Found: 19
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Oligomerization of Alzheimer's β-Amyloid within Processes and Synapses of Cultured Neurons and Brain.
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- Journal of Neuroscience, 2004, v. 24, n. 14, p. 3592, doi. 10.1523/JNEUROSCI.5167-03.2004
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- Article
BIN1 LOAD mutants increase intracellular Aβ by interfering with BACE1 recycling.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2021, v. 17, n. 3, p. 1, doi. 10.1002/alz.054178
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- Article
Lysosomal subcellular distribution in neurons: Are there synaptic lysosomes?
- Published in:
- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2021, v. 17, n. 3, p. 1, doi. 10.1002/alz.053844
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- Article
Alzheimer's disease risk factor CD2AP functions at synapses via spinal F‐actin.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2021, v. 17, n. 3, p. 1, doi. 10.1002/alz.053797
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- Article
The late‐onset Alzheimer's disease genetic risk factor CD2AP impacts synapses via actin dynamics: Basic science and pathogenesis: Genetics and omics of AD.
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- Alzheimer's & Dementia: The Journal of the Alzheimer's Association, 2020, v. 16, n. 11, p. 1, doi. 10.1002/alz.043900
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- Article
P4-260: Intraneuronal Aβ42 and microtubule-associated protein pathologies begin in synapses
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- 2008
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- Abstract
P4-050: Endogenous Abeta42 accumulation in neurons impairs the multivesicular body sorting pathway
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- 2006
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- Abstract
P4-041: The Arctic Alzheimer mutation favors Aβ production by making APP less available to α-secretase at the cell surface
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- 2006
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- Abstract
O4-06-08: Internalized antibodies to the Aβ domain of APP reduce intraneuronal Aβ and protect against synaptic alterations
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- 2006
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- Abstract
P4-050: Endogenous Abeta42 accumulation in neurons impairs the multivesicular body sorting pathway
- Published in:
- 2006
- By:
- Publication type:
- Abstract
P4-041: The Arctic Alzheimer mutation favors Aβ production by making APP less available to α-secretase at the cell surface
- Published in:
- 2006
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- Publication type:
- Abstract
O4-06-08: Internalized antibodies to the Aβ domain of APP reduce intraneuronal Aβ and protect against synaptic alterations
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- 2006
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- Publication type:
- Abstract
β-Amyloid Accumulation Impairs Multivesicular Body Sorting by Inhibiting the Ubiquitin-Proteasome System.
- Published in:
- Journal of Neuroscience, 2006, v. 26, n. 16, p. 4277, doi. 10.1523/JNEUROSCI.5078-05.2006
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- Article
Myosin 1b promotes the formation of post-Golgi carriers by regulating actin assembly and membrane remodelling at the trans-Golgi network.
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- Nature Cell Biology, 2011, v. 13, n. 7, p. 779, doi. 10.1038/ncb2262
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- Article
Amyloid‐β oligomers are inefficiently measured by enzyme‐linked immunosorbent assay.
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- Annals of Neurology, 2005, v. 58, n. 1, p. 147
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- Article
Amyloid‐β oligomers are inefficiently measured by enzyme‐linked immunosorbent assay.
- Published in:
- Annals of Neurology, 2005, v. 58, n. 1, p. 147
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- Publication type:
- Article
Regulation of NMDA receptor trafficking by amyloid-β.
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- Nature Neuroscience, 2005, v. 8, n. 8, p. 1051, doi. 10.1038/nn1503
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- Article
The Arctic Alzheimer mutation favors intracellular amyloid-β production by making amyloid precursor protein less available to α-secretase.
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- Journal of Neurochemistry, 2007, v. 101, n. 3, p. 854, doi. 10.1111/j.1471-4159.2006.04443.x
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- Article
Increased plaque burden in brains of APP mutant MnSOD heterozygous knockout mice.
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- Journal of Neurochemistry, 2004, v. 89, n. 5, p. 1308, doi. 10.1111/j.1471-4159.2004.02455.x
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- Article