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MOAP‐1‐mediated dissociation of p62/SQSTM1 bodies releases Keap1 and suppresses Nrf2 signaling.
- Published in:
- EMBO Reports, 2021, v. 22, n. 1, p. 1, doi. 10.15252/embr.202050854
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- Article
p62/SQSTM1 in liver diseases: the usual suspect with multifarious identities.
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- FEBS Journal, 2023, v. 290, n. 4, p. 892, doi. 10.1111/febs.16317
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- Article
Modulator of Apoptosis 1: A Highly Regulated RASSF1A-Interacting BH3-Like Protein.
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- Molecular Biology International, 2012, p. 1, doi. 10.1155/2012/536802
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- Article
BRIEF REVIEW PROTEINS OF THE BCL-2 FAMILY IN APOPTOSIS SIGNALLING: FROM MECHANISTIC INSIGHTS TO THERAPEUTIC OPPORTUNITIES.
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- Clinical & Experimental Pharmacology & Physiology, 2004, v. 31, n. 3, p. 119, doi. 10.1111/j.1440-1681.2004.03975.x
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- Article
Regulation of Cyclic AMP by the μ-Opioid Receptor in Human Neuroblastoma SH-SY5Y Cells.
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- Journal of Neurochemistry, 1990, v. 55, n. 4, p. 1390, doi. 10.1111/j.1471-4159.1990.tb03151.x
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- Article
Differentiation of Human Neuroblastoma Cells: Marked Potentiation of Prostaglandin E-Stimulated Accumulation of Cyclic AMP by Retinoic Acid.
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- Journal of Neurochemistry, 1988, v. 51, n. 6, p. 1892, doi. 10.1111/j.1471-4159.1988.tb01174.x
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- Article
Correction: The Tumor Suppressor Gene, RASSF1A, Is Essential for Protection against Inflammation -Induced Injury.
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- PLoS ONE, 2015, v. 10, n. 6, p. 1, doi. 10.1371/journal.pone.0131150
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- Article
The Tumor Suppressor Gene, RASSF1A, Is Essential for Protection against Inflammation -Induced Injury.
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- PLoS ONE, 2013, v. 8, n. 10, p. 1, doi. 10.1371/journal.pone.0075483
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- Article
Auto-FACE: An NMR Based Binding Site Mapping Program for Fast Chemical Exchange Protein-Ligand Systems.
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- PLoS ONE, 2010, v. 5, n. 2, p. 1, doi. 10.1371/journal.pone.0008943
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- Article
Mitochondrial translocation of cofilin is an early step in apoptosis induction.
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- Nature Cell Biology, 2003, v. 5, n. 12, p. 1083, doi. 10.1038/ncb1070
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- Article
Absence of Stress Response in Dorsal Raphe Nucleus in Modulator of Apoptosis 1-Deficient Mice.
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- Molecular Neurobiology, 2019, v. 56, n. 3, p. 2185, doi. 10.1007/s12035-018-1205-7
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- Article