of excitation.
Published in:
2011
By:
- Straiker, A;
- Wager-Miller, J;
- Hu, SS;
- Blankman, JL;
- Cravatt, BF;
- Mackie, K;
- Hu, S S;
- Blankman, J L;
- Cravatt, B F
Publication type:
journal article
Biochanin A, a naturally occurring inhibitor of fatty acid amide hydrolase.
Published in:
2010
By:
- Thors, L;
- Burston, JJ;
- Alter, BJ;
- McKinney, MK;
- Cravatt, BF;
- Ross, RA;
- Pertwee, RG;
- Gereau 4th, RW;
- Wiley, JL;
- Fowler, CJ;
- Burston, J J;
- Alter, B J;
- McKinney, M K;
- Cravatt, B F;
- Ross, R A;
- Pertwee, R G;
- Gereau, R W 4th;
- Wiley, J L;
- Fowler, C J
Publication type:
journal article

Found: 5

Control of experimental spasticity by targeting the degradation of endocannabinoids using selective fatty acid amide hydrolase inhibitors.

β-Amyloid exacerbates inflammation in astrocytes lacking fatty acid amide hydrolase through a mechanism involving PPAR-α, PPAR-γ and TRPV1, but not CB₁ or CB₂ receptors.

Inhibiting fatty acid amide hydrolase normalizes endotoxin-induced enhanced gastrointestinal motility in mice.

COX-2 and fatty acid amide hydrolase can regulate the time course of depolarization-induced suppression of excitation.

Biochanin A, a naturally occurring inhibitor of fatty acid amide hydrolase.