Found: 24
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Suppressing aberrant GluN3A expression rescues synaptic and behavioral impairments in Huntington's disease models.
- Published in:
- Nature Medicine, 2013, v. 19, n. 8, p. 1030, doi. 10.1038/nm.3246
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- Publication type:
- Article
Balance between synaptic versus extrasynaptic NMDA receptor activity influences inclusions and neurotoxicity of mutant huntingtin.
- Published in:
- Nature Medicine, 2009, v. 15, n. 12, p. 1407, doi. 10.1038/nm.2056
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- Publication type:
- Article
Activation of caspase‐6 and cleavage of caspase‐6 substrates is an early event in NMDA receptor–mediated excitotoxicity.
- Published in:
- Journal of Neuroscience Research, 2018, v. 96, n. 3, p. 391, doi. 10.1002/jnr.24153
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- Publication type:
- Article
Differential Susceptibility to Excitotoxic Stress in YAC128 Mouse Models of Huntington Disease between Initiation and Progression of Disease.
- Published in:
- Journal of Neuroscience, 2009, v. 29, n. 7, p. 2193, doi. 10.1523/JNEUROSCI.5473-08.2009
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- Publication type:
- Article
Polyglutamine-Modulated Striatal Calpain Activity in YAC Transgenic Huntington Disease Mouse Model: Impact on NMDA Receptor Function and Toxicity.
- Published in:
- Journal of Neuroscience, 2008, v. 28, n. 48, p. 12725, doi. 10.1523/JNEUROSCI.4619-08.2008
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- Publication type:
- Article
Prevention of depressive behaviour in the YAC128 mouse model of Huntington disease by mutation at residue 586 of huntingtin.
- Published in:
- 2009
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- Publication type:
- journal article
Prevention of depressive behaviour in the YAC128 mouse model of Huntington disease by mutation at residue 586 of huntingtin.
- Published in:
- Brain: A Journal of Neurology, 2009, v. 132, n. 4, p. 919
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- Publication type:
- Article
Diet enriched in omega‐3 fatty acids alleviates olfactory system deficits in APOE4 transgenic mice.
- Published in:
- European Journal of Neuroscience, 2021, v. 54, n. 9, p. 7092, doi. 10.1111/ejn.15472
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- Publication type:
- Article
Differential modulation of endotoxin responsiveness by human caspase-12 polymorphisms.
- Published in:
- Nature, 2004, v. 429, n. 6987, p. 75, doi. 10.1038/nature02451
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- Publication type:
- Article
Interactome network analysis identifies multiple caspase-6 interactors involved in the pathogenesis of HD.
- Published in:
- Human Molecular Genetics, 2016, v. 25, n. 8, p. 1600, doi. 10.1093/hmg/ddw036
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- Publication type:
- Article
The palmitoyl acyltransferase HIP14 shares a high proportion of interactors with huntingtin: implications for a role in the pathogenesis of Huntington's disease.
- Published in:
- Human Molecular Genetics, 2014, v. 23, n. 15, p. 4142, doi. 10.1093/hmg/ddu137
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- Publication type:
- Article
Rescue from excitotoxicity and axonal degeneration accompanied by age-dependent behavioral and neuroanatomical alterations in caspase-6-deficient mice.
- Published in:
- Human Molecular Genetics, 2012, v. 21, n. 9, p. 1954, doi. 10.1093/hmg/dds005
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- Publication type:
- Article
Full-length huntingtin levels modulate body weight by influencing insulin-like growth factor 1 expression.
- Published in:
- Human Molecular Genetics, 2010, v. 19, n. 8, p. 1528, doi. 10.1093/hmg/ddq026
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- Publication type:
- Article
Accumulation of N-terminal mutant huntingtin in mouse and monkey models implicated as a pathogenic mechanism in Huntington's disease.
- Published in:
- Human Molecular Genetics, 2008, v. 17, n. 17, p. 2738, doi. 10.1093/hmg/ddn175
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- Publication type:
- Article
Activated caspase-6 and caspase-6-cleaved fragments of huntingtin specifically colocalize in the nucleus.
- Published in:
- Human Molecular Genetics, 2008, v. 17, n. 15, p. 2390, doi. 10.1093/hmg/ddn139
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- Publication type:
- Article
Selective striatal neuronal loss in a YAC128 mouse model of Huntington disease.
- Published in:
- Human Molecular Genetics, 2003, v. 12, n. 13, p. 1555, doi. 10.1093/hmg/ddg169
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- Publication type:
- Article
Wild-type huntingtin protects neurons from excitotoxicity.
- Published in:
- Journal of Neurochemistry, 2006, v. 96, n. 4, p. 1121, doi. 10.1111/j.1471-4159.2005.03605.x
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- Publication type:
- Article
Life Without Huntingtin: Normal Differentiation into Functional Neurons.
- Published in:
- Journal of Neurochemistry, 1999, v. 72, n. 3, p. 1009, doi. 10.1046/j.1471-4159.1999.0721009.x
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- Publication type:
- Article
Amelioration of Cognitive and Olfactory System Deficits in APOE4 Transgenic Mice with DHA Treatment.
- Published in:
- Molecular Neurobiology, 2023, v. 60, n. 10, p. 5624, doi. 10.1007/s12035-023-03401-z
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- Publication type:
- Article
Sequence of the murine Huntington dusease gene: evidence for conservation, and polymorphism in a triplet (CCG) repeat alternate splicing.
- Published in:
- Human Molecular Genetics, 1994, v. 3, n. 1, p. 85
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- Publication type:
- Article
Differential 3′ polyadenylation of the Huntington disease gene results in two mRNA species with variable tissue expression.
- Published in:
- Human Molecular Genetics, 1993, v. 2, n. 10, p. 1541
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- Publication type:
- Article
Transgenic Mouse Model Expressing the Caspase 6 Fragment of Mutant Huntingtin.
- Published in:
- Journal of Neuroscience, 2012, v. 32, n. 1, p. 183, doi. 10.1523/JNEUROSCI.1305-11.2012
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- Publication type:
- Article
Cleavage at the 586 Amino Acid Caspase-6 Site in Mutant huntingtin Influences Caspase-6 Activation In Vivo.
- Published in:
- Journal of Neuroscience, 2010, v. 30, n. 45, p. 15019, doi. 10.1523/JNEUROSCI.2071-10.2010
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- Publication type:
- Article
Phosphorylation of Huntingtin at Ser<sup>421</sup> in YAC128 Neurons Is Associated with Protection of YAC 128 Neurons from NMDA-Mediated Excitotoxicity and Is Modulated by PP1 and PP2A.
- Published in:
- Journal of Neuroscience, 2010, v. 30, n. 43, p. 14318, doi. 10.1523/JNEUROSCI.1589-10.2010
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- Publication type:
- Article