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Mitochondrial DNA Together with miR-142-3p in Plasma Can Predict Unfavorable Outcomes in Patients after Acute Myocardial Infarction.
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- International Journal of Molecular Sciences, 2022, v. 23, n. 17, p. 9947, doi. 10.3390/ijms23179947
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- Article
Increased miR-142 Levels in Plasma and Atherosclerotic Plaques from Peripheral Artery Disease Patients with Post-Surgery Cardiovascular Events.
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- International Journal of Molecular Sciences, 2020, v. 21, n. 24, p. 9600, doi. 10.3390/ijms21249600
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- Article
MiR-486 and miR-92a Identified in Circulating HDL Discriminate between Stable and Vulnerable Coronary Artery Disease Patients.
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- PLoS ONE, 2015, v. 10, n. 10, p. 1, doi. 10.1371/journal.pone.0140958
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- Article
Oscillating Glucose Induces the Increase in Inflammatory Stress through Ninjurin-1 Up-Regulation and Stimulation of Transport Proteins in Human Endothelial Cells.
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- Biomolecules (2218-273X), 2023, v. 13, n. 4, p. 626, doi. 10.3390/biom13040626
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- Article
CRISPR/dCas9 Transcriptional Activation of Endogenous Apolipoprotein AI and Paraoxonase 1 in Enterocytes Alleviates Endothelial Cell Dysfunction.
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- Biomolecules (2218-273X), 2021, v. 11, n. 12, p. 1769, doi. 10.3390/biom11121769
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- Article
Aggregated LDL turn human macrophages into foam cells and induce mitochondrial dysfunction without triggering oxidative or endoplasmic reticulum stress.
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- PLoS ONE, 2021, v. 16, n. 1, p. 1, doi. 10.1371/journal.pone.0245797
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- Article
Inhibition of miR-486 and miR-92a decreases liver and plasma cholesterol levels by modulating lipid-related genes in hyperlipidemic hamsters.
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- Molecular Biology Reports, 2018, v. 45, n. 4, p. 497, doi. 10.1007/s11033-018-4186-8
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- Article
Foam cell-derived 4-hydroxynonenal induces endothelial cell senescence in a TXNIP-dependent manner.
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- Journal of Cellular & Molecular Medicine, 2015, v. 19, n. 8, p. 1887, doi. 10.1111/jcmm.12561
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- Article
Effect of irreversibly glycated LDL in human vascular smooth muscle cells: lipid loading, oxidative and inflammatory stress.
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- Journal of Cellular & Molecular Medicine, 2010, v. 14, n. 12, p. 2790, doi. 10.1111/j.1582-4934.2009.00933.x
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- Article
Oxidized LDL-Exposed Human Macrophages Display Increased MMP-9 Expression and Secretion Mediated by Endoplasmic Reticulum Stress.
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- Journal of Cellular Biochemistry, 2017, v. 118, n. 4, p. 661, doi. 10.1002/jcb.25637
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- Article
Regulation of microRNAs in high-fat diet induced hyperlipidemic hamsters.
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- Scientific Reports, 2020, v. 10, n. 1, p. N.PAG, doi. 10.1038/s41598-020-77539-4
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- Article
Hyperglycemia Determines Increased Specific MicroRNAs Levels in Sera and HDL of Acute Coronary Syndrome Patients and Stimulates MicroRNAs Production in Human Macrophages.
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- PLoS ONE, 2016, v. 11, n. 8, p. 1, doi. 10.1371/journal.pone.0161201
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- Article
Vascular endothelium in atherosclerosis.
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- Cell & Tissue Research, 2008, v. 335, n. 1, p. 191, doi. 10.1007/s00441-008-0678-5
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- Article
MiR-223-3p levels in the plasma and atherosclerotic plaques are increased in aged patients with carotid artery stenosis; association with HDL-related proteins.
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- Molecular Biology Reports, 2022, v. 49, n. 7, p. 6779, doi. 10.1007/s11033-021-06636-y
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- Article
Hyperlipidemia-induced hepatic and small intestine ER stress and decreased paraoxonase 1 expression and activity is associated with HDL dysfunction in Syrian hamsters.
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- Molecular Nutrition & Food Research, 2015, v. 59, n. 11, p. 2293, doi. 10.1002/mnfr.201500422
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- Article
Elevated Levels of Circulating lncRNAs LIPCAR and MALAT1 Predict an Unfavorable Outcome in Acute Coronary Syndrome Patients.
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- International Journal of Molecular Sciences, 2023, v. 24, n. 15, p. 12076, doi. 10.3390/ijms241512076
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- Article
Clusterin, paraoxonase 1, and myeloperoxidase alterations induce high‐density lipoproteins dysfunction and contribute to peripheral artery disease; aggravation by type 2 diabetes mellitus.
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- Biofactors, 2022, v. 48, n. 2, p. 454, doi. 10.1002/biof.1800
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- Article
Caffeic acid attenuates the inflammatory stress induced by glycated LDL in human endothelial cells by mechanisms involving inhibition of AGE-receptor, oxidative, and endoplasmic reticulum stress.
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- Biofactors, 2017, v. 43, n. 5, p. 685, doi. 10.1002/biof.1373
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- Article