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DNA Alkylation Damage by Nitrosamines and Relevant DNA Repair Pathways.
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- International Journal of Molecular Sciences, 2023, v. 24, n. 5, p. 4684, doi. 10.3390/ijms24054684
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- Article
Expression of DNA repair genes and its relevance for DNA repair in peripheral immune cells of patients with posttraumatic stress disorder.
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- Scientific Reports, 2022, v. 12, n. 1, p. 1, doi. 10.1038/s41598-022-22001-w
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- Article
Adaptive upregulation of DNA repair genes following benzo(a)pyrene diol epoxide protects against cell death at the expense of mutations.
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- Nucleic Acids Research, 2016, v. 44, n. 22, p. 10727, doi. 10.1093/nar/gkw873
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- Article
Transcriptional regulation of human DNA repair genes following genotoxic stress: trigger mechanisms, inducible responses and genotoxic adaptation.
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- Nucleic Acids Research, 2013, v. 41, n. 18, p. 8403, doi. 10.1093/nar/gkt635
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- Article
Three prime exonuclease I (TREX1) is Fos/AP-1 regulated by genotoxic stress and protects against ultraviolet light and benzo(a)pyrene-induced DNA damage.
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- Nucleic Acids Research, 2010, v. 38, n. 19, p. 6418, doi. 10.1093/nar/gkq455
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- Article
c-Fos is required for excision repair of UV-light induced DNA lesions by triggering the re-synthesis of XPF.
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- Nucleic Acids Research, 2006, v. 34, n. 22, p. 6530, doi. 10.1093/nar/gkl895
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- Article
Expression of DNA repair proteins hMSH2, hMSH6, hMLH1, O.
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- International Journal of Cancer, 1999, v. 80, n. 5, p. 744, doi. 10.1002/(SICI)1097-0215(19990301)80:5<744::AID-IJC19>3.0.CO;2-5
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- Article
Survival and Death Strategies in Glioma Cells: Autophagy, Senescence and Apoptosis Triggered by a Single Type of Temozolomide-Induced DNA Damage.
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- PLoS ONE, 2013, v. 8, n. 1, p. 1, doi. 10.1371/journal.pone.0055665
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- Article
Fen1 is induced p53 dependently and involved in the recovery from UV-light-induced replication inhibition.
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- Oncogene, 2005, v. 24, n. 56, p. 8304, doi. 10.1038/sj.onc.1208994
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- Article
Alterations in Molecular Profiles Affecting Glioblastoma Resistance to Radiochemotherapy: Where Does the Good Go?
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- Cancers, 2022, v. 14, n. 10, p. 2416, doi. 10.3390/cancers14102416
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- Article
Senescence Is the Main Trait Induced by Temozolomide in Glioblastoma Cells.
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- Cancers, 2022, v. 14, n. 9, p. N.PAG, doi. 10.3390/cancers14092233
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- Article
Targeting c-IAP1, c-IAP2, and Bcl-2 Eliminates Senescent Glioblastoma Cells Following Temozolomide Treatment.
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- Cancers, 2021, v. 13, n. 14, p. 3585, doi. 10.3390/cancers13143585
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- Article
Natural Merosesquiterpenes Activate the DNA Damage Response via DNA Strand Break Formation and Trigger Apoptotic Cell Death in p53-Wild-Type and Mutant Colorectal Cancer.
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- Cancers, 2021, v. 13, n. 13, p. 3282, doi. 10.3390/cancers13133282
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- Article
Oxaliplatin-Induced Senescence in Colorectal Cancer Cells Depends on p14 ARF -Mediated Sustained p53 Activation.
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- Cancers, 2021, v. 13, n. 9, p. 2019, doi. 10.3390/cancers13092019
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- Article
Hypertrophic agonists induce the binding of c-Fos to an AP-1 site in cardiac myocytes: implications for the expression of GLUT1
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- Cardiovascular Research, 2003, v. 59, n. 3, p. 639, doi. 10.1016/S0008-6363(03)00472-3
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- Article
Phosphorylation of mismatch repair proteins MSH2 and MSH6 affecting MutSα mismatch-binding activity.
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- Nucleic Acids Research, 2002, v. 30, n. 9, p. 1959, doi. 10.1093/nar/30.9.1959
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- Article
Localization matters: nuclear-trapped Survivin sensitizes glioblastoma cells to temozolomide by elevating cellular senescence and impairing homologous recombination.
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- Cellular & Molecular Life Sciences, 2021, v. 78, n. 14, p. 5587, doi. 10.1007/s00018-021-03864-0
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- Article
Repair of O<sup>6</sup>-carboxymethylguanine adducts by O<sup>6</sup>-methylguanine-DNA methyltransferase in human colon epithelial cells.
