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The landscape of the COVID-19 pandemic in Poland emerging from epidemiological and genomic data.
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- Scientific Reports, 2024, v. 14, n. 1, p. 1, doi. 10.1038/s41598-024-65468-5
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Circulating miRNA-19b as a biomarker of disease progression and treatment response to baricitinib in rheumatoid arthritis patients through miRNA profiling of monocytes.
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- Frontiers in Immunology, 2023, v. 14, p. 1, doi. 10.3389/fimmu.2023.980247
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Tuning Monocytes and Macrophages for Personalized Therapy and Diagnostic Challenge in Rheumatoid Arthritis.
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- Cells (2073-4409), 2021, v. 10, n. 8, p. 1860, doi. 10.3390/cells10081860
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Biologic Drugs for Rheumatoid Arthritis in the Context of Biosimilars, Genetics, Epigenetics and COVID-19 Treatment.
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- Cells (2073-4409), 2021, v. 10, n. 2, p. 323, doi. 10.3390/cells10020323
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Small Molecule Inhibitors in the Treatment of Rheumatoid Arthritis and Beyond: Latest Updates and Potential Strategy for Fighting COVID-19.
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- Cells (2073-4409), 2020, v. 9, n. 8, p. 1876, doi. 10.3390/cells9081876
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DNA Methylation as a Future Therapeutic and Diagnostic Target in Rheumatoid Arthritis.
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- Cells (2073-4409), 2019, v. 8, n. 9, p. 953, doi. 10.3390/cells8090953
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B lymphocytes acquire and present intracellular antigens that have relocated to the surface of apoptotic target cells.
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- European Journal of Immunology, 2011, v. 41, n. 7, p. 1850, doi. 10.1002/eji.201141472
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The Kinetics of Humoral and Cellular Responses after the Booster Dose of COVID-19 Vaccine in Inflammatory Arthritis Patients.
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- Viruses (1999-4915), 2023, v. 15, n. 3, p. 620, doi. 10.3390/v15030620
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Circulating miRNA Correlates with Lipid Profile and Disease Activity in Psoriatic Arthritis, Rheumatoid Arthritis, and Ankylosing Spondylitis Patients.
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- Biomedicines, 2022, v. 10, n. 4, p. 893, doi. 10.3390/biomedicines10040893
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Global miRNA and mRNA expression profiles identify miRNA‐26a‐2‐3p‐dependent repression of IFN signature in systemic sclerosis human monocytes.
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- European Journal of Immunology, 2020, v. 50, n. 7, p. 1057, doi. 10.1002/eji.201948428
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The role of micro RNA-5196 in the pathogenesis of systemic sclerosis.
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- European Journal of Clinical Investigation, 2017, v. 47, n. 8, p. 555, doi. 10.1111/eci.12776
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MiR-29a Reduces TIMP-1 Production by Dermal Fibroblasts via Targeting TGF-β Activated Kinase 1 Binding Protein 1, Implications for Systemic Sclerosis.
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- PLoS ONE, 2014, v. 9, n. 12, p. 1, doi. 10.1371/journal.pone.0115596
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Current frontiers in systemic sclerosis pathogenesis.
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- Experimental Dermatology, 2015, v. 24, n. 6, p. 401, doi. 10.1111/exd.12673
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Comprehensive microRNA and transcriptomic profiling of rheumatoid arthritis monocytes: role of microRNA-146b in pro-inflammatory progression.
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- Rheumatology, 2021, v. 60, n. 11, p. 5424, doi. 10.1093/rheumatology/keab407
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Epigenetic Modulation as a Therapeutic Prospect for Treatment of Autoimmune Rheumatic Diseases.
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- Mediators of Inflammation, 2016, p. 1, doi. 10.1155/2016/9607946
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Serum amyloid A induces interleukin-6 in dermal fibroblasts via Toll-like receptor 2, interleukin-1 receptor-associated kinase 4 and nuclear factor- κB.
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- Immunology, 2014, v. 143, n. 3, p. 331, doi. 10.1111/imm.12260
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Antigen-specific B lymphocytes acquire proteoglycan aggrecan from cartilage extracellular matrix resulting in antigen presentation and CD4<sup>+</sup> T-cell activation.
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- Immunology, 2014, v. 141, n. 1, p. 70, doi. 10.1111/imm.12169
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Age-related changes in the occurrence and characteristics of thymic CD4<sup>+</sup> CD25<sup>+</sup> T cells in mice.
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- Immunology, 2007, v. 122, n. 3, p. 445, doi. 10.1111/j.1365-2567.2007.02667.x
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Histone Demethylation and Toll-like Receptor 8-Dependent Cross-Talk in Monocytes Promotes Transdifferentiation of Fibroblasts in Systemic Sclerosis Via Fra-2.
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- Arthritis & Rheumatology, 2016, v. 68, n. 6, p. 1493, doi. 10.1002/art.39602
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Corrigendum: IL-13 mediates collagen deposition via STAT6 and microRNA-135b: a role for epigenetics.
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- Scientific Reports, 2016, p. 27325, doi. 10.1038/srep27325
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IL-13 mediates collagen deposition via STAT6 and microRNA-135b: a role for epigenetics.
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- Scientific Reports, 2016, p. 25066, doi. 10.1038/srep25066
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Potential Mechanism of Fatigue Induction and Its Management by JAK Inhibitors in Inflammatory Rheumatic Diseases.
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- Journal of Inflammation Research, 2023, v. 16, p. 3949, doi. 10.2147/JIR.S414739
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Effectiveness of Soluble CTLA-4-Fc in the Inhibition of Bone Marrow T-Cell Activation in Context of Indoleamine 2.3-Dioxygenase (IDO) and CD4<sup>+</sup>Foxp3<sup>+</sup> Treg Induction.
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- Journal of Inflammation Research, 2022, v. 15, p. 6813, doi. 10.2147/JIR.S359775
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Changes in MiRNA-5196 Expression as a Potential Biomarker of Anti-TNF-α Therapy in Rheumatoid Arthritis and Ankylosing Spondylitis Patients.
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- Archivum Immunologiae & Therapiae Experimentalis, 2018, v. 66, n. 5, p. 389, doi. 10.1007/s00005-018-0513-y
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Significance of Polymorphism and Expression of miR-146a and NFkB1 Genetic Variants in Patients with Rheumatoid Arthritis.
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- Archivum Immunologiae & Therapiae Experimentalis, 2016, v. 64, p. 131, doi. 10.1007/s00005-016-0443-5
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S100A8 and S100A12 Proteins as Biomarkers of High Disease Activity in Patients with Rheumatoid Arthritis That Can Be Regulated by Epigenetic Drugs.
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- International Journal of Molecular Sciences, 2023, v. 24, n. 1, p. 710, doi. 10.3390/ijms24010710
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Impact of Adipose-Derived Mesenchymal Stem Cells (ASCs) of Rheumatic Disease Patients on T Helper Cell Differentiation.
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- International Journal of Molecular Sciences, 2022, v. 23, n. 10, p. 5317, doi. 10.3390/ijms23105317
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