Found: 10
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Platypnea-Orthodeoxia Syndrome: Diagnostic Challenge and the Importance of Heightened Clinical Suspicion.
- Published in:
- 2015
- By:
- Publication type:
- Case Study
Ergotamine-Associated Valvulopathy.
- Published in:
- 2012
- By:
- Publication type:
- Case Study
ABCC9 mutations identified in human dilated cardiomyopathy disrupt catalytic K<sub>ATP</sub> channel gating.
- Published in:
- Nature Genetics, 2004, v. 36, n. 4, p. 382, doi. 10.1038/ng1329
- By:
- Publication type:
- Article
Gene knockout of the KCNJ8-encoded Kir6.1 K<sub>ATP</sub> channel imparts fatal susceptibility to endotoxemia.
- Published in:
- FASEB Journal, 2006, v. 20, n. 13, p. 2271, doi. 10.1096/fj.06-6349com
- By:
- Publication type:
- Article
ABCD-GENE Score and Clinical Outcomes Following Percutaneous Coronary Intervention: Insights from the TAILOR-PCI Trial.
- Published in:
- 2022
- By:
- Publication type:
- journal article
Genetic Disruption of Kir6.2, the Pore-Forming Subunit ofATP-Sensitive K[sup+] Channel, Predisposes toCatecholamine-Induced Ventricular Dysrhythmia.
- Published in:
- Diabetes, 2004, v. 53, p. S165, doi. 10.2337/diabetes.53.suppl_3.S165
- By:
- Publication type:
- Article
ATP-Sensitive K[sup+] Channel Knockout Compromises theMetabolic Benefit of Exercise Training, Resulting inCardiac Deficits.
- Published in:
- Diabetes, 2004, v. 53, p. S169, doi. 10.2337/diabetes.53.suppl_3.S169
- By:
- Publication type:
- Article
Safety and Efficacy of CYP2C19 Genotype-Guided Escalation of P2Y<sub>12</sub> Inhibitor Therapy After Percutaneous Coronary Intervention in Chronic Kidney Disease: a Post Hoc Analysis of the TAILOR-PCI Study.
- Published in:
- Cardiovascular Drugs & Therapy, 2024, v. 38, n. 3, p. 447, doi. 10.1007/s10557-022-07392-2
- By:
- Publication type:
- Article
KCNJ11 gene knockout of the Kir6.2 KATP channel causes maladaptive remodeling and heart failure in hypertension.
- Published in:
- Human Molecular Genetics, 2006, v. 15, n. 15, p. 2285, doi. 10.1093/hmg/ddl154
- By:
- Publication type:
- Article
Transgenic overexpression of human DMPK accumulates into hypertrophic cardiomyopathy, myotonic myopathy and hypotension traits of myotonic dystrophy.
- Published in:
- Human Molecular Genetics, 2004, v. 13, n. 20, p. 2505, doi. 10.1093/hmg/ddh266
- By:
- Publication type:
- Article