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- Carcinogenesis, 2021, v. 42, n. 8, p. 1110, doi. 10.1093/carcin/bgab049
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- Article
Long-term activation of SAPK/JNK, p38 kinase and fas-L expression by cisplatin is attenuated in human carcinoma cells that acquired drug resistance.
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- International Journal of Cancer, 2004, v. 112, n. 6, p. 974, doi. 10.1002/ijc.20522
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- Article
Acquired resistance of melanoma cells to the antineoplastic agent fotemustine is caused by reactivation of the DNA repair gene mgmt.
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- International Journal of Cancer, 2001, v. 92, n. 1, p. 123, doi. 10.1002/1097-0215(200102)9999:9999<::AID-IJC1160>3.0.CO;2-V
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- Article
Repair gene O<sup>6</sup>‐methylguanine‐DNA methyltransferase is controlled by SP1 and up‐regulated by glucocorticoids, but not by temozolomide and radiation.
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- Journal of Neurochemistry, 2018, v. 144, n. 2, p. 139, doi. 10.1111/jnc.14262
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MGMT activity, promoter methylation and immunohistochemistry of pretreatment and recurrent malignant gliomas: a comparative study on astrocytoma and glioblastoma.
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- International Journal of Cancer, 2010, v. 127, n. 9, p. 2106, doi. 10.1002/ijc.25229
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- Article
p53 triggers mitochondrial apoptosis following DNA damage-dependent replication stress by the hepatotoxin methyleugenol.
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- Cell Death & Disease, 2022, v. 13, n. 11, p. 1, doi. 10.1038/s41419-022-05446-9
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- Article
Delayed c-Fos activation in human cells triggers XPF induction and an adaptive response to UVC-induced DNA damage and cytotoxicity.
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- Cellular & Molecular Life Sciences, 2011, v. 68, n. 10, p. 1785, doi. 10.1007/s00018-010-0546-9
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- Article
Targeting O<sup>6</sup>-methylguanine-DNA methyltransferase with specific inhibitors as a strategy in cancer therapy.
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- Cellular & Molecular Life Sciences, 2010, v. 67, n. 21, p. 3663, doi. 10.1007/s00018-010-0491-7
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- Article
Functional mismatch repair and inactive p53 drive sensitization of colorectal cancer cells to irinotecan via the IAP antagonist BV6.
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- Archives of Toxicology, 2019, v. 93, n. 8, p. 2265, doi. 10.1007/s00204-019-02513-7
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- Article
Sensitization of colorectal cancer cells to irinotecan by the Survivin inhibitor LLP3 depends on XAF1 proficiency in the context of mutated p53.
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- Archives of Toxicology, 2018, v. 92, n. 8, p. 2645, doi. 10.1007/s00204-018-2240-x
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- Article
Loss of Wilms tumor 1 protein is a marker for apoptosis in response to replicative stress in leukemic cells.
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- Archives of Toxicology, 2018, v. 92, n. 6, p. 2119, doi. 10.1007/s00204-018-2202-3
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Analysis of the interplay between all- trans retinoic acid and histone deacetylase inhibitors in leukemic cells.
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- Archives of Toxicology, 2017, v. 91, n. 5, p. 2191, doi. 10.1007/s00204-016-1878-5
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- Article
Abrogation of Cellular Senescence Induced by Temozolomide in Glioblastoma Cells: Search for Senolytics.
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- Cells (2073-4409), 2022, v. 11, n. 16, p. 2588, doi. 10.3390/cells11162588
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Lipoic Acid Synergizes with Antineoplastic Drugs in Colorectal Cancer by Targeting p53 for Proteasomal Degradation.
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- Cells (2073-4409), 2019, v. 8, n. 8, p. 794, doi. 10.3390/cells8080794
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The p21CIP1-CDK4-DREAM axis is a master regulator of genotoxic stress-induced cellular senescence.
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- Nucleic Acids Research, 2024, v. 52, n. 12, p. 6945, doi. 10.1093/nar/gkae426
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- Article
Benzo[a]pyrene represses DNA repair through altered E2F1/E2F4 function marking an early event in DNA damage-induced cellular senescence.
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- Nucleic Acids Research, 2020, v. 48, n. 21, p. 12085, doi. 10.1093/nar/gkaa965
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HDAC1 and HDAC2 integrate checkpoint kinase phosphorylation and cell fate through the phosphatase-2A subunit PR130.
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- Nature Communications, 2018, v. 9, n. 1, p. 1, doi. 10.1038/s41467-018-03096-0
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- Article
MGMT promoter methylation determined by HRM in comparison to MSP and pyrosequencing for predicting high-grade glioma response.
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- Clinical Epigenetics, 2016, v. 8, p. 1, doi. 10.1186/s13148-016-0204-7
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- Article