Found: 15
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Oxidative Genome Damage and its Repair in Neurodegenerative Diseases: Function of Transition Metals as a Double-Edged Sword.
- Published in:
- Journal of Alzheimer's Disease, 2011, v. 24, n. S2, p. 183
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- Publication type:
- Article
Oxidative Genome Damage and its Repair in Neurodegenerative Diseases: Function of Transition Metals as a Double-Edged Sword.
- Published in:
- Journal of Alzheimer's Disease, 2011, v. 23, n. S1, p. 183, doi. 10.3233/JAD-2011-110281
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- Publication type:
- Article
Oxidative genome damage and its repair in neurodegenerative diseases: function of transition metals as a double-edged sword.
- Published in:
- 2011
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- Publication type:
- journal article
Microhomology-mediated end joining is activated in irradiated human cells due to phosphorylation-dependent formation of the XRCC1 repair complex.
- Published in:
- Nucleic Acids Research, 2017, v. 45, n. 5, p. 2585, doi. 10.1093/nar/gkw1262
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- Publication type:
- Article
Regulation of oxidized base damage repair by chromatin assembly factor 1 subunit A.
- Published in:
- Nucleic Acids Research, 2017, v. 45, n. 2, p. 739, doi. 10.1093/nar/gkw1024
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- Publication type:
- Article
Specific inhibition of neil-initiated repair of oxidized base damage in human genome by copper and iron and protection using curcumin: potential etiological linkage to neurodegenerative diseases
- Published in:
- 2010
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- Publication type:
- Abstract
FUS unveiled in mitochondrial DNA repair and targeted ligase-1 expression rescues repair-defects in FUS-linked motor neuron disease.
- Published in:
- Nature Communications, 2024, v. 15, n. 1, p. 1, doi. 10.1038/s41467-024-45978-6
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- Publication type:
- Article
New Perspectives on Oxidized Genome Damage and Repair Inhibition by Pro-Oxidant Metals in Neurological Diseases.
- Published in:
- Biomolecules (2218-273X), 2014, v. 4, n. 3, p. 678, doi. 10.3390/biom4030678
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- Publication type:
- Article
Loss of endosomal recycling factor RAB11 coupled with complex regulation of MAPK/ERK/AKT signaling in postmortem spinal cord specimens of sporadic amyotrophic lateral sclerosis patients.
- Published in:
- Molecular Brain, 2019, v. 12, n. 1, p. N.PAG, doi. 10.1186/s13041-019-0475-y
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- Publication type:
- Article
Correction: The Role of the Mammalian DNA End-processing Enzyme Polynucleotide Kinase 3'-Phosphatase in Spinocerebellar Ataxia Type 3 Pathogenesis.
- Published in:
- 2024
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- Publication type:
- Correction Notice
Human DNA Glycosylase NEIL1's Interactions with Downstream Repair Proteins Is Critical for Efficient Repair of Oxidized DNA Base Damage and Enhanced Cell Survival.
- Published in:
- Biomolecules (2218-273X), 2012, v. 2, n. 4, p. 564, doi. 10.3390/biom2040564
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- Publication type:
- Article
Amyotrophic lateral sclerosis-associated TDP-43 mutation Q331K prevents nuclear translocation of XRCC4-DNA ligase 4 complex and is linked to genome damage-mediated neuronal apoptosis.
- Published in:
- Human Molecular Genetics, 2019, v. 28, n. 18, p. 3161, doi. 10.1093/hmg/ddz141
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- Publication type:
- Article
Amyotrophic lateral sclerosis-associated TDP-43 mutation Q331K prevents nuclear translocation of XRCC4-DNA ligase 4 complex and is linked to genome damage-mediated neuronal apoptosis.
- Published in:
- Human Molecular Genetics, 2019, v. 28, n. 15, p. 2459, doi. 10.1093/hmg/ddz062
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- Publication type:
- Article
Mutant FUS causes DNA ligation defects to inhibit oxidative damage repair in Amyotrophic Lateral Sclerosis.
- Published in:
- Nature Communications, 2018, v. 9, n. 1, p. 1, doi. 10.1038/s41467-018-06111-6
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- Publication type:
- Article
The Role of the Mammalian DNA End-processing Enzyme Polynucleotide Kinase 3’-Phosphatase in Spinocerebellar Ataxia Type 3 Pathogenesis.
- Published in:
- PLoS Genetics, 2015, v. 11, n. 1, p. 1, doi. 10.1371/journal.pgen.1004749
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- Publication type:
- Article