Found: 23
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The MEK1-ERK1/2 signaling pathway promotes compensated cardiac hypertrophy in transgenic mice.
- Published in:
- EMBO Journal, 2000, v. 19, n. 23, p. 6341, doi. 10.1093/emboj/19.23.6341
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- Article
Integration of flux measurements to resolve changes in anabolic and catabolic metabolism in cardiac myocytes.
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- Biochemical Journal, 2017, v. 474, n. 16, p. 2785, doi. 10.1042/BCJ20170474
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- Article
High glucose induces mitochondrial dysfunction independently of protein O-GlcNAcylation.
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- Biochemical Journal, 2015, v. 467, n. 1, p. 115, doi. 10.1042/BJ20141018
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- Article
PDGF-mediated autophagy regulates vascular smooth muscle cell phenotype and resistance to oxidative stress.
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- Biochemical Journal, 2013, v. 451, n. 3, p. 375, doi. 10.1042/BJ20121344
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- Article
The management of unerupted maxillary canines.
- Published in:
- Dental Nursing, 2011, v. 7, n. 1, p. 18, doi. 10.12968/denn.2011.7.1.18
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- Article
The multidisciplinary management of hypodontia.
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- Dental Nursing, 2009, v. 5, n. 12, p. 682, doi. 10.12968/denn.2009.5.12.45386
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- Article
Protein O-GlcNAcylation Is a Novel Cytoprotective Signal in Cardiac Stem Cells.
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- Stem Cells, 2013, v. 31, n. 4, p. 765, doi. 10.1002/stem.1325
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- Article
A study to investigate the bond strengths of orthodontic brackets bonded to prosthetic acrylic teeth.
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- Journal of Orthodontics, 2015, v. 42, n. 3, p. 192, doi. 10.1179/1465313315Y.0000000003
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- Article
A comparison of skeletal maturation m patients with tooth agenesis and unaffected controls assessed by the cervical vertebral maturation (CVM) index.
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- Journal of Orthodontics, 2013, v. 40, n. 4, p. 286, doi. 10.1179/1465313313Y.0000000070
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- Article
An ex vivo laboratory study to determine the static frictional resistance of a variable ligation orthodontic bracket system.
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- Journal of Orthodontics, 2008, v. 35, n. 2, p. 112, doi. 10.1179/146531207225022527
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- Article
Excellent Sheep: The Miseducation of the American Elite and the Way to a Meaningful Life.
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- Critical Questions in Education, 2015, v. 6, n. 2, p. 103
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- Article
O-GlcNAcylation Negatively Regulates Cardiomyogenic Fate in Adult Mouse Cardiac Mesenchymal Stromal Cells.
- Published in:
- PLoS ONE, 2015, v. 10, n. 10, p. 1, doi. 10.1371/journal.pone.0142939
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- Article
High Fat Feeding in Mice Is Insufficient to Induce Cardiac Dysfunction and Does Not Exacerbate Heart Failure.
- Published in:
- PLoS ONE, 2013, v. 8, n. 12, p. 1, doi. 10.1371/journal.pone.0083174
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- Article
Reduced Cardiac Fructose 2,6 Bisphosphate Increases Hypertrophy and Decreases Glycolysis following Aortic Constriction.
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- PLoS ONE, 2013, v. 8, n. 1, p. 1, doi. 10.1371/journal.pone.0053951
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- Article
The COX-2/PGI2 Receptor Axis Plays an Obligatory Role in Mediating the Cardioprotection Conferred by the Late Phase of Ischemic Preconditioning.
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- PLoS ONE, 2012, v. 7, n. 7, p. 1, doi. 10.1371/journal.pone.0041178
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- Article
Humanness Under Assault: An Essay Questioning Technology in the Classroom.
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- Bulletin of Science, Technology & Society, 1998, v. 18, n. 2, p. 87, doi. 10.1177/027046769801800203
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- Article
Cardiomyocyte Oga haploinsufficiency increases O-GlcNAcylation but hastens ventricular dysfunction following myocardial infarction.
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- PLoS ONE, 2020, v. 15, n. 11, p. 1, doi. 10.1371/journal.pone.0242250
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- Article
Endothelial dysfunction as a nexus for endothelial cell-cardiomyocyte miscommunication.
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- Frontiers in Physiology, 2014, v. 5, p. 1, doi. 10.3389/fphys.2014.00328
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- Article
O-GlcNAcase Exacerbates Post-Hypoxic Cardiac Myocyte Death.
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- FASEB Journal, 2007, v. 21, n. 6, p. A1376
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- Article
O-GlcNAc Transferase is a Pro-Survival Enzyme in Post-Hypoxic Cardiac Myocytes.
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- FASEB Journal, 2007, v. 21, n. 6, p. A800, doi. 10.1096/fasebj.21.6.a800
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- Article
E2f1 deletion attenuates infarct-induced ventricular remodeling without affecting O-GlcNAcylation.
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- Basic Research in Cardiology, 2019, v. 114, n. 4, p. N.PAG, doi. 10.1007/s00395-019-0737-y
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- Article
Using gene-targeted mice to investigate the pathophysiology of myocardial reperfusion injury.
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- Basic Research in Cardiology, 2000, v. 95, n. 6, p. 499, doi. 10.1007/s003950070027
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- Article
Chronic Benzene Exposure Aggravates Pressure Overload-Induced Cardiac Dysfunction.
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- Toxicological Sciences, 2022, v. 185, n. 1, p. 64, doi. 10.1093/toxsci/kfab125
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- Article