Found: 26
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Cardiac glial cells release neurotrophic S100B upon catheter-based treatment of atrial fibrillation.
- Published in:
- Science Translational Medicine, 2019, v. 11, n. 493, p. N.PAG, doi. 10.1126/scitranslmed.aav7770
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- Publication type:
- Article
Decreasing microtubule detyrosination modulates Na<sub>v</sub>1.5 subcellular distribution and restores sodium current in mdx cardiomyocytes.
- Published in:
- Cardiovascular Research, 2024, v. 120, n. 7, p. 723, doi. 10.1093/cvr/cvae043
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- Publication type:
- Article
Absence of Functional Nav1.8 Channels in Non-diseased Atrial and Ventricular Cardiomyocytes.
- Published in:
- Cardiovascular Drugs & Therapy, 2019, v. 33, n. 6, p. 649, doi. 10.1007/s10557-019-06925-6
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- Publication type:
- Article
Mechanisms of inherited cardiac conduction disease.
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- EP: Europace, 2005, v. 7, n. 2, p. 122, doi. 10.1016/j.eupc.2004.11.004
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- Publication type:
- Article
Role of Ca2+-activated Cl- current in ventricular action potentials of sheep during adrenoceptor stimulation.
- Published in:
- Experimental Physiology, 2001, v. 86, n. 2, p. 151, doi. 10.1113/eph8602113
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- Publication type:
- Article
Disruption of cardiac cholinergic neurons enhances susceptibility to ventricular arrhythmias.
- Published in:
- Nature Communications, 2017, v. 8, n. 1, p. 14155, doi. 10.1038/ncomms14155
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- Publication type:
- Article
A Heterozygous Deletion Mutation in the Cardiac Sodium Channel Gene SCN5A with Loss- and Gain-of-Function Characteristics Manifests as Isolated Conduction Disease, without Signs of Brugada or Long QT Syndrome.
- Published in:
- PLoS ONE, 2013, v. 8, n. 6, p. 1, doi. 10.1371/journal.pone.0067963
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- Article
Organ Explant Culture of Neonatal Rat Ventricles: A New Model to Study Gene and Cell Therapy.
- Published in:
- PLoS ONE, 2013, v. 8, n. 3, p. 1, doi. 10.1371/journal.pone.0059290
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- Article
Ion Channels, the QT Interval, and Arrhythmias.
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- Pacing & Clinical Electrophysiology, 1997, v. 20, n. 8, p. 2048, doi. 10.1111/j.1540-8159.1997.tb03625.x
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- Article
Is IGF‐1 a useful inhibitor of Na<sup>+</sup>/H<sup>+</sup>‐exchanger activity?
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- 2018
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- Publication type:
- Editorial
Protease XIV abolishes NHE inhibition by empagliflozin in cardiac cells.
- Published in:
- Frontiers in Physiology, 2023, p. 01, doi. 10.3389/fphys.2023.1179131
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- Publication type:
- Article
The opioid tramadol blocks the cardiac sodium channel Na<sub>v</sub>1.5 in HEK293 cells.
- Published in:
- EP: Europace, 2023, v. 25, n. 9, p. 1, doi. 10.1093/europace/euad209
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- Publication type:
- Article
Anti-arrhythmic potential of the late sodium current inhibitor GS-458967 inmurine Scn5a-1798insD<sup>+/-</sup> and human SCN5A-1795insD<sup>+/-</sup> iPSC-derived cardiomyocytes.
- Published in:
- Cardiovascular Research, 2017, v. 113, n. 7, p. 829, doi. 10.1093/cvr/cvx077
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- Publication type:
- Article
Tubulin polymerization modifies cardiac sodium channel expression and gating.
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- Cardiovascular Research, 2010, v. 85, n. 4, p. 691, doi. 10.1093/cvr/cvp352
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- Publication type:
- Article
Intracellular calcium modulation of voltage-gated sodium channels in ventricular myocytes.
- Published in:
- Cardiovascular Research, 2009, v. 81, n. 1, p. 72, doi. 10.1093/cvr/cvn274
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- Publication type:
- Article
Characterization of a novel SCN5A mutation associated with Brugada syndrome reveals involvement of DIIIS4–S5 linker in slow inactivation
- Published in:
- Cardiovascular Research, 2007, v. 76, n. 3, p. 418, doi. 10.1016/j.cardiores.2007.08.005
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- Publication type:
- Article
Incorporated sarcolemmal fish oil fatty acids shorten pig ventricular action potentials
- Published in:
- Cardiovascular Research, 2006, v. 70, n. 3, p. 509, doi. 10.1016/j.cardiores.2006.02.022
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- Publication type:
- Article
Substitution of a conserved alanine in the domain IIIS4–S5 linker of the cardiac sodium channel causes long QT syndrome
- Published in:
- Cardiovascular Research, 2005, v. 67, n. 3, p. 459, doi. 10.1016/j.cardiores.2005.01.017
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- Publication type:
- Article
Effects of cell-to-cell uncoupling and catecholamines on Purkinje and ventricular action potentials: implications for phase-1b arrhythmias.
- Published in:
- Cardiovascular Research, 2001, v. 51, n. 1, p. 30, doi. 10.1016/S0008-6363(01)00246-2
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- Publication type:
- Article
Decrease of delayed rectifier currents in the subacute phase of infarction.
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- Cardiovascular Research, 2000, v. 48, n. 1, p. 11, doi. 10.1016/S0008-6363(00)00188-7
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- Publication type:
- Article
Injury current modulates afterdepolarizations in single human ventricular cells.
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- Cardiovascular Research, 2000, v. 47, n. 1, p. 124, doi. 10.1016/S0008-6363(00)00064-X
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- Publication type:
- Article
What we can learn from individual resuscitated patients.
- Published in:
- Cardiovascular Research, 2000, v. 46, n. 1, p. 14, doi. 10.1016/S0008-6363(00)00033-X
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- Publication type:
- Article
Implications of inhomogeneous distribution of IKS and IKr channels in ventricle with respect to effects of class III agents and beta-agonists.
- Published in:
- Cardiovascular Research, 1999, v. 43, n. 1, p. 20, doi. 10.1016/S0008-6363(99)00127-3
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- Publication type:
- Article
Decreased inward rectifier current in adult rabbit ventricular myocytes maintained in primary culture: a single-channel study.
- Published in:
- Cardiovascular Research, 1999, v. 42, n. 2, p. 424, doi. 10.1016/S0008-6363(99)00046-2
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- Publication type:
- Article
Neurokinin-3 receptor activation selectively prolongs atrial refractoriness by inhibition of a background K<sup>+</sup> channel.
- Published in:
- Nature Communications, 2018, v. 9, p. 1, doi. 10.1038/s41467-018-06530-5
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- Publication type:
- Article
Effects of acetylcholine and noradrenalin on action potentials of isolated rabbit sinoatrial and atrial myocytes.
- Published in:
- Frontiers in Physiology, 2012, v. 3, p. 1, doi. 10.3389/fphys.2012.00174
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- Publication type:
- Article