The article discusses a research study on the role of the gene Rho1 as a stress signal mediator to target of rapamycin complex 1 (TORC1). It references the study "The TOR complex 1 is a direct target of Rho1 GTPase," by Y. Jiang and colleagues in the same issue. The study suggests that Rho1 binds to Kog1, a TORC1 component, in order to respond to stress, thus releasing Tap42-2A, a TORC1 effector.