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- Title
Ambient prenatal air pollution exposure is associated with low cord blood IL‐17a in infants.
- Authors
Gorlanova, Olga; Oller, Heide; Marten, Andrea; Müller, Loretta; Laine‐Carmelli, Jessica; Decrue, Fabienne; Salem, Yasmin; Vienneau, Danielle; Hoogh, Kees de; Gisler, Amanda; Usemann, Jakob; Korten, Insa; Yammine, Sophie; Nahum, Uri; Künstle, Noemi; Sinues, Pablo; Schulzke, Sven; Latzin, Philipp; Fuchs, Oliver; Röösli, Martin
- Abstract
Epidemiological studies have shown that prenatal exposure to air pollution such as particulate matter (PM) and nitrogen dioxide (NO SB 2 sb ) is associated with impaired lung function after birth[1] and the development of childhood asthma and wheeze, for example.[2] Biological mechanisms linking prenatal air pollution exposure to respiratory morbidity are not completely understood. Some epidemiological studies have reported that air pollution exposure increases the serum levels of IL-17a in asthmatic children.[4] However, the effect of prenatal air pollution on IL-17a levels in humans is still unknown. Ambient prenatal air pollution exposure is associated with low cord blood IL-17a in infants.
- Subjects
CORD blood; AIR pollution; INFANTS; RHINOVIRUSES; WHEEZE; LUNG development; DELIVERY (Obstetrics)
- Publication
Pediatric Allergy & Immunology, 2023, Vol 34, Issue 1, p1
- ISSN
0905-6157
- Publication type
Article
- DOI
10.1111/pai.13902